scholarly journals Facial Nerve Palsy After Occipital Nerve Blocks — A Case Report

2021 ◽  
Author(s):  
Abdullah Shoaib

The use of bupivacaine and other local anesthetics to perform peripheral nerve blocks is a mainstay in the management of refractory headaches and migraines. In this case report, a patient presented with transient facial nerve palsy shortly after receiving occipital nerve blocks for headaches. Patient’s symptoms self-resolved, and her symptoms were thought to be due to displacement and spreading of bupivacaine to the facial nerve. The spread of bupivacaine can be facilitated by tracking across fascial planes or nerve sheaths. Similar side effects have been seen in dental anesthesia, but there has only been one other report of such a presentation associated with occipital nerve blocks, and none in pediatric patients. Physicians should be aware of this rare complication with peripheral nerve blocks.

2014 ◽  
Vol 54 (10) ◽  
pp. 1651-1655 ◽  
Author(s):  
Lauren Strauss ◽  
Elizabeth Loder ◽  
Paul Rizzoli

2018 ◽  
Vol 4 (5) ◽  
pp. 369-371
Author(s):  
Rajashree U Gandhe . ◽  
Chinmaya P Bhave . ◽  
Avinash S Kakde . ◽  
Neha T Gedam .

2018 ◽  
Vol 9 (1) ◽  
pp. 60 ◽  
Author(s):  
Hussein Kamel ◽  
Kazim Mohammed ◽  
Javeed Iqbal ◽  
John Mathew ◽  
Ghanem Al-Sulaiti

2020 ◽  
Vol 16 (4) ◽  
pp. 430-432
Author(s):  
Thilaga Rajendran ◽  
◽  
Jeyasakthy Saniasiaya ◽  
Yatiee Swany Lahuri ◽  
Norhaslinda Binti Abdul Gani ◽  
...  

The incidence of tick infestation reported by ear-nose-throat outpatient clinics is high, though the exact reason is unknown. Affected patients generally recover well without any local or systemic sequelae. Tick-induced facial nerve palsy is less commonly reported in the literature. In this case report, we present our experience in managing a case of delayed isolated facial nerve palsy from intra-aural tick infestation in a child. The case highlights the possibility of late effects of tick paralysis occurring hours after tick removal. Close observation and awareness are crucial to detect any signs of neurotoxicity associated with tick infestation.


Author(s):  
Jing Yang ◽  
Yu Cui ◽  
Rong Cao ◽  
Qing-Hua Huang ◽  
Qian-Qian Zhang

2019 ◽  
Vol 13 (1) ◽  
Author(s):  
B. M. D. B. Basnayake ◽  
A. W. M. Wazil ◽  
N. Nanayakkara ◽  
R. M. B. S. S. Mahanama ◽  
P. N. S. Premathilake ◽  
...  

2019 ◽  
Vol 48 (3) ◽  
pp. 030006051986749
Author(s):  
Yu-Ming Liu ◽  
Yan-Li Chen ◽  
Yan-Hua Deng ◽  
Yan-Ling Liang ◽  
Wei Li ◽  
...  

Miller Fisher syndrome (MFS), a variant of Guillain–Barré syndrome, is characterized by ataxia, areflexia and ophthalmoplegia. This case report describes a 40-year old male that presented with a 3-day history of unsteady walking and numbness on both hands, and a 2-day history of seeing double images and unclear articulation. Lumbar puncture revealed an opening pressure of 260 mm H2O. Plasma serology was positive for anti-ganglioside M1-immunoglobulin M (anti-GM1-IgM) antibodies and negative for anti-ganglioside Q1b (anti-GQ1b) antibodies. The patient was diagnosed with MFS based on the clinical course and neurophysiological findings. On the 4th day of treatment with intravenous immunoglobulin (IVIG), his ataxia and unsteady walking improved, but his bilateral eyeballs were fixed, and over the next few days he developed bilateral peripheral facial paralysis. After 5 days of IVIG treatment, methylprednisolone treatment was offered and the patient's symptoms gradually improved. Early intracranial hypertension and delayed facial nerve palsy may be atypical presentations of MFS. Anti-GM1-IgM antibodies may be the causative antibodies for MFS. If the IVIG therapy does not stop the progression of the disease, the addition of corticosteroid therapy may be effective. However, the relationship between IgM type, anti-GM1 antibody and MFS remains unclear and requires further research.


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