Cryosurgery on Postganglionic Fibers (Posterior Nasal Branches) of the Pterygopalatine Ganglion for Vasomotor Rhinitis

Eight dogs underwent denervation of the nasal mucosa by bilateral surgical interruption of the caudal nasal nerve, major palatine nerves, nerve branches traversing the pterygopalatine ganglion, and ethmoid nerve. Determinations of pulmonary resistance and compliance immediately before denervation and 1 hour, 24 hours, and 7 days after denervation did not demonstrate a tonic nasal influence on pulmonary mechanics. Changes within the range of normal did occur in the immediate period after denervation. Because pulmonary resistance and compliance approached control values by the seventh day after denervation, these variations are probably the result of atelectasis and pooling of mucus within the bronchi, secondary to prolonged general anesthesia. Thus, pulmonary mechanics probably are not significantly altered after denervation of the nasal mucosa.

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INCIDENCE OF VASOMOTOR RHINITIS DURING PREGNANCY

Archives of Otolaryngology - Head and Neck Surgery ◽

10.1001/archotol.1943.00670030713009 ◽

1943 ◽

Vol 37 (5) ◽

pp. 699-709 ◽

Cited By ~ 18

Author(s):

M. MOHUN

Keyword(s):

Vasomotor Rhinitis

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Management of vasomotor rhinitis patients

Spravočnik vrača obŝej praktiki (Journal of Family Medicine) ◽

10.33920/med-10-2009-03 ◽

2020 ◽

pp. 24-28

Author(s):

Konstantin Robertovich Gulyabin

Keyword(s):

Viral Infection ◽

Nasal Congestion ◽

Clinical Manifestations ◽

Posterior Pharyngeal Wall ◽

Respiratory Obstruction ◽

Vasomotor Rhinitis ◽

Respiratory Viral Infection ◽

Pharyngeal Wall ◽

Chronic Rhinitis ◽

The Subject

There has been a recent obvious trend towards the increased prevalence of chronic rhinitis – 10-20% of the population experiences this disorder. Vasomotor rhinitis, sometimes also called idiopathic rhinitis, is the indisputable leader among various chronic rhinitis forms (allergic, infectious, atrophic, catarrhal and hypertrophic). The term of vasomotor rhinitis has been the subject of experts' repeated criticism because neurovisceral innervation disorders that underlie this condition are found in almost every form of chronic rhinitis. The main clinical manifestations of vasomotor rhinitis include a feeling of nasal congestion and nasal respiratory obstruction, regular abundant discharge of clear mucus and a feeling of its trickling down the posterior pharyngeal wall. A past respiratory viral infection treated by excessive quantities of vasoconstrictor drops triggers the vasomotor rhinitis onset in most cases.

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Somatostatin immunoreactivity in quail pterygopalatine ganglion

Journal of Anatomy ◽

10.1111/j.1469-7580.2005.00380.x ◽

2005 ◽

Vol 206 (3) ◽

pp. 249-255 ◽

Cited By ~ 6

Author(s):

Falk Schrodl ◽

M. Egle De Stefano ◽

Francis Minvielle ◽

Axel Brehmer ◽

Winfried L. Neuhuber

Keyword(s):

Pterygopalatine Ganglion ◽

Somatostatin Immunoreactivity

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Rhinitis during pregnancy and rhinitis medicamentosa

Otolaryngology ◽

10.1177/019459989210700606.2 ◽

1992 ◽

Vol 107 (6_part_2) ◽

pp. 845-849 ◽

Cited By ~ 24

Author(s):

Mary D. Lekas

Keyword(s):

Intranasal Corticosteroid ◽

Short Term ◽

Vasomotor Rhinitis ◽

Nasal Sprays ◽

Nasal Steroids ◽

Rebound Phenomenon ◽

Nasal Blood Vessels ◽

Rhinitis Medicamentosa ◽

Corticosteroid Injections ◽

Catecholamine Levels

Vasomotor rhinitis is a nonspecific disorder that is caused neither by infection nor allergy but rather by an imbalance of the autonomic nervous system with a preponderant action of parasympathetic fibers on nasal blood vessels. Rhinitis during pregnancy appears to result from the increased production of estrogen; increased estrogen levels caused by treatment, puberty, or liver disease may also cause rhinitis. Nasal saline mist, antihistamines, and topical corticosteroids are recommended; intranasal corticosteroid injections are also useful but must be administered under expert care. Rhinitis medicamentosa results from overuse of topical vasoconstrictors, which produce a rebound phenomenon. Rebound can also result from numerous medications, including antihypertensive preparations that reduce catecholamine levels, antidepressants, antipsychotics, and tranquilizers. Management of rhinitis medicamentosa consists in limiting the use of vasoconstrictors to no more than 3 days and giving the patient saline nasal sprays, daytime oral vasoconstrictors, and nocturnal antihistamines. Corticosteroids, preferably topical nasal steroids rather than even a short-term course of systemic administration, should also be used.

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