Cerebral mycotic aneurysm of fungal origin

1978 ◽  
Vol 49 (1) ◽  
pp. 0107-0110 ◽  
Author(s):  
Gulshan K. Ahuja ◽  
Neeraj Jain ◽  
Malini Vijayaraghavan ◽  
Subimal Roy

✓ A young man who had a long history of sinusitis developed subarachnoid hemorrhage and died. Autopsy showed a mycotic aneurysm of fungal origin at the junction of the right posterior cerebral and internal carotid arteries. Four of five reported cases of fungal aneurysm were due to Aspergillus infection.

1997 ◽  
Vol 87 (3) ◽  
pp. 436-439 ◽  
Author(s):  
Keiichiro Maeda ◽  
Hiroki Kurita ◽  
Tsuneo Nakamura ◽  
Masaaki Usui ◽  
Kazuo Tsutsumi ◽  
...  

✓ The authors present two rare cases of severe cerebral vasospasm following the rupture of arteriovenous malformations (AVMs). Computerized tomography revealed intracerebral hemorrhage in the thalamus in one case and in the putamen in the other, both accompanied by cast formation of intraventricular clots without radiological evidence of subarachnoid hemorrhage. Initial angiograms showed arterial narrowing of the bilateral internal carotid arteries in the supraclinoid portion but failed to demonstrate an arteriovenous shunt. Subsequent angiograms clearly demonstrated the existence of an AVM. Radiological features and possible mechanisms are discussed.


1975 ◽  
Vol 42 (4) ◽  
pp. 478-482 ◽  
Author(s):  
Ivan W. Rosen ◽  
Damon F. Mills ◽  
Harold I. Nadel ◽  
Donald D. Kaiserman

✓ The authors present angiographic demonstration of bilateral, congenital, total absence of the internal carotid arteries in a patient with subarachnoid hemorrhage. Angiography demonstrated complete absence of both internal carotid arteries. The intracranial blood supply had been provided by communication between a hypertrophied basilar artery and the circle of Willis through the posterior communicating arteries. An aneurysm, believed to be the site of the subarachnoid hemorrhage, arose from the basilar artery. Absence of the carotid canals substantiated the congenital nature of the anomaly. The related embryology is reviewed.


1980 ◽  
Vol 53 (3) ◽  
pp. 300-304 ◽  
Author(s):  
Toshisuke Sakaki ◽  
Kazuhiko Kinugawa ◽  
Tatsuo Tanigake ◽  
Seiji Miyamoto ◽  
Kikuo Kyoi ◽  
...  

✓ Embolism from an aneurysm is one of the mechanisms involved in the pathogenesis of ischemic symptoms associated with intracranial aneurysms. Four cases are reported in which aneurysms of the internal carotid arteries and middle cerebral arteries were the source of emboli resulting in cerebral infarction. In the treatment of these aneurysms, it is best to clip the neck of the aneurysm with great care to avoid embolism due to extrusion of clot into the distal artery.


1991 ◽  
Vol 75 (3) ◽  
pp. 415-424 ◽  
Author(s):  
R. Loch Macdonald ◽  
Bryce K. A. Weir ◽  
Tim D. Runzer ◽  
Michael G. A. Grace ◽  
J. Max Findlay ◽  
...  

✓ A primate model was used to determine whether oxyhemoglobin (OxyHb), methemoglobin (MetHb), or bilirubin is likely to be responsible for cerebral vasospasm following subarachnoid hemorrhage (SAH). Forty cynomolgus monkeys were randomly assigned to one of five groups. On Day 0, each animal underwent angiography followed by right craniectomy and placement of an Ommaya reservoir with its catheter adjacent to the right middle cerebral artery (MCA). The animals received intrathecal injections twice a day for 6 days of one of the following solutions: mock cerebrospinal fluid (CSF); OxyHb; MetHb; bilirubin; or supernatant fluid from an incubated mixture of autologous blood and mock CSF. On Day 7, angiography was repeated and the animals were killed. Comparison of angiograms obtained on Day 0 and Day 7 of the experiment showed significant vasospasm of the right MCA and the right anterior cerebral and internal carotid arteries in the animal groups that had received OxyHb or supernatant fluid. There was a smaller reduction in diameter of the same vessels in the bilirubin group (not statistically significant), while no effects were observed in the groups receiving MetHb or mock CSF. Electron microscopy of the right MCA's gave results consistent with the angiographic findings. One monkey in the OxyHb group developed a delayed-onset right MCA infarction. These data suggest that OxyHb is the cause of cerebral vasospasm following SAH.


1979 ◽  
Vol 50 (6) ◽  
pp. 747-757 ◽  
Author(s):  
Roger W. Countee ◽  
Thurairasah Vijayanathan

✓ The authors describe their experiences with five patients with delayed transient ischemic symptoms homolateral to internal carotid artery (ICA) occlusions. In each instance, initial arteriograms were interpreted as showing irreversible occlusions of these arteries in the neck and microvascular bypass procedures were contemplated. However, after repeat arteriographic evaluations with a modified injection technique, certain angiographic features were identified that suggested the mechanism of the symptoms in these patients and that their ICA's could be reconstituted in the neck. This was successfully accomplished in each patient with complete relief of ischemic symptoms. The angiographic technique employed and the arterial flow patterns identified in these patients are discussed. The details of the operative technique are described. It is concluded that routine arteriographic techniques may be inadequate to identify the reversible carotid occlusion. This may account for some of the confusion which surrounds the surgery of these vessels as well as the failure of extracranial-intracranial bypass to relieve recurrent ischemic symptoms in these individuals.


1980 ◽  
Vol 53 (4) ◽  
pp. 566-569 ◽  
Author(s):  
Koichi Matsuo ◽  
Shigeaki Kobayashi ◽  
Kenichiro Sugita

✓ A case of bitemporal hemianopsia in a 68-year-old woman is reported. Surgical exploration revealed bilateral compression of the optic nerves by the arteriosclerotic internal carotid arteries. Microsurgical decompression with unroofing of the optic canals resulted in prompt recovery of the visual fields.


1987 ◽  
Vol 67 (5) ◽  
pp. 765-767 ◽  
Author(s):  
Patrick T. Tracy

✓ The case is presented of a 34-year-old man with subarachnoid hemorrhage from rupture of an anterior communicating artery aneurysm. The magnetic resonance imaging and angiographic findings are reported. Angiography showed the aneurysm plus an unusual anastomosis between the intracavernous portions of both internal carotid arteries. The anastomosis crossed posterior to the base of the dorsum sellae and was associated with absence of the right internal carotid artery and the A1 segment of the right anterior cerebral artery. This is the 11th case of such an unusual intercarotid anastomosis reported in the literature.


1978 ◽  
Vol 48 (6) ◽  
pp. 1026-1029 ◽  
Author(s):  
Antti Servo ◽  
Matti Puranen

✓ A case is reported with occlusion and stenosis of the internal carotid arteries in association with basal telangiectasia. Fifteen years after postoperative irradiation for an optic glioma, radiological signs typical of the moyamoya syndrome were observed. Radiation therapy is discussed as the cause of the vascular damage in this case.


1988 ◽  
Vol 69 (2) ◽  
pp. 263-268 ◽  
Author(s):  
Yuichiro Tanaka ◽  
Shigetoshi Chiba

✓ The interaction between oxyhemoglobin (oxyHb) and 5-hydroxytryptamine (5-HT) was investigated in an experimental model of isolated canine internal and common carotid arteries with insertion of stainless steel cannulae. Extraluminal application of 10−5 M oxyHb induced marked and long-lasting vasoconstriction in the internal carotid but not in the common carotid arteries. The 5-HT-induced vasoconstriction was potentiated significantly in both the internal and common carotid arteries. These potentiations were not influenced by the presence or absence of endothelium, a finding which was confirmed by vascular responses to intraluminal acetylcholine. It is concluded that the interaction between extraluminal oxyHb and intraluminal 5-HT may be one of the possible etiological factors behind the chronic phase of vascular spasm following subarachnoid hemorrhage.


1971 ◽  
Vol 34 (6) ◽  
pp. 823-826 ◽  
Author(s):  
Naim K. Atallah ◽  
Sami I. Nassar

✓ The sources of blood supply of calvarial hemangiomas are not well established angiographically but have been reported to derive from the middle meningeal artery. A case is presented in which both the external and the internal carotid arteries supplied the hemangioma. The protean arteriographic picture is explained in terms of the various tissues the tumor involves (galea, calvarium, or meninges).


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