Angiographic demonstration of congenital absence of both internal carotid arteries

1975 ◽  
Vol 42 (4) ◽  
pp. 478-482 ◽  
Author(s):  
Ivan W. Rosen ◽  
Damon F. Mills ◽  
Harold I. Nadel ◽  
Donald D. Kaiserman
Keyword(s):  
Subarachnoid Hemorrhage ◽  
Blood Supply ◽  
Basilar Artery ◽  
Carotid Arteries ◽  
Internal Carotid ◽  
Content Type ◽  
Total Absence ◽  
Angiographic Demonstration ◽  
Internal Carotid Arteries ◽  
Fine Print

✓ The authors present angiographic demonstration of bilateral, congenital, total absence of the internal carotid arteries in a patient with subarachnoid hemorrhage. Angiography demonstrated complete absence of both internal carotid arteries. The intracranial blood supply had been provided by communication between a hypertrophied basilar artery and the circle of Willis through the posterior communicating arteries. An aneurysm, believed to be the site of the subarachnoid hemorrhage, arose from the basilar artery. Absence of the carotid canals substantiated the congenital nature of the anomaly. The related embryology is reviewed.

Cerebral mycotic aneurysm of fungal origin

1978 ◽  
Vol 49 (1) ◽  
pp. 0107-0110 ◽  
Author(s):  
Gulshan K. Ahuja ◽  
Neeraj Jain ◽  
Malini Vijayaraghavan ◽  
Subimal Roy
Keyword(s):  
Subarachnoid Hemorrhage ◽  
Mycotic Aneurysm ◽  
Carotid Arteries ◽  
Internal Carotid ◽  
Content Type ◽  
History Of ◽  
The Right ◽  
Internal Carotid Arteries ◽  
Fine Print

✓ A young man who had a long history of sinusitis developed subarachnoid hemorrhage and died. Autopsy showed a mycotic aneurysm of fungal origin at the junction of the right posterior cerebral and internal carotid arteries. Four of five reported cases of fungal aneurysm were due to Aspergillus infection.

Occurrence of severe vasospasm following intraventricular hemorrhage from an arteriovenous malformation

1997 ◽  
Vol 87 (3) ◽  
pp. 436-439 ◽  
Author(s):  
Keiichiro Maeda ◽  
Hiroki Kurita ◽  
Tsuneo Nakamura ◽  
Masaaki Usui ◽  
Kazuo Tsutsumi ◽  
...  
Keyword(s):  
Subarachnoid Hemorrhage ◽  
Intraventricular Hemorrhage ◽  
Arteriovenous Malformations ◽  
Carotid Arteries ◽  
Internal Carotid ◽  
The Other ◽  
Content Type ◽  
Radiological Features ◽  
Internal Carotid Arteries ◽  
Fine Print

✓ The authors present two rare cases of severe cerebral vasospasm following the rupture of arteriovenous malformations (AVMs). Computerized tomography revealed intracerebral hemorrhage in the thalamus in one case and in the putamen in the other, both accompanied by cast formation of intraventricular clots without radiological evidence of subarachnoid hemorrhage. Initial angiograms showed arterial narrowing of the bilateral internal carotid arteries in the supraclinoid portion but failed to demonstrate an arteriovenous shunt. Subsequent angiograms clearly demonstrated the existence of an AVM. Radiological features and possible mechanisms are discussed.

Calvarial hemangioma with blood supply from branches of the internal carotid artery

1971 ◽  
Vol 34 (6) ◽  
pp. 823-826 ◽  
Author(s):  
Naim K. Atallah ◽  
Sami I. Nassar
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Blood Supply ◽  
Carotid Arteries ◽  
Internal Carotid ◽  
Middle Meningeal Artery ◽  
Content Type ◽  
Internal Carotid Arteries ◽  
Meningeal Artery ◽  
Fine Print

✓ The sources of blood supply of calvarial hemangiomas are not well established angiographically but have been reported to derive from the middle meningeal artery. A case is presented in which both the external and the internal carotid arteries supplied the hemangioma. The protean arteriographic picture is explained in terms of the various tissues the tumor involves (galea, calvarium, or meninges).

Embolism from intracranial aneurysms

1980 ◽  
Vol 53 (3) ◽  
pp. 300-304 ◽  
Author(s):  
Toshisuke Sakaki ◽  
Kazuhiko Kinugawa ◽  
Tatsuo Tanigake ◽  
Seiji Miyamoto ◽  
Kikuo Kyoi ◽  
...  
Keyword(s):  
Intracranial Aneurysms ◽  
Carotid Arteries ◽  
Internal Carotid ◽  
Cerebral Arteries ◽  
Great Care ◽  
Content Type ◽  
Middle Cerebral Arteries ◽  
Internal Carotid Arteries ◽  
Distal Artery ◽  
Fine Print

✓ Embolism from an aneurysm is one of the mechanisms involved in the pathogenesis of ischemic symptoms associated with intracranial aneurysms. Four cases are reported in which aneurysms of the internal carotid arteries and middle cerebral arteries were the source of emboli resulting in cerebral infarction. In the treatment of these aneurysms, it is best to clip the neck of the aneurysm with great care to avoid embolism due to extrusion of clot into the distal artery.

Reconstitution of “totally” occluded internal carotid arteries

1979 ◽  
Vol 50 (6) ◽  
pp. 747-757 ◽  
Author(s):  
Roger W. Countee ◽  
Thurairasah Vijayanathan
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Carotid Arteries ◽  
Internal Carotid ◽  
Injection Technique ◽  
Complete Relief ◽  
Content Type ◽  
Internal Carotid Arteries ◽  
Angiographic Technique ◽  
Fine Print

✓ The authors describe their experiences with five patients with delayed transient ischemic symptoms homolateral to internal carotid artery (ICA) occlusions. In each instance, initial arteriograms were interpreted as showing irreversible occlusions of these arteries in the neck and microvascular bypass procedures were contemplated. However, after repeat arteriographic evaluations with a modified injection technique, certain angiographic features were identified that suggested the mechanism of the symptoms in these patients and that their ICA's could be reconstituted in the neck. This was successfully accomplished in each patient with complete relief of ischemic symptoms. The angiographic technique employed and the arterial flow patterns identified in these patients are discussed. The details of the operative technique are described. It is concluded that routine arteriographic techniques may be inadequate to identify the reversible carotid occlusion. This may account for some of the confusion which surrounds the surgery of these vessels as well as the failure of extracranial-intracranial bypass to relieve recurrent ischemic symptoms in these individuals.

Bitemporal hemianopsia associated with sclerosis of the intracranial internal carotid arteries

1980 ◽  
Vol 53 (4) ◽  
pp. 566-569 ◽  
Author(s):  
Koichi Matsuo ◽  
Shigeaki Kobayashi ◽  
Kenichiro Sugita
Keyword(s):  
Carotid Arteries ◽  
Internal Carotid ◽  
Visual Fields ◽  
Optic Nerves ◽  
Content Type ◽  
Surgical Exploration ◽  
Microsurgical Decompression ◽  
Internal Carotid Arteries ◽  
Fine Print

✓ A case of bitemporal hemianopsia in a 68-year-old woman is reported. Surgical exploration revealed bilateral compression of the optic nerves by the arteriosclerotic internal carotid arteries. Microsurgical decompression with unroofing of the optic canals resulted in prompt recovery of the visual fields.

Moyamoya syndrome as a complication of radiation therapy

1978 ◽  
Vol 48 (6) ◽  
pp. 1026-1029 ◽  
Author(s):  
Antti Servo ◽  
Matti Puranen
Keyword(s):  
Radiation Therapy ◽  
Carotid Arteries ◽  
Internal Carotid ◽  
Vascular Damage ◽  
Optic Glioma ◽  
Moyamoya Syndrome ◽  
Content Type ◽  
Postoperative Irradiation ◽  
Internal Carotid Arteries ◽  
Fine Print

✓ A case is reported with occlusion and stenosis of the internal carotid arteries in association with basal telangiectasia. Fifteen years after postoperative irradiation for an optic glioma, radiological signs typical of the moyamoya syndrome were observed. Radiation therapy is discussed as the cause of the vascular damage in this case.

Potentiating effects of extraluminal oxyhemoglobin to intraluminal 5-hydroxytryptamine in isolated canine internal carotid arteries

1988 ◽  
Vol 69 (2) ◽  
pp. 263-268 ◽  
Author(s):  
Yuichiro Tanaka ◽  
Shigetoshi Chiba
Keyword(s):  
Carotid Arteries ◽  
Internal Carotid ◽  
Chronic Phase ◽  
Content Type ◽  
Vascular Responses ◽  
The Common ◽  
Vascular Spasm ◽  
Common Carotid Arteries ◽  
Internal Carotid Arteries ◽  
Fine Print

✓ The interaction between oxyhemoglobin (oxyHb) and 5-hydroxytryptamine (5-HT) was investigated in an experimental model of isolated canine internal and common carotid arteries with insertion of stainless steel cannulae. Extraluminal application of 10−5 M oxyHb induced marked and long-lasting vasoconstriction in the internal carotid but not in the common carotid arteries. The 5-HT-induced vasoconstriction was potentiated significantly in both the internal and common carotid arteries. These potentiations were not influenced by the presence or absence of endothelium, a finding which was confirmed by vascular responses to intraluminal acetylcholine. It is concluded that the interaction between extraluminal oxyHb and intraluminal 5-HT may be one of the possible etiological factors behind the chronic phase of vascular spasm following subarachnoid hemorrhage.

Fibromuscular hyperplasia of the extracranial internal carotid artery

1971 ◽  
Vol 34 (5) ◽  
pp. 647-651 ◽  
Author(s):  
Francesco Galligioni ◽  
Giorgio Iraci ◽  
Gino Marin
Keyword(s):  
Differential Diagnosis ◽  
Arterial Wall ◽  
Carotid Arteries ◽  
Internal Carotid ◽  
Extracranial Internal Carotid Artery ◽  
Content Type ◽  
Fibromuscular Hyperplasia ◽  
Internal Carotid Arteries ◽  
Angiographic Findings ◽  
Fine Print

✓ Fibromuscular hyperplasia is an alteration of the arterial wall, affecting mostly middle-aged women. When localized to the internal carotid arteries, it can give symptoms of intermittent or permanent cerebral ischemia, but is usually asymptomatic. Three cases, all of them accidental angiographic findings, are used as a basis for discussion of the angiographic features, differential diagnosis, and surgical treatment.

Resolution of occlusion in spontaneously dissected carotid arteries

1982 ◽  
Vol 56 (6) ◽  
pp. 857-860 ◽  
Author(s):  
Eugenio Pozzati ◽  
Giulio Gaist ◽  
Massimo Poppi
Keyword(s):  
Carotid Arteries ◽  
Internal Carotid ◽  
Artery Occlusion ◽  
Carotid Artery Occlusion ◽  
Spontaneous Resolution ◽  
Transient Ischemic Attacks ◽  
Content Type ◽  
Antiplatelet Aggregation ◽  
Spontaneous Dissection ◽  
Fine Print

✓ Two cases of internal carotid artery occlusion secondary to spontaneous dissection are reported. Both patients presented with transient ischemic attacks. Both had antiplatelet aggregation therapy, followed by spontaneous resolution of the occlusion. The period of healing seems to be relatively short. In both cases, restoration of flow was angiographically documented 14 days and 10 weeks after the initial arteriogram. Strategies for treatment of such patients are discussed.

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