scholarly journals KIR3DL1-Negative CD8 T Cells and KIR3DL1-Negative Natural Killer Cells Contribute to the Advantageous Control of Early Human Immunodeficiency Virus Type 1 Infection in HLA-B Bw4 Homozygous Individuals

2018 ◽  
Vol 9 ◽  
Author(s):  
Xin Zhang ◽  
Xiaofan Lu ◽  
Christiane Moog ◽  
Lin Yuan ◽  
Zhiying Liu ◽  
...  
Keyword(s):  
Human Immunodeficiency Virus ◽  
T Cells ◽  
Natural Killer Cells ◽  
Natural Killer ◽  
Human Immunodeficiency Virus Type ◽  
Virus Type ◽  
Cd8 T Cells ◽  
Immunodeficiency Virus ◽  
Early Human

scholarly journals Lysis of Human Immunodeficiency Virus Type 1 Antigen-Expressing Cells by CD4 and CD8 T Cells Ex Vivo

1996 ◽  
Vol 174 (1) ◽  
pp. 209-213 ◽  
Author(s):  
M. Heinkelein ◽  
I. Euler-Konig ◽  
H. Klinker ◽  
H. Ruckle-Lanz ◽  
C. Jassoy
Keyword(s):  
Human Immunodeficiency Virus ◽  
T Cells ◽  
Human Immunodeficiency Virus Type ◽  
Virus Type ◽  
Cd8 T Cells ◽  
Ex Vivo ◽  
Immunodeficiency Virus

scholarly journals Virus-Specific Interleukin-17-Producing CD4+ T Cells Are Detectable in Early Human Immunodeficiency Virus Type 1 Infection

2008 ◽  
Vol 82 (13) ◽  
pp. 6767-6771 ◽  
Author(s):  
Feng Yun Yue ◽  
Asad Merchant ◽  
Colin M. Kovacs ◽  
Mona Loutfy ◽  
Desmond Persad ◽  
...  
Keyword(s):  
Human Immunodeficiency Virus ◽  
T Cells ◽  
Human Immunodeficiency Virus Type ◽  
Virus Type ◽  
Interleukin 17 ◽  
Immunodeficiency Virus ◽  
Early Human ◽  
Hiv 1

ABSTRACT TH-17 cells have been shown to play a role in bacterial defense, acute inflammation, and autoimmunity. We examined the role of interleukin 17 (IL-17) production in human immunodeficiency virus type 1 (HIV-1) infection. Both HIV-1- and cytomegalovirus (CMV)-specific IL-17-producing CD4+ T cells were detectable in early HIV-1 infection but were reduced to nondetectable levels in chronic and nonprogressive HIV-1 infection. IL-17-producing CMV-specific cells were not detected in blood from HIV-1-uninfected normal volunteers. Virus-specific TH-17 cells could coexpress other cytokines and could express CCR4 or CXCR3. Although the etiology of these cells has yet to be established, we propose that microbial translocation may induce them.

Sign in / Sign up

Export Citation Format

Share Document