An atypical case of compressive optic neuropathy and cranial nerve 6th palsy caused by a cholesterol granuloma

2021 ◽  
Vol 3 (1) ◽  
Keyword(s):  
Optic Neuropathy ◽  
Cranial Nerve ◽  
Cholesterol Granuloma ◽  
Compressive Optic Neuropathy ◽  
Atypical Case

scholarly journals Compressive Optic Neuropathy Caused by a Cholesterol Granuloma of the Sphenoidal Sinus

2011 ◽  
Vol 35 (2) ◽  
pp. 78-80
Author(s):  
Niranjan Pehere ◽  
C. Anjaneyulu ◽  
Ruchi Mittal ◽  
Geeta Vemuganti
Keyword(s):  
Optic Neuropathy ◽  
Cholesterol Granuloma ◽  
Compressive Optic Neuropathy ◽  
Sphenoidal Sinus

A case of compressive optic neuropathy caused by sphenoid sinus cholesterol granuloma

2009 ◽  
Vol 53 (4) ◽  
pp. 441-442 ◽  
Author(s):  
Duck Jin Hwang ◽  
Yun Suk Chung ◽  
Sun Young Jun ◽  
Yun Jeong Kim ◽  
Joo Yeon Lee ◽  
...  
Keyword(s):  
Optic Neuropathy ◽  
Sphenoid Sinus ◽  
Cholesterol Granuloma ◽  
Compressive Optic Neuropathy

Bilateral dysthyroid compressive optic neuropathy responsive to teprotumumab

2021 ◽  
pp. 112067212199104
Author(s):  
Catherine J Hwang ◽  
Erin E Nichols ◽  
Brian H Chon ◽  
Julian D Perry
Keyword(s):  
Side Effects ◽  
Optic Neuropathy ◽  
Surgical Decompression ◽  
Thyroid Eye Disease ◽  
Eye Disease ◽  
High Dose ◽  
Compressive Optic Neuropathy ◽  
Traditional Management ◽  
Immune Mediated ◽  
Orbital Radiation

Thyroid eye disease is an auto-immune mediated orbitopathy which can cause dysthyroid compressive optic neuropathy. Traditional management of active thyroid eye disease includes temporizing high-dose steroids, orbital radiation and surgical decompression, which each possess significant limitations and/or side effects. Teprotumumab is an IGF-IR inhibitor recently FDA-approved for active thyroid eye disease. The authors report reversal of bilateral dysthyroid compressive optic neuropathy managed medically utilizing teprotumumab.

scholarly journals Compressive Optic Neuropathy Caused by Orbital Non-Hodgkin's Lymphoma

2012 ◽  
Vol 2012 ◽  
pp. 1-3
Author(s):  
Mohammed M. Ziaei ◽  
Hadi Ziaei
Keyword(s):  
Optic Nerve ◽  
Paranasal Sinus ◽  
Optic Neuropathy ◽  
Hodgkin’S Lymphoma ◽  
Visual Loss ◽  
Hodgkin's Lymphoma ◽  
Compressive Optic Neuropathy ◽  
Acute Visual Loss ◽  
Non Hodgkin’S Lymphoma ◽  
Non Hodgkin's Lymphoma

Purpose. To present a unique case of Non-Hodgkin’s-Lymphoma- (NHL) associated compressive optic neuropathy.Method. An 89-year-old male presenting with acute unilateral visual loss and headache.Results. Patient was initially diagnosed with occult giant cell arteritis; however after visual acuity deteriorated despite normal inflammatory markers, an urgent MRI scan revealed an extensive paranasal sinus mass compressing the optic nerve.Conclusion. Paranasal sinus malignancies occasionally present to the ophthalmologist with signs of optic nerve compression and must be included in the differential diagnosis of acute visual loss.

scholarly journals Amaurosis by Traumatic Optic Neuropathy direct secondary to the “transorbitario” foreign body: Report of an atypical case

2018 ◽  
Vol 77 (6) ◽  
Author(s):  
Abelardo de Souza Couto Junior ◽  
Joaquim Ferreira de Paula ◽  
Richard Raphael Borges Tavares Vieira ◽  
Guilherme da Silva Guimarães Júnior ◽  
Jhonatan Oliveira Lucateli ◽  
...  
Keyword(s):  
Foreign Body ◽  
Optic Neuropathy ◽  
Traumatic Optic Neuropathy ◽  
Atypical Case

scholarly journals Tongue and tail necrosis in an atypical case of acute steroid responsive meningitis-arteritis in a dog

2012 ◽  
Vol 81 (2) ◽  
pp. 93-97
Author(s):  
S. A. E. Van Meervenne ◽  
J. Declercq ◽  
A. Tipold ◽  
K. Chiers ◽  
I. Van Soens ◽  
...  
Keyword(s):  
Case Report ◽  
Neck Pain ◽  
Cranial Nerve ◽  
Clinical Symptoms ◽  
Systemic Vasculitis ◽  
Neurological Deficits ◽  
Calcinosis Cutis ◽  
Atypical Case ◽  
Acute Form ◽  
Multiple Cranial Nerve

Acute steroid responsive meningitis-arteritis (SRMA) is a common neurological disorder in young dogs. Typical clinical symptoms of the acute form of SRMA are neck pain, depression and fever. This case report describes a 1.5-year-old Pointer with uncommon neurological deficits (unilateral multiple cranial nerve deficits and Horner’s syndrome) and an exceptional necrosis of the tongue. This was believed to be part of the systemic vasculitis accompanying SRMA. The patient also developed tail necrosis and iatrogenic calcinosis cutis, which complicated further treatment of the dog.

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