scholarly journals Pulmonary Embolism and Wilms’ Tumor. Can It Be Any Worse? Case Report

2021 ◽  
Vol 6 (2) ◽  
pp. 10-16
Author(s):  
Abdulrahaman Al Zhran ◽  
Abdullah Baothman

It is common for Wilms tumor to invade the inferior vena cava and right atrium, but a massive pulmonary embolism is rare. This case report describes an 8-year-old male patient who presented to an outpatient clinic with mild pain associated with subjective fever for 1 week. A left renal mass with extension of an inferior vena cava thrombus was seen on abdominal computed tomography, suggesting a Wilms tumor. An exploratory laparotomy biopsy was scheduled, but not completed because the patient’s health suddenly deteriorated, and he died despite high-quality cardiopulmonary resuscitation. A massive pulmonary embolism was found to cause a sudden and fatal cardiac arrest.  

2020 ◽  
Vol 73 ◽  
pp. 176-178
Author(s):  
Sarah Winterland ◽  
Tim Reese ◽  
Georgios Makridis ◽  
Karl J. Oldhafer

2020 ◽  
Vol 68 ◽  
pp. 95-97
Author(s):  
Tiziana Cena ◽  
Simona Bazzano ◽  
Paola Berni ◽  
Gian Luca Vignazia ◽  
Francesca Grossi ◽  
...  

2020 ◽  
Author(s):  
Mohamed Osman Jeele ◽  
Mohamed Farah Yusuf Mohamud ◽  
Rukia Omar Barre Addow

Abstract Nephrotic syndrome (NS); the first described in 1827 as the presence of proteinuria of ≥ 3.5 g/24 hours, albuminemia < 3.0 g, peripheral edema, hyperlipidemia, lipiduria, and increased thrombotic risk which has an incidence of three new cases per 100 000 each year in adults. Nephrotic syndrome had complicated by the induction of a hypercoagulable state with both various venous and arterial thromboembolic events. The pathophysiology of hypercoagulability in the nephrotic syndrome is imbalances of prothrombotic and antithrombotic factors, as well as impaired thrombolytic activity occurs. Here we present, a 19 years old female came to the emergency department with a complaint of chest pain and shortness of breath for three days. The patient quickly diagnosed pulmonary embolism and inferior vena cava thrombosis as a complication of nephrotic syndrome, allowing prompt initiation of anticoagulant therapy. After two weeks of admission, the patient's resolved dramatically, and his laboratory results returned to almost normal, and the patient discharged with Oral Prednisolone, Coumadin, Atorvastatin, and Ramipril. We aim to determine which is the likely cause of pulmonary embolism in patients with the nephrotic syndrome.


2006 ◽  
Vol 20 (1) ◽  
pp. 60-65
Author(s):  
Tomoaki Hirose ◽  
Keiji Kushibe ◽  
Makoto Takahama ◽  
Takeshi Kawaguchi ◽  
Michitaka Kimura ◽  
...  

2021 ◽  
Vol 14 (1) ◽  
Author(s):  
Mohamed Osman Omar Jeele ◽  
Rukia Omar Barei Addow ◽  
Mohamed Farah Yusuf Mohamud

AbstractNephrotic syndrome (NS) was first described in 1827 as the presence of proteinuria of ≥ 3.5 g/24 h, hypoalbuminemia < 3.0 g/dl, peripheral edema, hyperlipidemia, lipiduria, and increased thrombotic risk. Nephrotic syndrome has an incidence of three cases per 100,000 each year in adults. Nephrotic syndrome also has serious complications due to hypercoagulable state in both various venous and arteries which could lead thromboembolic events. The pathophysiology of hypercoagulability in the nephrotic syndrome is due to an imbalance of prothrombotic and antithrombotic factors, as well as impaired thrombolytic activities.Here, we will present a 19-year-old woman who presented to the emergency department complaining of chest pain and shortness of breath for 3 days. The patient was quickly diagnosed with pulmonary embolism and inferior vena cava thrombosis as a complication of nephrotic syndrome, allowing prompt initiation of anticoagulant therapy. After 2 weeks of admission, the patient’s condition resolved, her laboratory results returned to almost normal and the patient was discharged with oral prednisolone, coumadin, atorvastatin, and ramipril. We aim to determine which is the likely cause of pulmonary embolism in patients with nephrotic syndrome.


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