Induction and Repression of Gene Expression Mediating Ethylene Biosynthesis and Sodium/Proton Exchange in Rice Seedlings Under Submergence Stress

2012 ◽  
Vol 26 (3) ◽  
pp. 2945-2951
Author(s):  
Hien Thi Thu Vu ◽  
Oliver Escano Manangkil ◽  
Naoki Mori ◽  
Shinya Yoshida ◽  
Chiharu Nakamura
Keyword(s):  
Gene Expression ◽  
Ethylene Biosynthesis ◽  
Proton Exchange ◽  
Rice Seedlings ◽  
Submergence Stress ◽  
Sodium Proton Exchange

Sodium/Proton Exchange in the Maintenance of Intracellular pH in FRTL-5 Thyroid Cells*

Endocrinology ◽  
1988 ◽  
Vol 123 (4) ◽  
pp. 1705-1711 ◽  
Author(s):  
CLAUDIO MARCOCCI ◽  
EVELYN F. GROLLMAN
Keyword(s):  
Intracellular Ph ◽  
Proton Exchange ◽  
Thyroid Cells ◽  
Sodium Proton Exchange

Pancreatic beta-cell electrical activity: the role of anions and the control of pH

1983 ◽  
Vol 244 (3) ◽  
pp. C188-C197 ◽  
Author(s):  
G. T. Eddlestone ◽  
P. M. Beigelman
Keyword(s):  
Membrane Potential ◽  
Cell Membrane ◽  
Beta Cell ◽  
Pancreatic Beta Cell ◽  
Proton Exchange ◽  
Disulfonic Acid ◽  
Control Mechanisms ◽  
Exchange System ◽  
Sodium Proton Exchange

The influence of chloride on the mouse pancreatic beta-cell membrane potential and the cell membrane mechanisms controlling intracellular pH (pHi) have been investigated using glass microelectrodes to monitor the membrane potential. It has been shown that chloride is distributed passively across the beta-cell membrane such that chloride potential is equal to the membrane potential. Withdrawal of perifusate chloride or bicarbonate and the application of the drugs 4-acetamido-4'-isethiocyanostilbene-2,2'-disulfonic acid (SITS) and probenecid, both blockers of transmembrane anion movement, have been used to establish that a chloride-bicarbonate exchange system is operative in the cell membrane and that it is one of the control mechanisms of pHi. Amiloride, a specific blocker of the transmembrane sodium proton exchange, has been used to demonstrate that this mechanism is also operative in the beta-cell membrane in the control of pHi. The hypothesis that the calcium-activated potassium permeability is proton sensitive at an intracellular site, a fall in pHi causing a fall in permeability and an increase in pHi causing an increase in permeability, has been used to explain many of the effects observed in this study.

scholarly journals Defective jejunal brush border membrane sodium/proton exchange in association with lethal familial protracted diarrhoea.

Gut ◽  
1990 ◽  
Vol 31 (10) ◽  
pp. 1156-1158 ◽  
Author(s):  
K M Keller ◽  
S Wirth ◽  
W Baumann ◽  
D Sule ◽  
I W Booth
Keyword(s):  
Brush Border ◽  
Brush Border Membrane ◽  
Proton Exchange ◽  
Sodium Proton Exchange
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