Mıld Asymptomatıc Hyperparathyroıdısm

10.5580/24c2 ◽  
2011 ◽  
Vol 6 (2) ◽  
Keyword(s):  
Asymptomatic Hyperparathyroidism

scholarly journals Mild asymptomatic hyperparathyroidism.

BMJ ◽  
1980 ◽  
Vol 281 (6234) ◽  
pp. 174-175 ◽  
Keyword(s):  
Asymptomatic Hyperparathyroidism

ASYMPTOMATIC HYPERPARATHYROIDISM 30 YEARS AFTER PARATHYROIDECTOMY

The Lancet ◽  
1968 ◽  
Vol 291 (7545) ◽  
pp. 757 ◽  
Author(s):  
C FRIDERICHSEN
Keyword(s):  
Asymptomatic Hyperparathyroidism

The incidence of nephrolithiasis and osteoporosis in patients with asymptomatic hyperparathyroidism

2020 ◽  
Author(s):  
Antonis Polymeris ◽  
Nikolaos Kalogeris ◽  
Apostolos Lafkas ◽  
Nektaria Papanikola ◽  
Eleni Herolidi ◽  
...  
Keyword(s):  
Asymptomatic Hyperparathyroidism

Management of asymptomatic hyperparathyroidism

1984 ◽  
Vol 147 (4) ◽  
pp. 498-502 ◽  
Author(s):  
Randall D. Gaz ◽  
Chiu-an Wang
Keyword(s):  
Asymptomatic Hyperparathyroidism

Asymptomatic hyperparathyroidism

1981 ◽  
Vol 2 (9) ◽  
pp. 452-452
Author(s):  
P. J. Phillips ◽  
A. R. Robertson ◽  
A. G. Need ◽  
P. E. Harding
Keyword(s):  
Asymptomatic Hyperparathyroidism

Asymptomatic Hyperparathyroidism

JAMA ◽  
1977 ◽  
Vol 237 (20) ◽  
pp. 2186
Author(s):  
Elliott M. Antman
Keyword(s):  
Asymptomatic Hyperparathyroidism

scholarly journals The syndrome of gastric carcinoid and hyperparathyroidism: a family study and literature review

2009 ◽  
Vol 160 (4) ◽  
pp. 689-694 ◽  
Author(s):  
C Christopoulos ◽  
V Balatsos ◽  
E Rotas ◽  
I Karoumpalis ◽  
D Papavasileiou ◽  
...  
Keyword(s):  
Molecular Genetic ◽  
Gene Mutations ◽  
Pernicious Anaemia ◽  
Chronic Atrophic Gastritis ◽  
Gastric Carcinoid ◽  
Men1 Syndrome ◽  
Large Deletions ◽  
Asymptomatic Hyperparathyroidism ◽  
Age Range

ObjectiveTo present evidence supporting the hypothesis that the coexistence of gastric carcinoids (GCs) and hyperparathyroidism may represent a distinct clinical entity, not related to multiple endocrine neoplasia type 1 (MEN1).MethodsWe studied a cohort of five young siblings (age range 26–42 years), one of whom had been found to have GC and hyperparathyroidism. All siblings underwent serial gastroscopies for the assessment of gastric neuroendocrine cell proliferations over a mean follow-up period of 31.2 months. Imaging, biochemical and hormonal as well as molecular genetic investigations were performed in the direction of MEN1 syndrome. The literature was searched for cases with coexistence of GCs and hyperparathyroidism not associated with MEN1.ResultsFour of the siblings, all male, were found to have GCs in a background of Helicobacter pylori-associated chronic atrophic gastritis and pernicious anaemia, with no serological evidence of gastric autoimmunity. In two of them, asymptomatic hyperparathyroidism was also present. Screening for MEN1 gene mutations or large deletions was negative, and hormone and imaging investigations did not support a diagnosis of familial MEN1 syndrome. A literature search revealed sporadic reports of cases with GC and hyperparathyroidism not attributable to MEN1.ConclusionsThe association of GCs and hyperparathyroidism appears to constitute a distinct syndrome that can be encountered in genetically predisposed individuals, and should not be regarded as ‘atypical’ or ‘incomplete’ expression of MEN1. Its prevalence and aetiology should be the subject of future studies. Screening for hyperparathyroidism seems to be justified in patients with GC of any type.

scholarly journals Hyperparathyroidism due to Parathyroid Adenoma with Co-existing Vitamin D Deficiency – A Case Report

2016 ◽  
Vol 5 (2) ◽  
pp. 104-106
Author(s):  
Md Raziur Rahman ◽  
Mohammad Abdul Hannan ◽  
Samira Rahat Afroze ◽  
Sultana Marufa Shefin ◽  
Muhammad Abdur Rahim ◽  
...  
Keyword(s):  
Vitamin D ◽  
Case Report ◽  
Primary Hyperparathyroidism ◽  
Secondary Hyperparathyroidism ◽  
Parathyroid Adenoma ◽  
Vitamin D Deficiency ◽  
Musculoskeletal Symptoms ◽  
Asymptomatic Hyperparathyroidism ◽  
Routine Investigations

Primary hyperparathyroidism is often asymptomatic, may be detected during routine investigations or may present with features of hypercalcaemia. Vitamin D deficiency causing secondary hyperparathyroidism presents with musculoskeletal symptoms. Studies have shown an association between primary hyperparathyroidism and vitamin D deficiency. It has also been observed that when co-exist, these two conditions contribute to each other’s disease presentation and severity. This case report depicts initial asymptomatic hyperparathyroidism developing symptoms due to co-existing parathyroid adenoma and vitamin D deficiency in a 26-year-old muslim Bangladeshi female who responded well to adequate pre, per and post parathyroidectomy medical managementBirdem Med J 2015; 5(2): 104-106

Sign in / Sign up

Export Citation Format

Share Document