Verapamil for Control of Ventricular Rate in Paroxysmal Supraventricular Tachycardia and Atrial Fibrillation or Flutter

1981 ◽  
Vol 94 (1) ◽  
pp. 1 ◽  
Author(s):  
HARVEY L. WAXMAN
PEDIATRICS ◽  
1983 ◽  
Vol 72 (2) ◽  
pp. 245-246
Author(s):  
Thomas W. Rowland

Verapamil has gained increasing acceptance as a first-line drug in the treatment of paroxysmal supraventricular tachycardia in both adults1 and children.2-4 Conversion to sinus rhythm in more than 90% of cases has been reported, an effect related to the drug's prolongation of refractory period and conduction through the atrioventricular node. The electrophysiologic effects of verapamil on accessory atrioventricular conduction pathways are more variable, and in some patients the drug will cause a decrease in refractory period.5-7 In individuals with reentrant paroxysmal supraventricular tachycardia this action is inconsequential, but in those with ventricular preexcitation accompanied by atrial fibrillation or flutter the effect may be catastrophic.


1986 ◽  
Vol 57 (8) ◽  
pp. 571-575 ◽  
Author(s):  
Steven F. Roark ◽  
Elizabeth A. McCarthy ◽  
Kerry L. Lee ◽  
Edward L.C. Pritchett

1995 ◽  
Vol 25 (5) ◽  
pp. 984-988 ◽  
Author(s):  
Mark E. Hamer ◽  
William E. Wilkinson ◽  
Walter K. Clair ◽  
Richard L. Page ◽  
Elizabeth A. McCarthy ◽  
...  

2020 ◽  
Vol 4 (1) ◽  
pp. 61-64
Author(s):  
M.R. Khidoyatova ◽  
◽  
G.Sh. Khamraeva

The spread of coronavirus infection poses a particular danger concerningcardiovascular lesions. In the situation of a pandemic caused by coronavirus infection, one of the available and informative methods for diagnosing cardiac disorders in patients with COVID-19 is electrocardiography. According to our observations, we described two cases of COVID-19, which showed different ECG manifestations, which caused a worsening of the course of the disease. In thefirst case, a paroxysm of atrial fibrillation was presented, in the second, paroxysmal supraventricular tachycardia. The mechanisms underlying these ECG abnormalities in severe COVID-19 may be associated with hypoxia and inflammatory damage caused by the virus


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