Anomalous Origin of Left Coronary Artery with Left Anterior Descending Artery Connected to Left Pulmonary Artery from Single Right Coronary Artery

2013 ◽  
Vol 62 (3) ◽  
Author(s):  
s Williams-Phillips
2019 ◽  
Vol 47 (6) ◽  
pp. 2687-2693
Author(s):  
QiongYa Qiu ◽  
JinXiu Yang ◽  
XingXiang Wang

A 61-year-old Chinese man presented with a nearly 30-year history of an anomalous origin of the left coronary artery. He had been diagnosed with an anomalous origin of the left coronary artery in 1989. He then underwent regular echocardiographic examinations and it was found that his heart was gradually enlarging. After a >20-year asymptomatic period, he developed recurrent chest discomfort and palpitation. Coronary computed tomography angiography suggested that the left coronary artery anomaly originated from the pulmonary artery; additionally, the right coronary artery was tortuous and thickened. Coronary angiography showed that the right coronary artery was huge and buckling. The patient underwent corrective surgery of the anomalous origin of the left coronary artery from the pulmonary artery, aortic valve mechanical valve replacement, mitral valve plasty, and tricuspid valve plasty in Fuwai Hospital (National Center of Cardiovascular Disease of China), and the anatomic results of the surgery were good.


PEDIATRICS ◽  
1958 ◽  
Vol 22 (5) ◽  
pp. 909-909

The majority of infants born with an anomalous origin of the left coronary artery from the pulmonary artery die within the first year of life. This malformation produces a clinical picture of episodes of sweating, cyanosis and pallor usually following feeding. Associated findings are gross cardiac enlargement and electrocardiographic evidence of myocardial ischemia. Perfusion studies at necropsy indicate that extensive anastomoses allow free passage of blood from the normal right coronary artery to the left. There now appears evidence that flow through the anomalous left coronary artery is into the pulmonary artery rather than away from it. Thus although there is oxygenated blood in the left coronary artery, it does not reach the myocardium but passes instead to the low-resistance circuit of the pulmonary artery. Ligation of the aberrant vessel at its origin would prevent retrograde flow and allow perfusion of the left ventricle by blood supplied through the anastomoses from the right coronary artery. This would in effect convert the anomaly into that of a single coronary artery arising from the aorta, which situation is usually compatible with a normal life expectancy. Preliminary experience with this suggested surgical treatment appears encouraging in one of the cases reported.


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