blood flow intensity
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2020 ◽  
Vol 2 (3) ◽  
pp. 141-147
Author(s):  
Eva Johanne Leknes Jensen ◽  
Ellen Aagaard Nohr ◽  
Thomas Scholbach ◽  
Torbjørn Moe Eggebø

2018 ◽  
Vol 4 (87) ◽  
Author(s):  
Kęstutis Bunevičius ◽  
Albinas Grūnovas ◽  
Karolis Tijūnaitis

Research background and hypothesis. Different weights, resistance, scope of work, rest periods, frequency, and performance velocity are used to increase strength in training sessions. The traditional training facility with high resistance can be replaced by low resistance while limiting muscle blood flow. Hypothesis: a single 15-minute 200 mm Hg occlusion pressure can affect physical working capacity and blood flow intensity.Research  aim.  was  to  analyze  changes  in  the  intensity  of  the  calf  muscle  arterial  blood  flow  and  physical working capacity with and without 200 mm Hg pressure occlusion.   Research  methods.  were  dynamometry,  ergometry,  venous  occlusive  plethysmography.  The  control  group included six and experimental group – 12 male athletes in endurance sports. In both groups we recorded arterial blood flow at rest and after 75% of maximum voluntary contraction force (MVC) physical work lifting a weight until complete fatigue. Between the first and second physical workloads in the experimental group we applied 15 min occlusion with 40 mm wide cuff in the groin area.Research results. During the physical load in the control group, arterial blood flow significantly increased, and during recovery it did not reach to baseline. In the experimental group arterial blood flow significantly increased and during recovery it did not reach the baseline. Blood flow intensity both the first and the second physical loads altered analogically. Before the second physical load in the experimental group, 200 mm Hg occlusion had a negative effect on skeletal muscle working capacity compared with the passive rest in the control group.Discussion and conclusions. Occlusion of 200 mm Hg in the groin area reduces arterial blood flow intensity in calf skeletal muscles. Immediately after the removal of 200 mm Hg occlusion, arterial blood flow intensity increases and then decreases to its original value. 200 mm Hg occlusion pressure reduces blood flow intensity in the skeletal muscles.    Before  the  second  physical  load,  200  mm  Hg  occlusion  decreases  skeletal  muscle  working  capacity compared with passive rest in the control group.Keywords: occlusion, physical working capacity, arterial blood flow.


2018 ◽  
Vol 3 (90) ◽  
Author(s):  
Albinas Grūnovas ◽  
Jonas Poderys ◽  
Eugenijus Trinkūnas ◽  
Viktoras Šilinskas

Research  background  and  hypothesis.  Blood  flow  intensity  plays  an  important  role  in  the  recovery  after exercising. Research aim was to compare the effect of passive rest and passive foot movement on calf muscle blood flow applying dosed static physical loads. Research methods. Eighteen adult males were divided into two sub-groups. Participants of the study performed two isometric 30-s workouts at 75% of MVC with 20 minutes interval for the recovery between the workouts. During the first stage one sub-group performed workout and a passive recovery was applied while the subjects of the second sub-group performed passive foot flexion movements. During the second stage the form of recovery was changed.  Arterial  blood  flow  intensity  was  registered  during  venous  occlusion  plethysmography  and  passive  foot  flexion  movements were performed by special mechanical equipment.Research results. The results obtained during the study showed that maximal increase of blood flow registered at 21 second after the workout was (52.0 ± 2.9 ml/min/100 ml), while the application of passive movements before the workout decreased the blood flow intensity (45.0  ± 2.6 ml/min/100 ml). It was significantly (p  < 0.05) lower compared to passive rest. Discussion and conclusions. These effects can be explained by reduced venous filling and increased venous vascular reserve capacity in the calves. The results obtained during this research allow concluding that passive foot flexion manoeuvre applied before the isometric workload faster decreases the blood flow intensity during the  recovery.Keywords: arterial blood flow recovery, isometric physical workout, passive foot movement, passive rest.


2018 ◽  
Vol 1 (108) ◽  
pp. 2-8
Author(s):  
Kęstutis Bunevičius ◽  
Albinas Grunovas ◽  
Jonas Poderys

Background. Occlusion pressure intensity influences the blood flow intensity. Immediately after the cuff pressure is released, reactive hyperaemia occurs. Increased blood flow and nutritive delivery are critical for an anabolic stimulus, such as insulin. The aim of study was to find which occlusion pressure was optimal to increase the highest level of post occlusion reactive hyperaemia. Methods. Participants were randomly assigned into one of the four conditions (n = 12 per group): control group without blood flow restriction, experimental groups with 120; 200 or 300 mmHg occlusion pressure. We used venous occlusion plethysmography and arterial blood pressure measurements. Results. After the onset of 120 and 200 mm Hg pressure occlusion, the blood flow intensity significantly decreased. Occlusion induced hyperaemia increased arterial blood flow intensity 134 ± 11.2% (p < .05) in the group with 120 mmHg, in the group with 200 mmHg it increased 267 ± 10.5% (p < .05), in the group with 300 mmHg it increased 233 ± 10.9% (p < .05). Applied 300 mmHg occlusion from the 12 minute diastolic and systolic arterial blood pressure decreased statistically significantly. Conclusions. Occlusion manoeuvre impacted the vascular vasodilatation, but the peak blood flow registered after occlusion did not relate to applied occlusion pressure. The pressure of 200 mmHg is optimal to impact the high level of vasodilatation. Longer than 12 min 300 mmHg could not be recommended due to the steep decrease of systolic and diastolic blood pressures.


NeuroImage ◽  
1996 ◽  
Vol 3 (3) ◽  
pp. 175-184 ◽  
Author(s):  
Stephan Arndt ◽  
Ted Cizadlo ◽  
Daniel O'Leary ◽  
Sherri Gold ◽  
Nancy C. Andreasen

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