scholarly journals EFFECT OF DIFFERENT OCCLUSION PRESSURE ON PECULIARITIES OF MUSCLE BLOOD FLOW

2018 ◽  
Vol 1 (108) ◽  
pp. 2-8
Author(s):  
Kęstutis Bunevičius ◽  
Albinas Grunovas ◽  
Jonas Poderys
Keyword(s):  
Blood Pressure ◽  
Blood Flow ◽  
Arterial Blood Pressure ◽  
Arterial Blood Flow ◽  
Control Group ◽  
Reactive Hyperaemia ◽  
Occlusion Pressure ◽  
Arterial Blood ◽  
Flow Intensity ◽  
Blood Flow Intensity

Background. Occlusion pressure intensity influences the blood flow intensity. Immediately after the cuff pressure is released, reactive hyperaemia occurs. Increased blood flow and nutritive delivery are critical for an anabolic stimulus, such as insulin. The aim of study was to find which occlusion pressure was optimal to increase the highest level of post occlusion reactive hyperaemia. Methods. Participants were randomly assigned into one of the four conditions (n = 12 per group): control group without blood flow restriction, experimental groups with 120; 200 or 300 mmHg occlusion pressure. We used venous occlusion plethysmography and arterial blood pressure measurements. Results. After the onset of 120 and 200 mm Hg pressure occlusion, the blood flow intensity significantly decreased. Occlusion induced hyperaemia increased arterial blood flow intensity 134 ± 11.2% (p < .05) in the group with 120 mmHg, in the group with 200 mmHg it increased 267 ± 10.5% (p < .05), in the group with 300 mmHg it increased 233 ± 10.9% (p < .05). Applied 300 mmHg occlusion from the 12 minute diastolic and systolic arterial blood pressure decreased statistically significantly. Conclusions. Occlusion manoeuvre impacted the vascular vasodilatation, but the peak blood flow registered after occlusion did not relate to applied occlusion pressure. The pressure of 200 mmHg is optimal to impact the high level of vasodilatation. Longer than 12 min 300 mmHg could not be recommended due to the steep decrease of systolic and diastolic blood pressures.

scholarly journals Influence Of 200 mm Hg Occlusion Pressure on Arterial Blood Flow in Skeletal Muscles and Physical Working Capacity

2018 ◽  
Vol 4 (87) ◽  
Author(s):  
Kęstutis Bunevičius ◽  
Albinas Grūnovas ◽  
Karolis Tijūnaitis
Keyword(s):  
Blood Flow ◽  
Skeletal Muscles ◽  
Arterial Blood Flow ◽  
Physical Working Capacity ◽  
Occlusion Pressure ◽  
Working Capacity ◽  
Arterial Blood ◽  
Flow Intensity ◽  
Experimental Group ◽  
Blood Flow Intensity

Research background and hypothesis. Different weights, resistance, scope of work, rest periods, frequency, and performance velocity are used to increase strength in training sessions. The traditional training facility with high resistance can be replaced by low resistance while limiting muscle blood flow. Hypothesis: a single 15-minute 200 mm Hg occlusion pressure can affect physical working capacity and blood flow intensity.Research  aim.  was  to  analyze  changes  in  the  intensity  of  the  calf  muscle  arterial  blood  flow  and  physical working capacity with and without 200 mm Hg pressure occlusion.   Research  methods.  were  dynamometry,  ergometry,  venous  occlusive  plethysmography.  The  control  group included six and experimental group – 12 male athletes in endurance sports. In both groups we recorded arterial blood flow at rest and after 75% of maximum voluntary contraction force (MVC) physical work lifting a weight until complete fatigue. Between the first and second physical workloads in the experimental group we applied 15 min occlusion with 40 mm wide cuff in the groin area.Research results. During the physical load in the control group, arterial blood flow significantly increased, and during recovery it did not reach to baseline. In the experimental group arterial blood flow significantly increased and during recovery it did not reach the baseline. Blood flow intensity both the first and the second physical loads altered analogically. Before the second physical load in the experimental group, 200 mm Hg occlusion had a negative effect on skeletal muscle working capacity compared with the passive rest in the control group.Discussion and conclusions. Occlusion of 200 mm Hg in the groin area reduces arterial blood flow intensity in calf skeletal muscles. Immediately after the removal of 200 mm Hg occlusion, arterial blood flow intensity increases and then decreases to its original value. 200 mm Hg occlusion pressure reduces blood flow intensity in the skeletal muscles.    Before  the  second  physical  load,  200  mm  Hg  occlusion  decreases  skeletal  muscle  working  capacity compared with passive rest in the control group.Keywords: occlusion, physical working capacity, arterial blood flow.

Regulation of arterial blood pressure in the common green iguana

1975 ◽  
Vol 228 (2) ◽  
pp. 386-391 ◽  
Author(s):  
LA Hohnke
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Blood Flow ◽  
Arterial Pressure ◽  
Arterial Blood Pressure ◽  
Venous Pressure ◽  
Arterial Blood Flow ◽  
Arterial Blood ◽  
Head Up Tilt ◽  
Carotid Arterial

Arterial blood pressure (ABP) responses to graded hemorrhage and passive head-up tilt were studied in restrained, anesthetized, and unanesthetized iguanas. The ABP fell slowly in response to hemorrhage up to a critical deficit of 35 plus or minus 19% of the estimated blood volume; the rate of ABP fall then increased nearly 40-fold to continued hemorrhage. Increased heart rate and decreased femoral arterial blood flow accompanied progressive hemorrhage. Propranolol (2-3 mug/kg) did not appreciably alter arterial pressure-hemorrhage curves but hemorrhage-induced increases in heart rate were diminished nearly 50%. Atropine had little effect on either the blood pressure or heart rate changes induced by hemorrhage. During passive tilts of 0-90 degrees carotid arterial pressure fell 33% before returning to control levels (2 min). Heart rate increased and femoral arterial blood flow and central venous pressure fell in response to head-up tilts. It is concluded that hemorrhage and passive head-up tilting can induce reflex cardiovascular changes that assist ABP regulation in iguanas.

scholarly journals Influence of Passive Foot Flection Movements Applied after Exertion Isometric Workouts on Muscular Blood Flow

2018 ◽  
Vol 3 (90) ◽  
Author(s):  
Albinas Grūnovas ◽  
Jonas Poderys ◽  
Eugenijus Trinkūnas ◽  
Viktoras Šilinskas
Keyword(s):  
Blood Flow ◽  
Muscle Blood Flow ◽  
Venous Occlusion ◽  
Arterial Blood Flow ◽  
Adult Males ◽  
Maximal Increase ◽  
Vascular Reserve ◽  
Arterial Blood ◽  
Flow Intensity ◽  
Blood Flow Intensity

Research  background  and  hypothesis.  Blood  flow  intensity  plays  an  important  role  in  the  recovery  after exercising. Research aim was to compare the effect of passive rest and passive foot movement on calf muscle blood flow applying dosed static physical loads. Research methods. Eighteen adult males were divided into two sub-groups. Participants of the study performed two isometric 30-s workouts at 75% of MVC with 20 minutes interval for the recovery between the workouts. During the first stage one sub-group performed workout and a passive recovery was applied while the subjects of the second sub-group performed passive foot flexion movements. During the second stage the form of recovery was changed.  Arterial  blood  flow  intensity  was  registered  during  venous  occlusion  plethysmography  and  passive  foot  flexion  movements were performed by special mechanical equipment.Research results. The results obtained during the study showed that maximal increase of blood flow registered at 21 second after the workout was (52.0 ± 2.9 ml/min/100 ml), while the application of passive movements before the workout decreased the blood flow intensity (45.0  ± 2.6 ml/min/100 ml). It was significantly (p  < 0.05) lower compared to passive rest. Discussion and conclusions. These effects can be explained by reduced venous filling and increased venous vascular reserve capacity in the calves. The results obtained during this research allow concluding that passive foot flexion manoeuvre applied before the isometric workload faster decreases the blood flow intensity during the  recovery.Keywords: arterial blood flow recovery, isometric physical workout, passive foot movement, passive rest.

The relationship of blood flow velocity fluctuations to intracranial pressure B waves

1992 ◽  
Vol 76 (3) ◽  
pp. 415-421 ◽  
Author(s):  
David W. Newell ◽  
Rune Aaslid ◽  
Renate Stooss ◽  
Hans J. Reulen
Keyword(s):  
Blood Pressure ◽  
Blood Flow ◽  
Intracranial Pressure ◽  
Arterial Blood Pressure ◽  
Wave Amplitude ◽  
Transcranial Doppler Ultrasound ◽  
Normal Subjects ◽  
Content Type ◽  
Arterial Blood ◽  
Flow Fluctuations

✓ Intracranial pressure (ICP) and continuous transcranial Doppler ultrasound signals were monitored in 20 head-injured patients and simultaneous synchronous fluctuations of middle cerebral artery (MCA) velocity and B waves of the ICP were observed. Continuous simultaneous monitoring of MCA velocity, ICP, arterial blood pressure, and expired CO2 revealed that both velocity waves and B waves occurred despite a constant CO2 concentration in ventilated patients and were usually not accompanied by fluctuations in the arterial blood pressure. Additional recordings from the extracranial carotid artery during the ICP B waves revealed similar synchronous fluctuations in the velocity of this artery, strongly supporting the hypothesis that blood flow fluctuations produce the velocity waves. The ratio between ICP wave amplitude and velocity wave amplitude was highly correlated to the ICP (r = 0.81, p < 0.001). Velocity waves of similar characteristics and frequency, but usually of shorter duration, were observed in seven of 10 normal subjects in whom MCA velocity was recorded for 1 hour. The findings in this report strongly suggest that B waves in the ICP are a secondary effect of vasomotor waves, producing cerebral blood flow fluctuations that become amplified in the ICP tracing, in states of reduced intracranial compliance.

Forearm and finger blood flow responses to passive body tilts

1979 ◽  
Vol 46 (2) ◽  
pp. 288-292 ◽  
Author(s):  
Y. A. Mengesha ◽  
G. H. Bell
Keyword(s):  
Blood Pressure ◽  
Blood Flow ◽  
Arterial Blood Pressure ◽  
Vascular Resistance ◽  
Pulse Rate ◽  
Forearm Blood Flow ◽  
Body Tilt ◽  
Finger Blood Flow ◽  
Arterial Blood ◽  
Forearm Vascular Resistance

Ten to fifteen healthy subjects, ages 18--30 yr, were used to assess the correlation of forearm blood flow with graded passive body tilts and vascular resistance and also to discern the relative effects of body tilts on finger blood flow. In the head-up tilts forearm blood flow and arterial blood pressure fell progressively, whereas forearm vascular resistance and pulse rate increased. In the head-down tilts the forearm blood flow and the arterial blood pressure increased, whereas the forearm vascular resistance and pulse rate decreased. These changes were found to be significantly correlated with the different tilt angles and with one another. In a preliminary study it was found that infrared heating of the carpometacarpal region produced finger vasodilatation similar to the forearm vasodilatation observed by Crockford and Hellon (6). However, unlike forearm blood flow, finger blood flow showed no appreciable response to either the head-up or head-down tilts. This indicates that the sympathetic tone and the volume of blood in the finger are not appreciably altered by this test procedure at least 1 min after the body tilt is assumed.

Increased vascular resistance in paralyzed legs after spinal cord injury is reversible by training

2002 ◽  
Vol 93 (6) ◽  
pp. 1966-1972 ◽  
Author(s):  
Maria T. E. Hopman ◽  
Jan T. Groothuis ◽  
Marcel Flendrie ◽  
Karin H. L. Gerrits ◽  
Sibrand Houtman
Keyword(s):  
Blood Pressure ◽  
Spinal Cord ◽  
Spinal Cord Injury ◽  
Blood Flow ◽  
Mean Arterial Pressure ◽  
Arterial Pressure ◽  
Vascular Resistance ◽  
Arterial Blood Flow ◽  
Arterial Blood ◽  
Cord Injury

The purpose of the present study was to determine the effect of a spinal cord injury (SCI) on resting vascular resistance in paralyzed legs in humans. To accomplish this goal, we measured blood pressure and resting flow above and below the lesion (by using venous occlusion plethysmography) in 11 patients with SCI and in 10 healthy controls (C). Relative vascular resistance was calculated as mean arterial pressure in millimeters of mercury divided by the arterial blood flow in milliliters per minute per 100 milliliters of tissue. Arterial blood flow in the sympathetically deprived and paralyzed legs of SCI was significantly lower than leg blood flow in C. Because mean arterial pressure showed no differences between both groups, leg vascular resistance in SCI was significantly higher than in C. Within the SCI group, arterial blood flow was significantly higher and vascular resistance significantly lower in the arms than in the legs. To distinguish between the effect of loss of central neural control vs. deconditioning, a group of nine SCI patients was trained for 6 wk and showed a 30% increase in leg blood flow with unchanged blood pressure levels, indicating a marked reduction in vascular resistance. In conclusion, vascular resistance is increased in the paralyzed legs of individuals with SCI and is reversible by training.

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