Samples needed for louping ill survey

Until 2019, TBE was considered only to be an imported disease to the United Kingdom. In that year, evidence became available that the TBEV is likely circulating in the country1,2 and a first “probable case” of TBE originating in the UK was reported.3 In addition to TBEV, louping ill virus (LIV), a member of the TBEV-serocomplex, is also endemic in parts of the UK. Reports of clinical disease caused by LIV in livestock are mainly from Scotland, parts of North and South West England and Wales.4

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ANTIBODY TO MURRAY VALLEY ENCEPHALITIS AND LOUPING‐ILL VIRUSES IN AUSTRALIA AND PAPUA‐NEW GUINEA

The Medical Journal of Australia ◽

10.5694/j.1326-5377.1961.tb24198.x ◽

1961 ◽

Vol 1 (12) ◽

pp. 444-447 ◽

Cited By ~ 3

Author(s):

S. G. Anderson ◽

A. V. G. Price ◽

M. C. Williams ◽

Margaret McElroy ◽

Nanadai‐Koia

Keyword(s):

Papua New Guinea ◽

New Guinea ◽

Louping Ill

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JAPANESE B ENCEPHALITIS VIRUS: ITS DIFFERENTIATION FROM ST. LOUIS ENCEPHALITIS VIRUS AND RELATIONSHIP TO LOUPING-ILL VIRUS

Science ◽

10.1126/science.86.2235.402 ◽

1937 ◽

Vol 86 (2235) ◽

pp. 402-403 ◽

Cited By ~ 6

Author(s):

L. T. Webster

Keyword(s):

Encephalitis Virus ◽

Louping Ill

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INFECTION OF THE CENTRAL NERVOUS SYSTEM BY LOUPING ILL VIRUS

Australian Journal of Experimental Biology and Medical Science ◽

10.1038/icb.1938.25 ◽

1938 ◽

Vol 16 (3) ◽

pp. 233-240 ◽

Cited By ~ 14

Author(s):

FM Burnet ◽

Dora Lush

Keyword(s):

Central Nervous System ◽

Nervous System ◽

The Central Nervous System ◽

Louping Ill

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The serological response and long-lasting resistance against infection with louping-ill virus in sheep immunized with a highly attenuated tick-borne encephalitis virus

Epidemiology and Infection ◽

10.1017/s0022172400042170 ◽

1969 ◽

Vol 67 (4) ◽

pp. 731-738 ◽

Cited By ~ 1

Author(s):

V. Mayer ◽

D. Blaškovič ◽

E. Ernek ◽

H. Libíková

Keyword(s):

Neutralizing Antibodies ◽

Encephalitis Virus ◽

Serological Response ◽

Immune Control ◽

Summer Period ◽

Tick Borne Encephalitis ◽

Louping Ill ◽

Tick Borne Encephalitis Virus

SUMMARYThe vaccination of sheep with one dose of the monkey-and mouse-attenuated tick-borne encephalitis virus (the Hy-HK 28 ‘2’ clone) causes seroconversion from negative into positive in 85% of animals. In sheep with pre-existing virus-neutralizing antibodies and increas of their titres was observed in 81%. The antibodies persisted for at least 12 months after the vaccination and during the summer period of grazing the number of serologically positive animals even increased.The vaccinated animals, in contrast to the non-immune control sheep, developed no viraemia after challenge with the virulent louping-ill virus, performed 11 months after immunization.

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The Golgi Material of the Neurones of the Central Nervous System of Sheep Infected with Louping-Ill

Journal of Cell Science ◽

10.1242/jcs.s3-88.1.55 ◽

1947 ◽

Vol s3-88 (1) ◽

pp. 55-63

Author(s):

R. A. R. GRESSON ◽

I. ZLOTNIK

Keyword(s):

Spinal Cord ◽

Central Nervous System ◽

Nervous System ◽

Purkinje Cells ◽

Medulla Oblongata ◽

Motor Nerve ◽

Pyramidal Cells ◽

Cell Processes ◽

The Central Nervous System ◽

Louping Ill

1. The Golgi material of the pyramidal cells of the cerebral cortex, the Purkinje cells of the cerebellum, and the multipolar cells of the medulla oblongata and ventral horns of the spinal cord of the sheep is present as filaments and as irregularly shaped bodies. In some of the cells, particularly in the lamb (Sheep V), the Golgi material has the appearance of a network. As it is frequently present as separate bodies it is suggested that it may always consist of discrete Golgi elements which are sometimes situated in close proximity or in contact with one another. Filamentous Golgi elements are present in the basal part of the cell processes. 2. An examination of neurones from the corresponding regions of the central nervous system of sheep infected experimentally with louping-ill showed that the Golgi material undergoes changes consequent upon the invasion of the cells by the virus. The Golgi material undergoes hypertrophy, and at the same time there is a reduction in the number of filamentous Golgi elements and a reduction in the amount of Golgi substance present in the cell processes. These changes are followed by fragmentation. All the neurones of a particular region are not affected equally at the same time. The Golgi material of the Purkinje cells tends to form groups in the cytoplasm prior to fragmentation. In the multipolar cells of the medulla oblongata the hypertrophy of the Golgi material is not as great as in the other regions of the central nervous system. The Golgi material of the motor nerve-cells of the ventral horns of the spinal cord undergoes considerable hypertrophy which is followed by a grouping of the Golgi elements and fragmentation.

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