Thoracic Decompression

2019 ◽  
pp. 155-166
Author(s):  
Mena G. Kerolus ◽  
Mazda K. Turel ◽  
Albert P. Wong ◽  
Zachary A. Smith ◽  
Richard G. Fessler
Keyword(s):  
Thoracic Decompression

Thoracic Decompression

2014 ◽  
pp. 99-108
Author(s):  
Albert P. Wong ◽  
Zachary A. Smith ◽  
Rohan R. Lall ◽  
Richard G. Fessler
Keyword(s):  
Thoracic Decompression

scholarly journals Cerebrospinal Fluid Leakage after Thoracic Decompression

2016 ◽  
Vol 129 (16) ◽  
pp. 1994-2000 ◽  
Author(s):  
Pan-Pan Hu ◽  
Xiao-Guang Liu ◽  
Miao Yu
Keyword(s):  
Cerebrospinal Fluid ◽  
Cerebrospinal Fluid Leakage ◽  
Fluid Leakage ◽  
Thoracic Decompression

How i do it: biportal endoscopic thoracic decompression for ossification of the ligamentum flavum

2021 ◽  
Author(s):  
Min-Seok Kang ◽  
Hoon-Jae Chung ◽  
Ki-Han You ◽  
Hyun-Jin Park
Keyword(s):  
Ligamentum Flavum ◽  
Thoracic Decompression

Abstract 2230: Lower Extremity Counterpulsation During The Decompression Phase of CPR Improves Hemodynamics And Provides Continuous Forward Carotid Blood Flow.

Circulation ◽  
2007 ◽  
Vol 116 (suppl_16) ◽  
Author(s):  
Demetris Yannopoulos ◽  
Henry R Halperin ◽  
Johns Hopkins ◽  
Keith G Lurie
Keyword(s):  
Blood Flow ◽  
Lower Extremity ◽  
Cerebral Perfusion Pressure ◽  
Cerebral Perfusion ◽  
Perfusion Pressure ◽  
Fold Increase ◽  
Aortic Pressure ◽  
Hemodynamic Parameters ◽  
Carotid Blood Flow ◽  
Thoracic Decompression

Background: We hypothesize that lower extremity counterpulsations (LECP) during the decompression phase of CPR will increase diastolic aortic pressure without raising the intracranial pressure (ICP) and significantly increase forward carotid blood flow. Methods: In 6 (24±4Kg) pigs, 4 minutes of untreated VF were followed by 2 minutes epochs of standard (STD) CPR, STD +LECP, active compression decompression (ACD) +ITD and ACD + inspiratory impedance threshold device (ITD)+LECP. Compressions and ventilations were performed according to the 2005 AHA guidelines. A large blood pressure cuff that was circumferentially placed around the pig’s thighs avoiding the abdominal cavity was inflated with air to the point that manometer pressure started to rise. Compressions over the cuff were performed manually and synchronized with the thoracic decompression phase. The cuff was compressed up to 200mmHg. Basic hemodynamic parameters and common carotid blood flow (CCBF) (ml/min) were continuously measured. Cerebral perfusion pressure (CerPP) (mean arterial pressure - mean ICP) and CPP (diastolic AoP - diastolic RAP) were calculated at the end of the 2 minute epochs. Statistical analysis was performed with ANOVA. Results: LECP significantly increased diastolic aortic pressure, CPP and CerPP both when added on STD and on ACD+ITD CPR. The largest benefit was observed with ACD+ITD+LECP were there was a 2-fold increase in CPP and CerPP with a 2.5-fold increase in common carotid blood flow compared to STD CPR. LECP did not alter ICP. (Table 1 ). Decompression phase CCBF during ACD+ITD+LECP CPR was positive and reached pre cardiac arrest levels. Conclusion: LECP during thoracic decompression significantly augmented aortic diastolic pressure, CPP and CerPP without increasing the ICP . LECP led to a continuous positive cerebral perfusion pressure gradient during CPR cycles and in combination with ACD + ITD CPR common carotid blood flow reached pre-arrest levels. Hemodynamic Parameters

Paget-Schroetter syndrome

2003 ◽  
Vol 18 (1) ◽  
pp. 2-11 ◽  
Author(s):  
S N Khan ◽  
G Stansby
Keyword(s):  
Active Treatment ◽  
Randomized Trials ◽  
Recent Literature ◽  
Management Strategies ◽  
Decompression Surgery ◽  
Or Management ◽  
Outcome Management ◽  
Intimal Injury ◽  
Thoracic Decompression ◽  
Review Current

Objective: To review current approaches to investigation and management of Paget-Schroetter syndrome (PSS). Methods: Relevant clinical studies and reports were searched for using MEDLINE and Embase databases and cross-referenced articles. Articles were extracted using keywords by the two authors independently. Principal findings: There is growing evidence of functional or positional anatomical defects, which lead to subtle intimal injury and a tendency for recurrence or poor outcome. Management of PSS remains controversial. Anticoagulation as a stand-alone treatment has lost favour. Active treatment with thrombolysis followed by consideration of thoracic decompression is recommended by most in the recent literature. Thrombolytic therapy appears to be a safe and efficacious method of establishing immediate patency of the axillary/subclavian vein. The timing and indications of decompression surgery are yet to be defined clearly. There are no randomized trials of treatment or management strategies in PSS. Conclusions: Treatment of PSS remains contentious. Most authors recommend active treatment with thrombolysis followed by thoracic decompression. Multicentre randomized trials are needed.

Minimally invasive thoracic decompression for multi-level thoracic pathologies

2014 ◽  
Vol 21 (3) ◽  
pp. 467-472 ◽  
Author(s):  
Zachary A. Smith ◽  
Cort D. Lawton ◽  
Albert P. Wong ◽  
Nader S. Dahdaleh ◽  
Alexander T. Nixon ◽  
...  
Keyword(s):  
Minimally Invasive ◽  
Multi Level ◽  
Thoracic Decompression
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