Connecting the Dots: Constructing Spatiotemporal Episodes from Events Schemas

2009 ◽  
pp. 57-76
Author(s):  
Arie Croitoru
Keyword(s):  
Connecting The Dots

Connecting the Dots

10.1596/29751 ◽  
2017 ◽  
Author(s):  
Keyword(s):  
Connecting The Dots

Connecting the Dots: Lasers Link Students to Their 3-D World

Science Scope ◽  
2015 ◽  
Vol 039 (02) ◽  
Author(s):  
Ross Parsons ◽  
Jan Eitel ◽  
Brian Whitney ◽  
Troy Magney ◽  
Karla Eitel ◽  
...  
Keyword(s):  
Connecting The Dots

Connecting the Dots: Market Reactions to Forecasts of Policy Rates and Forward Guidance Provided by the Fed

2016 ◽  
Author(s):  
Michelle Bongard ◽  
Gabriele Galati ◽  
Richhild Moessner ◽  
William Nelson
Keyword(s):  
Market Reactions ◽  
Forward Guidance ◽  
Connecting The Dots

scholarly journals Anti-phospholipid syndrome and COVID-19 thrombosis: connecting the dots

2021 ◽  
Vol 5 (1) ◽  
Author(s):  
Moon Ley Tung ◽  
Bryce Tan ◽  
Robin Cherian ◽  
Bharatendu Chandra
Keyword(s):  
Endothelial Dysfunction ◽  
Severe Acute Respiratory Syndrome ◽  
Potential Role ◽  
Molecular Mimicry ◽  
Thromboembolic Events ◽  
The Past ◽  
High Prevalence ◽  
Connecting The Dots ◽  
Anti Phospholipid Syndrome

Abstract As the coronavirus disease 2019 (COVID-19) pandemic, which is caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), is spreading rapidly worldwide, it has emerged as a leading cause of mortality, resulting in >1 million deaths over the past 10 months. The pathophysiology of COVID-19 remains unclear, posing a great challenge to the medical management of patients. Recent studies have reported an unusually high prevalence of thromboembolic events in COVID-19 patients, although the mechanism remains elusive. Several studies have reported the presence of aPLs in COVID-19 patients. We have noticed similarities between COVID-19 and APS, which is an autoimmune prothrombotic disease that is often associated with an infective aetiology. Molecular mimicry and endothelial dysfunction could plausibly explain the mechanism of thrombogenesis in acquired APS. In this review, we discuss the clinicopathological similarities between COVID-19 and APS, and the potential role of therapeutic targets based on the anti-phospholipid model for COVID-19 disease.

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