The Neuromuscular Junction: Areas of Uncertainty

Degeneration of the neuromuscular junction as a cause for sarcopenia measured by C-terminal Agrin Fragment

Aktuelle Ernährungsmedizin ◽

10.1055/s-0032-1312562 ◽

2012 ◽

Vol 37 (03) ◽

Author(s):

M Drey ◽

JM Bauer ◽

CC Sieber ◽

P Dahinden ◽

RG Fariello ◽

...

Keyword(s):

Neuromuscular Junction

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Faculty Opinions recommendation of Glial cells maintain synaptic structure and function and promote development of the neuromuscular junction in vivo.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1016572.200166 ◽

2003 ◽

Author(s):

Lennart Brodin

Keyword(s):

Neuromuscular Junction ◽

Glial Cells ◽

Structure And Function ◽

Synaptic Structure ◽

And Function

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Faculty Opinions recommendation of PAR-1 kinase phosphorylates Dlg and regulates its postsynaptic targeting at the Drosophila neuromuscular junction.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1061875.513822 ◽

2007 ◽

Author(s):

Johannes Hell

Keyword(s):

Neuromuscular Junction

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Faculty Opinions recommendation of Defective peripheral nerve myelination and neuromuscular junction formation in fukutin-deficient chimeric mice.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.1108935.566085 ◽

2008 ◽

Author(s):

Rhona Mirsky

Keyword(s):

Neuromuscular Junction ◽

Peripheral Nerve ◽

Chimeric Mice ◽

Junction Formation

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Faculty Opinions recommendation of Botulinum neurotoxin type A is internalized and translocated from small synaptic vesicles at the neuromuscular junction.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.717988167.793477443 ◽

2013 ◽

Author(s):

Patrick McNutt ◽

Ian Gut

Keyword(s):

Neuromuscular Junction ◽

Synaptic Vesicles ◽

Botulinum Neurotoxin ◽

Type A ◽

Botulinum Neurotoxin Type A

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Faculty Opinions recommendation of Identification of Mob2, a novel regulator of larval neuromuscular junction morphology, in natural populations of Drosophila melanogaster.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.718087215.793483743 ◽

2013 ◽

Author(s):

J Troy Littleton ◽

Kathryn Harris

Keyword(s):

Drosophila Melanogaster ◽

Neuromuscular Junction ◽

Natural Populations ◽

Larval Neuromuscular Junction

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Faculty Opinions recommendation of Neuromuscular disease. DOK7 gene therapy benefits mouse models of diseases characterized by defects in the neuromuscular junction.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.718882818.793519640 ◽

2016 ◽

Author(s):

Angela Vincent

Keyword(s):

Gene Therapy ◽

Neuromuscular Junction ◽

Mouse Models ◽

Neuromuscular Disease

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Faculty Opinions recommendation of Cross-disease comparison of amyotrophic lateral sclerosis and spinal muscular atrophy reveals conservation of selective vulnerability but differential neuromuscular junction pathology.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.725879208.793528046 ◽

2017 ◽

Author(s):

William Phillips

Keyword(s):

Amyotrophic Lateral Sclerosis ◽

Neuromuscular Junction ◽

Spinal Muscular Atrophy ◽

Muscular Atrophy ◽

Selective Vulnerability ◽

Lateral Sclerosis

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Faculty Opinions recommendation of Calcium Influx and Release Cooperatively Regulate AChR Patterning and Motor Axon Outgrowth during Neuromuscular Junction Formation.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.733518672.793557613 ◽

2019 ◽

Author(s):

William Phillips

Keyword(s):

Neuromuscular Junction ◽

Calcium Influx ◽

Axon Outgrowth ◽

Motor Axon ◽

Junction Formation

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Faculty Opinions recommendation of The Drosophila miR-310 cluster negatively regulates synaptic strength at the neuromuscular junction.

Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature ◽

10.3410/f.8336957.8775055 ◽

2011 ◽

Author(s):

Tudor Fulga

Keyword(s):

Neuromuscular Junction ◽

Synaptic Strength

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