Slow Potentials of ERG in Hemeralopia Congenita

MacDonald, Kurt D., Eva Fifkova, Michael S. Jones, and Daniel S. Barth. Focal stimulation of the thalamic reticular nucleus induces focal gamma waves in cortex. J. Neurophysiol. 79: 474–477, 1998. Electrical stimulation of the thalamic reticular nucleus (TRN; 0.5-s trains of 500-Hz 0.5-ms pulses at 5–10 μA) evokes focal oscillations of cortical electrical potentials in the gamma frequency band (∼35–55 Hz). These evoked oscillations are specific to either the somatosensory or auditory cortex and to subregions of the cortical receptotopic map, depending on what part of the TRN is stimulated. Focal stimulation of the internal capsule, however, evokes focal slow potentials, without gamma activity. Our results suggest that the TRN's role extends beyond that of general cortical arousal to include specific modality and submodality activation of the forebrain.

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Slow Potentials and Microprocessor Applications. Proceedings of the 20th International Society for Clinical Electrophysiology of Vision Symposium, Iowa City, October 25-28, 1982

American Journal of Ophthalmology ◽

10.1016/0002-9394(84)90538-5 ◽

1984 ◽

Vol 97 (6) ◽

pp. 806

Author(s):

Harold W. Skalka

Keyword(s):

International Society ◽

Iowa City ◽

Clinical Electrophysiology ◽

Slow Potentials

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Slow Activity in the Nervous System of the Earthworm, Lumbricus Terrestris

Journal of Experimental Biology ◽

10.1242/jeb.46.3.571 ◽

1967 ◽

Vol 46 (3) ◽

pp. 571-583

Author(s):

M. B. V. ROBERTS

Keyword(s):

Nervous System ◽

Nerve Cord ◽

Action Potentials ◽

Longitudinal Muscle ◽

Lumbricus Terrestris ◽

Longitudinal Contraction ◽

Peripheral Stimulation ◽

Segmental Nerve ◽

Slow Potentials ◽

Slow Activity

1. Three thresholds are demonstrated in the first segmental nerve and two (sometimes three) in the second and third segmental nerves together. 2. Slow potentials recorded from the ventral nerve cord consist of several peaks. The first peak is composed of three spikes which make their appearance at different thresholds. Transmission of at least some of the slow potentials is decremental. 3. Transmission speeds in the nerve cord and segmental nerves range from 0.4 to 0.6 m./sec. 4. Action potentials in the longitudinal muscle are recorded in response to slow potentials in the nerve cord. 5. Two slow reflexes, one involving elongation, the other longitudinal contraction, are described. The latter has the lower threshold with peripheral stimulation. 6. Slow activity in the nervous system is discussed in relation to reflex activity of the earthworm and the neurone anatomy of the nerve cord and segmental nerves.

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INTRODUCTION TO AN EXPERIMENTAL APPROACH TO BRAIN SLOW POTENTIALS

The Responsive Brain ◽

10.1016/b978-0-7236-0443-3.50058-1 ◽

1976 ◽

pp. 211-212

Keyword(s):

Experimental Approach ◽

Slow Potentials

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Evaluation of Event-related Slow Potentials in Selected Groups of Psychiatric Patients

The Responsive Brain ◽

10.1016/b978-0-7236-0443-3.50044-1 ◽

1976 ◽

pp. 150-153 ◽

Cited By ~ 6

Author(s):

B. DUBROVSKY ◽

M. DONGIER

Keyword(s):

Psychiatric Patients ◽

Slow Potentials

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Associations between cortical slow potentials and clinical rating scales in panic disorder: a 1.5- year follow-up study

European Psychiatry ◽

10.1016/s0924-9338(99)00229-1 ◽

1999 ◽

Vol 14 (7) ◽

pp. 399-404 ◽

Cited By ~ 4

Author(s):

W. Dengler ◽

G. Wiedemann ◽

P. Pauli

Keyword(s):

Panic Disorder ◽

Slow Wave ◽

Rating Scales ◽

Relative Magnitude ◽

Healthy Controls ◽

Follow Up Study ◽

Slow Potentials ◽

Psychometric Measures ◽

Hamilton Anxiety Scale

SummaryIn a previous study of 15 panic patients, we demonstrated that body-related (somatic) word stimuli elicited an enhanced positive cortical slow wave compared to non-somatic word stimuli. Healthy controls did not show this difference. The present paper reports on psychometric ratings in relation to cortical slow waves in these patients. Patients were clinically reexamined after about 1.5 years. Although no significant correlations between neurophysiology and psychometric measures could be found at the onset of the study, there was a significant correlation between improvement over the follow-up period and neurophysiology. A decline in the Hamilton Anxiety Scale (HAMA), which proved to be the best estimate for improvement, was associated with the relative magnitude of the positive slow wave elicited by somatic stimuli. Our findings support cognitive models of panic disorder, which stress that abnormal processing of bodily symptoms is relevant for the development and/or maintenance of the disorder.

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