Assignment of three loci: tumor suppressor protein-53 (TP53), retinoic acid receptor A (RARA), collagen 1A1 (COL1A1) to Syrian hamster Chromosome 9. Evidence that this chromosome is homologous with human Chromosome 17, mouse Chromosome 11, and rat Chromosome 10

1995 ◽  
Vol 6 (12) ◽  
pp. 882-884 ◽  
Author(s):  
M. Q. Islam ◽  
G. F�ng ◽  
K. Islam ◽  
G. Levan
Keyword(s):  
Retinoic Acid ◽  
Human Chromosome ◽  
Syrian Hamster ◽  
Retinoic Acid Receptor ◽  
Chromosome 9 ◽  
Chromosome 17 ◽  
Tumor Suppressor Protein ◽  
Chromosome 11 ◽  
Human Chromosome 17 ◽  
Mouse Chromosome 11

The Gene Encoding Protein Kinase SEK1 Maps to Mouse Chromosome 11 and Human Chromosome 17

Genomics ◽  
1996 ◽  
Vol 34 (3) ◽  
pp. 430-432 ◽  
Author(s):  
Robert A. White ◽  
Rowland T. Hughes ◽  
Linda R. Adkison ◽  
Gail Bruns ◽  
Leonard I. Zon
Keyword(s):  
Protein Kinase ◽  
Human Chromosome ◽  
Mouse Chromosome ◽  
Chromosome 17 ◽  
Chromosome 11 ◽  
Gene Encoding ◽  
Human Chromosome 17 ◽  
Mouse Chromosome 11 ◽  
Encoding Protein

Assignment ofAmog (adhesion molecule on glia) gene to mouse chromosome 11 nearZfp-3 andAsgr-1,2 and to human chromosome 17

1990 ◽  
Vol 16 (4) ◽  
pp. 401-405 ◽  
Author(s):  
Chih-Lin Hsieh ◽  
Andrea Cheng-Deutsch ◽  
Sergio Gloor ◽  
Melitta Schachner ◽  
Uta Francke
Keyword(s):  
Human Chromosome ◽  
Mouse Chromosome ◽  
Adhesion Molecule ◽  
Chromosome 17 ◽  
Chromosome 11 ◽  
Human Chromosome 17 ◽  
Mouse Chromosome 11

scholarly journals A comprehensive genetic map of murine chromosome 11 reveals extensive linkage conservation between mouse and human.

Genetics ◽  
1989 ◽  
Vol 122 (1) ◽  
pp. 153-161 ◽  
Author(s):  
A M Buchberg ◽  
E Brownell ◽  
S Nagata ◽  
N A Jenkins ◽  
N G Copeland
Keyword(s):  
Human Chromosome ◽  
Linkage Map ◽  
Mouse Chromosome ◽  
Chromosome 17 ◽  
Human Chromosomes ◽  
Chromosome 11 ◽  
Murine Chromosome ◽  
Human Chromosome 17 ◽  
Mouse Chromosome 11 ◽  
Linkage Conservation

Abstract Interspecific backcross animals from a cross between C57BL/6J and Mus spretus mice were used to generate a comprehensive linkage map of mouse chromosome 11. The relative map positions of genes previously assigned to mouse chromosome 11 by somatic cell hybrid or genetic backcross analysis were determined (Erbb, Rel, 11-3, Csfgm, Trp53-1, Evi-2, Erba, Erbb-2, Csfg, Myhs, Cola-1, Myla, Hox-2 and Pkca). We also analyzed genes that we suspected would map to chromosome 11 by virtue of their location in human chromosomes and the known linkage homologies that exist between murine chromosome 11 and human chromosomes (Mpo, Ngfr, Pdgfr and Fms). Two of the latter genes, Mpo and Ngfr, mapped to mouse chromosome 11. Both genes also mapped to human chromosome 17, extending the degree of linkage conservation observed between human chromosome 17 and mouse chromosome 11. Pdgfr and Fms, which are closely linked to II-3 and Csfgm in humans on chromosome 5, mapped to mouse chromosome 18 rather than mouse chromosome 11, thereby defining yet another conserved linkage group between human and mouse chromosomes. The mouse chromosome 11 linkage map generated in these studies substantially extends the framework for identifying homologous genes in the mouse that are involved in human disease, for elucidating the genes responsible for several mouse mutations, and for gaining insights into chromosome evolution and genome organization.

Genetic map of nine polymorphic loci comprising a single linkage group on rat chromosome 10: Evidence for linkage conservation with human chromosome 17 and mouse chromosome 11

Genomics ◽  
1992 ◽  
Vol 14 (3) ◽  
pp. 618-623 ◽  
Author(s):  
Elaine F. Remmers ◽  
Ellen A. Goldmuntz ◽  
Joseph M. Cash ◽  
Leslie J. Crofford ◽  
Barbara Misiewicz-Poltorak ◽  
...  
Keyword(s):  
Linkage Group ◽  
Human Chromosome ◽  
Mouse Chromosome ◽  
Genetic Map ◽  
Chromosome 17 ◽  
Chromosome 11 ◽  
Single Linkage ◽  
Human Chromosome 17 ◽  
Mouse Chromosome 11 ◽  
Linkage Conservation

scholarly journals Hormonal regulation of TSE1-repressed genes: evidence for multiple genetic controls in extinction.

1989 ◽  
Vol 9 (7) ◽  
pp. 2837-2846 ◽  
Author(s):  
M J Thayer ◽  
R E Fournier
Keyword(s):  
Hormonal Regulation ◽  
Phosphoenolpyruvate Carboxykinase ◽  
Genetic Locus ◽  
Tyrosine Aminotransferase ◽  
Chromosome 17 ◽  
Chromosome 11 ◽  
Liver Functions ◽  
Genes Encoding ◽  
Human Chromosome 17 ◽  
Mouse Chromosome 11

Somatic cell hybrids formed by fusing hepatoma cells with fibroblasts generally fail to express liver functions, a phenomenon termed extinction. Previous studies demonstrated that extinction of the genes encoding tyrosine aminotransferase, phosphoenolpyruvate carboxykinase, and argininosuccinate synthetase is mediated by a specific genetic locus (TSE1) that maps to mouse chromosome 11 and human chromosome 17. In this report, we show that full repression of these genes requires a genetic factor in addition to TSE1. This conclusion is based on the observation that residual gene activity was apparent in monochromosomal hybrids retaining human TSE1 but not in complex hybrids retaining many fibroblast chromosomes. Furthermore, TSE1-repressed genes were hormone inducible, whereas fully extinguished genes were not. Analysis of hybrid segregants indicated that genetic loci required for the complete repression phenotype were distinct from TSE1.

A new mutation Rim3 resembling Re den is mapped close to retinoic acid receptor alpha (Rara) gene on mouse Chromosome 11

1998 ◽  
Vol 9 (1) ◽  
pp. 20-25 ◽  
Author(s):  
Hajime Sato ◽  
Tsuyoshi Koide ◽  
Hiroshi Masuya ◽  
Shigeharu Wakana ◽  
Tomoko Sagai ◽  
...  
Keyword(s):  
Retinoic Acid ◽  
Mouse Chromosome ◽  
Retinoic Acid Receptor ◽  
Chromosome 11 ◽  
Retinoic Acid Receptor Alpha ◽  
New Mutation ◽  
Acid Receptor ◽  
Receptor Alpha ◽  
Mouse Chromosome 11

Hormonal regulation of TSE1-repressed genes: evidence for multiple genetic controls in extinction

1989 ◽  
Vol 9 (7) ◽  
pp. 2837-2846
Author(s):  
M J Thayer ◽  
R E Fournier
Keyword(s):  
Hormonal Regulation ◽  
Phosphoenolpyruvate Carboxykinase ◽  
Genetic Locus ◽  
Tyrosine Aminotransferase ◽  
Chromosome 17 ◽  
Chromosome 11 ◽  
Liver Functions ◽  
Genes Encoding ◽  
Human Chromosome 17 ◽  
Mouse Chromosome 11

Somatic cell hybrids formed by fusing hepatoma cells with fibroblasts generally fail to express liver functions, a phenomenon termed extinction. Previous studies demonstrated that extinction of the genes encoding tyrosine aminotransferase, phosphoenolpyruvate carboxykinase, and argininosuccinate synthetase is mediated by a specific genetic locus (TSE1) that maps to mouse chromosome 11 and human chromosome 17. In this report, we show that full repression of these genes requires a genetic factor in addition to TSE1. This conclusion is based on the observation that residual gene activity was apparent in monochromosomal hybrids retaining human TSE1 but not in complex hybrids retaining many fibroblast chromosomes. Furthermore, TSE1-repressed genes were hormone inducible, whereas fully extinguished genes were not. Analysis of hybrid segregants indicated that genetic loci required for the complete repression phenotype were distinct from TSE1.

An Integrated Radiation Hybrid Map of Bovine Chromosome 19 and Ordered Comparative Mapping with Human Chromosome 17

Genomics ◽  
1998 ◽  
Vol 48 (1) ◽  
pp. 93-99 ◽  
Author(s):  
Ya-Ping Yang ◽  
Caird E. Rexroad ◽  
Jörg Schläpfer ◽  
James E. Womack
Keyword(s):  
Human Chromosome ◽  
Radiation Hybrid ◽  
Comparative Mapping ◽  
Bovine Chromosome ◽  
Chromosome 17 ◽  
Chromosome 19 ◽  
Radiation Hybrid Map ◽  
Human Chromosome 17
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