Interhemispheric relations in patients with chronic alcoholism

1985 ◽  
Vol 15 (2) ◽  
pp. 161-164 ◽  
Author(s):  
T. N. Reshchikova
Keyword(s):  
Chronic Alcoholism ◽  
Interhemispheric Relations

Virus-Like Hepatic Cell Particles in Patients with Chronic Alcoholism

1963 ◽  
Vol 44 (3) ◽  
pp. 243-250 ◽  
Author(s):  
Yutaka Kikkawa ◽  
Boris Gueft
Keyword(s):  
Chronic Alcoholism ◽  
Hepatic Cell

Impaired abilities in chronic alcoholism as measured by the General Aptitude Test Battery

1968 ◽  
Author(s):  
George B. Kish ◽  
Truman M. Cheney
Keyword(s):  
Chronic Alcoholism ◽  
Test Battery ◽  
Aptitude Test ◽  
General Aptitude Test

Chronic Alcoholism Decreases Polyunsaturated Fatty Acid Levels in Human Plasma, Erythrocytes, and Platelets – Influence of Chronic Liver Disease

1997 ◽  
Vol 78 (02) ◽  
pp. 808-812 ◽  
Author(s):  
María-Luisa Pita ◽  
José-María Rubio ◽  
María-Luisa Murillo ◽  
Olimpia Carreras ◽  
Mariá-José Delgado
Keyword(s):  
Fatty Acid ◽  
Liver Disease ◽  
Chronic Liver Disease ◽  
Chronic Alcoholism ◽  
Chronic Liver ◽  
Chronic Ethanol ◽  
Ethanol Ingestion ◽  
Control Group ◽  
Docosatetraenoic Acid ◽  
Long Chain

SummaryThe effect of chronic ethanol ingestion on fatty acid composition of plasma, erythrocyte and platelet phospholipids and on plasma 6-keto-PGF1α was studied. Two groups of alcoholic subjects, one of them with chronic liver disease, were studied and compared to a control group of healthy subjects. Linoleic acid was not affected by alcoholism but its larger metabolites arachidonic acid (20:4n6) and docosatetraenoic acid (22: 4n6) tended to be lower in erythrocytes and platelets of both groups of alcoholic patients; the decrease was more marked in the presence of chronic liver disease. Docosahexaenoic acid (22:6n3) was markedly decreased in plasma, erythrocytes and platelets obtained from alcoholic patients with chronic liver disease. Plasma levels of 6-keto-PGF1α, a metabolite of prostacyclin (PGI2), remained unchanged. We conclude that chronic ethanol ingestion induces important changes in long-chain polyunsaturated fatty acids, mainly in platelets, and that these changes are exacerbated when patients suffer from chronic liver disease.

Chronic Alcoholism: A Psychological Survey

1941 ◽  
Vol 2 (1) ◽  
pp. 12-17 ◽  
Author(s):  
Edward A. Strecker
Keyword(s):  
Chronic Alcoholism

Effect of Xinnaojia Recipe on glutamate content and NR2B expression in hippocampus of rats with chronic alcoholism

2012 ◽  
Vol 32 (1) ◽  
pp. 112-114
Author(s):  
Shuang LI ◽  
Chun-yang XU ◽  
Jia WAN ◽  
Juan JIA ◽  
Guang-rui WAN
Keyword(s):  
Chronic Alcoholism

Ayurvedic understanding of Central Pontine Myelinolysis - A Case Report

2018 ◽  
Vol 3 (3) ◽  
Author(s):  
Anoop AS ◽  
Lakshmiprasad L. Jadhav ◽  
Sruthy Nair ◽  
Rohan Mohandas
Keyword(s):  
Serum Sodium Level ◽  
Central Pontine Myelinolysis ◽  
Chronic Alcoholism ◽  
Serum Electrolyte ◽  
Oral Medications ◽  
Shoulder Joints ◽  
Pontine Myelinolysis ◽  
Chief Complaints ◽  
Hyponatremic Encephalopathy ◽  
Central Pontine

A 56 year old male patient was admitted to S.D.M Ayurveda Hospital, Hassan, Karnataka with the confirmed diagnosis of Central Pontine Myelinolysis (CPM) on 11/12/17. The chief complaints were weakness of both hands and legs, stiffness in both hands and legs, pain in both shoulder joints, slurred speech, difficulty in walking with gait changes. H/O chronic alcoholism. MRI brain showed pontine and basal ganglia diffusion restriction - Acute Pontine Myelinolysis. The serum electrolyte showed serum sodium level as 128 mmol/litre. This disease can be understood as Samana Avruta Vyana in hyponatremic encephalopathy stage and the stage of myelinolysis can be understood as Sarvanga Vata with Kapha Avruta Udana and Vyana. After clinical evaluation, Avarana Chikitsa was started followed by Kevala Vatika Chikitsa and significant improvement was seen. Significant result was observed in subjective and objective parameters after the treatment. The patient was discharged with oral medications for 1 month.

THE TREATMENT OF CHRONIC ALCOHOLISM

1944 ◽  
Vol 2 (20) ◽  
pp. 516-516
Keyword(s):  
Chronic Alcoholism

scholarly journals Role of Chronic Alcoholism Causing Cancer in Omnivores and Vegetarians through Epigenetic Modifications

2020 ◽  
Vol 07 (03) ◽  
pp. 080-086
Author(s):  
Syed Aaquil Hasan Syed Javid Hasan ◽  
Raisa Arifanie O'Zelian Pawirotaroeno ◽  
Syed Abrar Hasan Syed Javid Hasan ◽  
Elene Abzianidze
Keyword(s):  
Folate Deficiency ◽  
Chronic Alcoholism ◽  
Epigenetic Modifications ◽  
Alcohol Metabolism ◽  
Dna Hypomethylation ◽  
Contributing Factors ◽  
Vitamin Deficiencies ◽  
Adenosyl Methionine ◽  
And Oxidative Stress

AbstractOne of the significant consequences of alcohol consumption is cancer formation via several contributing factors such as action of alcohol metabolites, vitamin deficiencies, and oxidative stress. All these factors have been shown to cause epigenetic modifications via DNA hypomethylation, thus forming a basis for cancer development. Several published reviews and studies were systematically reviewed. Omnivores and vegetarians differ in terms of nutritional intake and deficiencies. As folate deficiency was found to be common among the omnivores, chronic alcoholism could possibly cause damage and eventually cancer in an omnivorous individual via DNA hypomethylation due to folate deficiency. Furthermore, as niacin was found to be deficient among vegetarians, damage in vegetarian chronic alcoholics could be due to increased NADH/NAD+ ratio, thus slowing alcohol metabolism in liver leading to increased alcohol and acetaldehyde which inhibit methyltransferase enzymes, eventually leading to DNA hypomethylation. Hence correcting the concerned deficiency and supplementation with S-adenosyl methionine could prove to be protective in chronic alcohol use.

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