Is it necessary to strictly diagnose fibromyalgia syndrome in patients with chronic widespread pain?

2015 ◽  
Vol 34 (8) ◽  
pp. 1473-1479 ◽  
Author(s):  
Arzu Yagiz On ◽  
Dilek Aykanat ◽  
Funda Calis Atamaz ◽  
Can Eyigor ◽  
Hayriye Kocanogullari ◽  
...  
Keyword(s):  
Fibromyalgia Syndrome ◽  
Chronic Widespread Pain ◽  
Widespread Pain

Fibromyalgia and chronic widespread pain syndromes—adult onset

2013 ◽  
Author(s):  
Anoop Kuttikat ◽  
Nicholas Shenker
Keyword(s):  
Fibromyalgia Syndrome ◽  
Multidisciplinary Approach ◽  
Chronic Widespread Pain ◽  
Widespread Pain ◽  
Adult Onset ◽  
Societal Costs ◽  
Psychological Therapies ◽  
Pain Syndromes ◽  
Unrefreshing Sleep ◽  
The Central Nervous System

Fibromyalgia syndrome (FMS) is characterized by chronic widespread pain, excessive fatigue, unrefreshing sleep, and other associated somatic symptoms. FMS is common in the general population with an estimated prevalence of 2-4% and is about six times more common in females than males. FMS causes significant individual and societal costs. The precise aetiology of FMS remains unclear. Dysfunctional pain processing within the central nervous system is the primary abnormality. FMS is a clinical diagnosis based on pattern recognition and it can coexist with other conditions. A multidisciplinary approach, incorporating patient education, physical therapies, psychological therapies, and pharmacotherapy, is effective in managing these patients.

scholarly journals Use of Ultrasonography to Discriminate Psoriatic Arthritis from Fibromyalgia: A Post-Hoc Analysis of the ULISSE Study

2021 ◽  
Vol 11 (1) ◽  
pp. 180
Author(s):  
Antonio Marchesoni ◽  
Pierluigi Macchioni ◽  
Stefania Gasparini ◽  
Carlo Perricone ◽  
Fabio Massimo Perrotta ◽  
...  
Keyword(s):  
Psoriatic Arthritis ◽  
Fibromyalgia Syndrome ◽  
Operating Characteristic ◽  
Characteristic Curve ◽  
Power Doppler ◽  
Area Under The Curve ◽  
Principal Component ◽  
Chronic Widespread Pain ◽  
Widespread Pain ◽  
Post Hoc

In psoriatic arthritis (PsA) patients with concomitant chronic widespread pain, the differential diagnosis with fibromyalgia syndrome (FMS) can be challenging. We evaluated whether ultrasound (US) examination of entheseal sites can distinguish pain from (PsA) enthesitis versus FMS. PsA and FMS patients underwent clinical evaluation and gray-scale (GS; B-mode) and power Doppler (PD) US examination of the entheses. At least one enthesis with GS- and PD-mode changes was found in 90% and 59.3% of PsA patients (n = 140) and 62.7% and 35.3% of FMS patients (n = 51), respectively. GS and PD identified changes in 49.5% and 19.2% of the 840 PsA entheses and 22.5% and 7.9% of the 306 FMS entheses, respectively. Receiver operating characteristic curve analysis showed an area under the curve of 0.77 and 0.66 for B- and PD-mode, respectively, 3.5 being the best cut-off GS-score to discriminate the two conditions. Multivariate regression showed that Achilles and proximal patellar tendon enthesitis (B-mode) were strongly associated with PsA (odds ratio, ~2). Principal component analysis (B-mode) confirmed that PsA patients have a higher number of involved entheses and patterns of entheseal involvement than FMS patients. US evaluation of the entheses may help differentiate chronic widespread pain from PsA versus FMS.

scholarly journals Chronic Widespread Pain and Fibromyalgia Syndrome: Life-Course Risk Markers in Young People

2019 ◽  
Vol 2019 ◽  
pp. 1-13 ◽  
Author(s):  
Aidan C. Tan ◽  
Tiina Jaaniste ◽  
David Champion
Keyword(s):  
Life Course ◽  
Fibromyalgia Syndrome ◽  
Laboratory Finding ◽  
Childhood Adversity ◽  
Chronic Widespread Pain ◽  
Extensive Literature ◽  
Pathological Feature ◽  
Widespread Pain ◽  
Risk Markers ◽  
Physical Trauma

Although the life-course concept of risk markers as potential etiological influences is well established in epidemiology, it has not featured in academic publications or clinical practice in the context of chronic widespread pain (CWP) and fibromyalgia syndrome (FMS). Studies of risk markers are required considerations for evaluation of patients and for research because there is no single cause, pathological feature, laboratory finding, or biomarker for CWP or FMS. The early-life risk markers identified by extensive literature review with best evidence for potential causal influence on the development and progression of CWP and FMS include genetic factors, premature birth, female sex, early childhood adversity, cognitive and psychosocial influences, impaired sleep, primary pain disorders, multiregional pain, physical trauma, infectious illness, obesity and inactivity, hypermobility of joints, iron deficiency, and small-fiber polyneuropathy. The case history illustrates the potential etiological influence of multiple risk markers offset by personal resilience.

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