Prepancreatic postduodenal portal vein: a new hypothesis for the development of the portal venous system

2010 ◽  
Vol 28 (2) ◽  
pp. 157-161 ◽  
Author(s):  
Nobuo Tomizawa ◽  
Hiroyuki Akai ◽  
Masaaki Akahane ◽  
Kenji Ino ◽  
Shigeru Kiryu ◽  
...  
Keyword(s):  
Portal Vein ◽  
Venous System ◽  
Portal Venous System ◽  
New Hypothesis

scholarly journals No Association of Homocysteine, Anticardiolipin Antibody, and Anti-β2 Glycoprotein I Antibody With Portal Venous System Thrombosis in Liver Cirrhosis

2021 ◽  
Vol 27 ◽  
pp. 107602962110109
Author(s):  
Le Wang ◽  
Xiaozhong Guo ◽  
Xiangbo Xu ◽  
Shixue Xu ◽  
Juqiang Han ◽  
...  
Keyword(s):  
Liver Cirrhosis ◽  
Portal Vein ◽  
Anticardiolipin Antibody ◽  
Venous System ◽  
Common Complication ◽  
Portal Venous System ◽  
Main Portal Vein ◽  
Glycoprotein I ◽  
Cirrhotic Patients ◽  
Clinically Significant

Portal venous system thrombosis (PVST), a common complication of liver cirrhosis, is closely associated with thrombophilia. To explore the association of homocysteine (Hcy), anticardiolipin antibody (aCL), and anti-β2 glycoprotein I antibody (aβ2GPI), which are possible thrombophilic factors, with PVST in liver cirrhosis. Overall, 654 non-malignant patients (219 with and 435 without liver cirrhosis) admitted between January 2016 and June 2020 were retrospectively evaluated. Presence of PVST, degree of main portal vein (MPV) thrombosis, and clinically significant PVST were identified. Hcy level, hyperhomocysteinemia (HHcy), aCL positivity, and aβ2GPI positivity were compared according to the presence of liver cirrhosis and PVST. Positive aβ2GPI was significantly more frequent in patients with liver cirrhosis than those without, but Hcy level and proportions of HHcy and positive aCL were not significantly different between them. PVST could be evaluated in 136 cirrhotic patients. Hcy level [10.57 μmol/L (2.71-56.82) versus 9.97 μmol/L (2.05-53.44); P = 0.796] and proportions of HHcy [4/44 (9.1%) versus 13/81 (16.0%); P = 0.413] and positive aCL [1/23 (4.3%) versus 10/52 (19.2%); P = 0.185] and aβ2GPI [9/23 (39.1%) versus 21/52 (40.4%); P = 0.919] were not significantly different between cirrhotic patients with and without PVST. There was still no significant association of Hcy level, HHcy, aCL, or aβ2GPI with PVST based on Child-Pugh classification, MPV thrombosis >50%, and clinically significant PVST. Hcy, aCL, and aβ2GPI may not be associated with PVST in liver cirrhosis, suggesting that routine screening for Hcy, aCL, and aβ2GPI should be unnecessary in such patients.

scholarly journals The ABCD of portal vein thrombosis: a systematic approach

2020 ◽  
Vol 53 (6) ◽  
pp. 424-429
Author(s):  
Alexandre Makoto Minoda ◽  
Raissa Brito Fernandes Cadete ◽  
Sara Reis Teixeira ◽  
Valdair Francisco Muglia ◽  
Jorge Elias Junior ◽  
...  
Keyword(s):  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Systematic Approach ◽  
Careful Analysis ◽  
Venous System ◽  
Portal Venous System ◽  
Vein Thrombosis ◽  
Case Files ◽  
Collateral Vessels ◽  
And Diffusion

Abstract Portal vein thrombosis refers to complete or partial obstruction of the portal venous system, in the intrahepatic or extrahepatic venous tract or even in the splenic or superior mesenteric veins. This common and potentially fatal condition can develop in various clinical contexts, especially those of liver cirrhosis, hepatocellular carcinoma, and other solid tumors. Certain characteristics, such as the time since the onset of the thrombus (acute or chronic), its biology (hematic or tumoral), the presence of collateral vessels, and the magnetic resonance imaging aspects, are important components of a thorough, careful analysis, as well as informing decisions regarding the appropriate therapeutic strategy. Here, we present a brief review of the anatomy of the portal venous system and a systematic approach to analyzing the condition, using a mnemonic (ABCD, for age, biology, collaterals, and diffusion). We discuss the various imaging methods and illustrate our discussion with images selected from the case files archived at our facility.

Aberrant Left Gastric Vein Directly Draining into Left Portal Venous System

1992 ◽  
Vol 33 (5) ◽  
pp. 462-463 ◽  
Author(s):  
H. Tajima ◽  
R. Murakami ◽  
T. Kumazaki
Keyword(s):  
Gastric Cancer ◽  
Liver Cirrhosis ◽  
Early Gastric Cancer ◽  
Portal Vein ◽  
Venous System ◽  
Portal Venous System ◽  
Left Gastric Vein ◽  
Direct Communication ◽  
Vein System ◽  
Left Portal Vein

A 66-year-old man with early gastric cancer and liver cirrhosis was diagnosed by preoperative angiography as having an aberrant left gastric vein communicating directly with the left lateral portal vein system. This communication was confirmed during operation for the gastric cancer. Our report is the first of an aberrant left gastric vein showing direct communication with the left portal vein system.

scholarly journals Percutaneous transhepatic treatment of a unique portal vein malformation with portal hypertension in a pediatric patient

2021 ◽  
Vol 4 (1) ◽  
Author(s):  
Paolo Marra ◽  
Ludovico Dulcetta ◽  
Claudia Pellegrinelli ◽  
Lorenzo D’Antiga ◽  
Sandro Sironi
Keyword(s):  
Portal Hypertension ◽  
Portal Vein ◽  
Variceal Bleeding ◽  
Umbilical Vein ◽  
Venous System ◽  
Portal Venous System ◽  
Vascular Abnormality ◽  
Non Invasive ◽  
Percutaneous Transhepatic Portography ◽  
Transhepatic Portography

Abstract Background Anomalies of the portal venous system can be congenital or acquired, the latter being related to spontaneous thrombosis or iatrogenic alterations such as complications of perinatal catheterization of the umbilical vein. These conditions can be clinically silent for years and then manifest abruptly causing severe clinical emergencies. Case presentation This case report describes the diagnosis and interventional management of a singular abnormality in the portal venous system of an 8-year-old female that led to severe portal hypertension and acute variceal bleeding. Peculiar imaging findings were not pathognomonic for any of the known congenital and acquired portal vein anomalies: absence of a normal extrahepatic portal vein; splenic and mesenteric veins merging into a dilated left gastric vein; presence of an aberrant mesenteric venous collateral with a stenotic connection with the intrahepatic right portal branch; and absence of porto-systemic shunt. The case was successfully managed with percutaneous transhepatic portography and angioplasty. Conclusions Prompt non-invasive imaging characterization allowed to understand the singular vascular abnormality and mini-invasive interventional radiology management resolved portal hypertension and variceal bleeding.

scholarly journals Anomalies of the Portal Venous System in Dogs and Cats as Seen on Multidetector-Row CT: An Overview and Systematization Proposal

2019 ◽  
Author(s):  
Giovanna Bertolini
Keyword(s):  
Portal Vein ◽  
Venous System ◽  
Low Flow ◽  
Portal System ◽  
Multidetector Row Computed Tomography ◽  
Portal Venous System ◽  
Multidetector Row Ct ◽  
Multidetector Row ◽  
Vein Thrombosis ◽  
Portosystemic Shunts

This article offers an overview of congenital and acquired vascular anomalies involving the portal venous system in dogs and cats, as determined by multidetector-row computed tomography angiography. Congenital absence of the portal vein, portal vein hypoplasia, portal vein thrombosis and portal collaterals are described. Portal collaterals are further discussed as high- and low-flow connections, and categorized in hepatic arterioportal malformation, arteriovenous fistula, end-to-side and side-to-side congenital portosystemic shunts, acquired portosystemic shunts, cavoportal and porto-portal collaterals. Knowledge of different portal system anomalies helps understand the underlying physiopathological mechanism and is essential for surgical and interventional approaches.

Sign in / Sign up

Export Citation Format

Share Document