Comparison of high- and low-intensity exercise training early after acute myocardial infarction

1988 ◽  
Vol 61 (1) ◽  
pp. 26-30 ◽  
Author(s):  
James A. Blumenthal ◽  
W.Jack Rejeski ◽  
Margaret Walsh-Riddle ◽  
Charles F. Emery ◽  
Henry Miller ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Exercise Training ◽  
Low Intensity ◽  
Low Intensity Exercise

scholarly journals Early programmes of high and low intensity exercise and quality of life after acute myocardial infarction.

BMJ ◽  
1993 ◽  
Vol 307 (6914) ◽  
pp. 1244-1247 ◽  
Author(s):  
M C Worcester ◽  
D L Hare ◽  
R G Oliver ◽  
M A Reid ◽  
A J Goble
Keyword(s):  
Quality Of Life ◽  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Low Intensity ◽  
Low Intensity Exercise

scholarly journals Effect of early programmes of high and low intensity exercise on physical performance after transmural acute myocardial infarction.

Heart ◽  
1991 ◽  
Vol 65 (3) ◽  
pp. 126-131 ◽  
Author(s):  
A J Goble ◽  
D L Hare ◽  
P S Macdonald ◽  
R G Oliver ◽  
M A Reid ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Physical Performance ◽  
Low Intensity ◽  
Low Intensity Exercise

scholarly journals Adaptation to Low-Intensity Exercise on a Cycle Ergometer by Patients With Acute Myocardial Infarction Undergoing Phase I Cardiac Rehabilitation

2004 ◽  
Vol 68 (10) ◽  
pp. 938-945 ◽  
Author(s):  
Atsuhiko Matsunaga ◽  
Takashi Masuda ◽  
Misao N Ogura ◽  
Masakazu Saitoh ◽  
Yusuke Kasahara ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Cardiac Rehabilitation ◽  
Phase I ◽  
Cycle Ergometer ◽  
Low Intensity ◽  
Low Intensity Exercise

scholarly journals Low-Intensity Exercise Improves Aerobic Exercise Capacity in Rats After Myocardial Infarction and the Possible Role of Skeletal Muscle Autophagy

2021 ◽  
Vol 34 (2) ◽  
pp. 226-226
Author(s):  
Li-mei Chen ◽  
Wen-wen Peng ◽  
Gui-qing Xu ◽  
Yue Guo ◽  
Ling-jie Wei ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Skeletal Muscle ◽  
Exercise Training ◽  
Aerobic Exercise ◽  
Exercise Capacity ◽  
Sham Group ◽  
Heart Weight ◽  
Low Intensity ◽  
Aerobic Exercise Capacity ◽  
Low Intensity Exercise

Abstract Background To investigate the effects of low-intensity exercise on aerobic exercise capacity and autophagy of skeletal muscle in rats after myocardial infarction (MI) and its possible mechanisms. Methods Thirty male Sprague Dawley rats, weighing 180–200 g, were randomly divided into sham, MI, and MI with exercise training (MI + Ex) groups. MI was induced by ligation of the left anterior descending artery. One week after surgery, low-intensity exercise training was carried out on a treadmill 5 days per week for 4 weeks. Results Infarct size of MI and MI + Ex groups was 30.8 ± 5.5% and 27.6 ± 5.0% of left ventricle, respectively (P > 0.05). Heart weight and heart to body weight ratio in the MI group were significantly higher than those in the sham group (P < 0.01), and were lowered by exercise training (P < 0.01). The maximal exercise distance and duration in the MI group were lower than those in the sham group (P < 0.01), but were significantly increased by exercise training (P < 0.05). Autophagosome of the gastrocnemius was not detectable in the sham group, scattered in the MI group but clustered in the MI + Ex group. Microtubule-associated protein light chain 3 (LC3-I/II) and Beclin-1 protein levels in the gastrocnemius were similar between MI and sham groups, but were significantly higher in the MI + Ex group (P < 0.05). Conclusions Low-intensity exercise improves exercise capacity in rats after MI. The effect is associated with enhanced autophagy of the skeletal muscle.

scholarly journals Exercise training enhances autonomic function after acute myocardial infarction: A randomized controlled study

2012 ◽  
Vol 31 (2) ◽  
pp. 135-141
Author(s):  
Fernando Ribeiro ◽  
Alberto Jorge Alves ◽  
Madalena Teixeira ◽  
Fátima Miranda ◽  
Cristina Azevedo ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Exercise Training ◽  
Autonomic Function ◽  
Randomized Controlled Study ◽  
Controlled Study ◽  
Randomized Controlled

Heart rate recovery improvement in patients following acute myocardial infarction: exercise training, β-blocker therapy or both

2017 ◽  
Vol 38 (3) ◽  
pp. 351-359 ◽  
Author(s):  
Wladimir M. Medeiros ◽  
Fabio A. de Luca ◽  
Alcides R. de Figueredo Júnior ◽  
Felipe A. R. Mendes ◽  
Carlos Gun
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Heart Rate ◽  
Exercise Training ◽  
Heart Rate Recovery ◽  
Blocker Therapy ◽  
Β Blocker

Abstract 332: Exercise Training Protects Against Acute Myocardial Infarction via Improving Myocardial Energy Metabolism and Mitochondrial Biogenesis

2015 ◽  
Vol 117 (suppl_1) ◽  
Author(s):  
Lichan Tao ◽  
Yihua Bei ◽  
Haifeng Zhang ◽  
Yanli Zhou ◽  
Jingfa Jiang ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Energy Metabolism ◽  
Exercise Training ◽  
Mitochondrial Biogenesis ◽  
Myocardial Infarct ◽  
Ischemia Reperfusion ◽  
Cardiac Injury ◽  
Myocardial Infarct Size ◽  
Myocardial Energy Metabolism

Acute myocardial infarction (AMI) represents a major cause of morbidity and mortality worldwide. Exercise has been proved to reduce myocardial ischemia-reperfusion (I/R) injury. However it remains unclear whether, and (if so) how, exercise could protect against AMI. Methods: Mice were trained using a 3-week swimming protocol, and then subjected to left coronary artery (LCA) ligation, and finally sacrificed 24 h after AMI. Results: Exercise training reduces myocardial infarct size and abolishes AMI-induced autophagy and apoptosis. MI leads to a shift from fatty acid to glucose metabolism in the myocardium with a downregulation of PPAR-α and PPAR-γ. Also, AMI induces an adaptive increase of mitochondrial DNA replication and transcription in the acute phase of MI, accompanied by an activation of PGC-1α signaling. Exercise abolishes the derangement of myocardial glucose and lipid metabolism and further enhances the adaptive increase of mitochondrial biogenesis. Conclusion: Exercise training protects against AMI-induced acute cardiac injury through improving myocardial energy metabolism and enhancing the early adaptive change of mitochondrial biogenesis.

scholarly journals Effect of Chair aerobics as low intensity exercise training on heart rate, blood pressure and six minute walk distance in post coronary artery bypass graft surgery patients through phase I cardiac rehabilitation

2016 ◽  
Vol 13 (1) ◽  
pp. 19-23
Author(s):  
Shaili Thapa ◽  
Renu B. Pattanshetty
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Exercise Training ◽  
Cardiac Rehabilitation ◽  
Phase I ◽  
Walk Distance ◽  
Low Intensity ◽  
Low Intensity Exercise ◽  
Minute Walk Distance ◽  
Minute Walk

Background and Aims: Cardiac Rehabilitation after Coronary arteries bypass graft (CABG) surgery is known to have several favorable effects. Aerobics exercise in the form of chair aerobics shown to have beneficial effects with little evidence. The purpose of the present study was to demonstrate the effect of chair aerobics as low intensity exercise training in heart rate, blood pressure and six minute walk distance in post CABG patients during phase I cardiac rehabilitation.   Methods: It was a single centre, prospective study carried out in Indian tertiary care set-up. Fifty patients post CABG patients were included. Chair aerobics as a low intensity exercise training was given.  Pre and post-exercise outcome measurements in the form of heart rate, blood pressure and six-minute walk distance test were recorded. Results: The study showed statistically significant difference in systolic blood pressure (p= 0.018), heart rate (p≤0.001) and increase in six minute walk distance (p≤0.001). Conclusion: Chair aerobics as low intensity exercise training demonstrated improvement in heart  rate, systolic blood pressure and six minute walk distance test in subjects with CABG through phase I  cardiac rehabilitation.  Nepalese Heart Journal 2016; 13(1) 19-23

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