Lithiation and silylation reactions of 1,4-bis(1,1,1,3,3,3-hexafluoro-2-hydroxy-2-propyl)benzene

1996 ◽  
Vol 522 (1) ◽  
pp. 55-57 ◽  
Author(s):  
John W. Fitch ◽  
Patrick E. Cassidy ◽  
M.Jamil Ahmed
Keyword(s):  
Propyl Benzene

The polymerization of styrene by sulphuric acid IV. Chain-stopping agents and mechanisms

1961 ◽  
Vol 263 (1312) ◽  
pp. 82-88 ◽  
Keyword(s):  
Molecular Weight ◽  
Sulphuric Acid ◽  
Cationic Polymerization ◽  
Transfer Equation ◽  
Molecular Transfer ◽  
Propyl Benzene

Alcohols, nitromethane and triphenyl methyl compounds are shown to cause a large depres­sion of molecular weight and yield in the cationic polymerization of styrene by sulphuric acid at 25°C. Ether, acetone and iso propyl benzene have a weaker effect. Where the equivalent of the Mayo transfer equation is obeyed, the ‘molecular transfer constants’ have been determined. Nitromethane and the triphenyl methyl compounds are anomalous in this respect. There is evidence that all the polar substances interact to a greater or less extent with the catalyst, as well as with the growing chains.

scholarly journals Control of laser induced molecular fragmentation of n-propyl benzene using chirped femtosecond laser pulses

2009 ◽  
Vol 360 (1-3) ◽  
pp. 47-52 ◽  
Author(s):  
Tapas Goswami ◽  
S.K. Karthick Kumar ◽  
Aveek Dutta ◽  
Debabrata Goswami
Keyword(s):  
Femtosecond Laser ◽  
Laser Pulses ◽  
Femtosecond Laser Pulses ◽  
Molecular Fragmentation ◽  
Propyl Benzene

scholarly journals The Regulatory Role of Nuclear Factor Kappa B in the Heart of Hereditary Hypertriglyceridemic Rat

2016 ◽  
Vol 2016 ◽  
pp. 1-6 ◽  
Author(s):  
Stanislava Vranková ◽  
Andrej Barta ◽  
Jana Klimentová ◽  
Ima Dovinová ◽  
Silvia Líšková ◽  
...  
Keyword(s):  
Oxidative Damage ◽  
Nuclear Factor ◽  
Sod Activity ◽  
Protein Expressions ◽  
Propyl Benzene ◽  
Oxide Synthase ◽  
Nos Isoforms ◽  
Enos Protein Expression ◽  
Enos Protein

Activation of nuclear factor-κB (NF-κB) by increased production of reactive oxygen species (ROS) might induce transcription and expression of different antioxidant enzymes and also of nitric oxide synthase (NOS) isoforms. Thus, we aimed at studying the effect of NF-κB inhibition, caused by JSH-23 (4-methyl-N1-(3-phenyl-propyl)-benzene-1,2-diamine) injection, on ROS and NO generation in hereditary hypertriglyceridemic (HTG) rats. 12-week-old, male Wistar and HTG rats were treated with JSH-23 (bolus, 10μmol, i.v.). After one week, blood pressure (BP), superoxide dismutase (SOD) activity, SOD1, endothelial NOS (eNOS), and NF-κB (p65) protein expressions were higher in the heart of HTG rats compared to control rats. On the other hand, NOS activity was decreased. In HTG rats, JSH-23 treatment increased BP and heart conjugated dienes (CD) concentration (measured as the marker of tissue oxidative damage). Concomitantly, SOD activity together with SOD1 expression was decreased, while NOS activity and eNOS protein expression were increased significantly. In conclusion, NF-κB inhibition in HTG rats led to decreased ROS degradation by SOD followed by increased oxidative damage in the heart and BP elevation. In these conditions, increased NO generation may represent rather a counterregulatory mechanism activated by ROS. Nevertheless, this mechanism was not sufficient enough to compensate BP increase in HTG rats.

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