Photochemical Oxidants

2014 ◽  
pp. 926-927
Author(s):  
S.C. Gad
Science ◽  
1970 ◽  
Vol 168 (3936) ◽  
pp. 1223-1224 ◽  
Author(s):  
G. P. Hanson ◽  
W. S. Stewart

2020 ◽  
Vol 10 (1) ◽  
Author(s):  
Masanobu Ishii ◽  
Tomotsugu Seki ◽  
Kenji Sakamoto ◽  
Koichi Kaikita ◽  
Yoshihiro Miyamoto ◽  
...  

Abstract Air pollution causes hypertension, cardiovascular disease, and mortality. Asian dust (AD) reportedly induces asthma or acute myocardial infarction along with air pollution, but its impact on blood pressure (BP) is unknown. We investigated the association between short-term AD exposure and BP fluctuations in 300,952 individuals whose BP was measured during April 2005–March 2015 and divided them into AD and non-AD groups based on visitation for AD-related events. AD’s occurrence, air pollutants’ concentration (suspended particulate matter, SO2, NO2, photochemical oxidants), and meteorological variables (mean ambient temperature, relative humidity) were obtained from a monitoring station; AD events correlated with decreased visibility (< 10 km). We observed 61 AD days, with 3897 participants undergoing medical check-ups. Short-term AD exposure at lag day-0 was significantly associated with higher systolic BP (SBP), diastolic BP (DBP), and pulse rate (PR) risk (β = 1.85, 95% confidence interval (CI) 1.35–2.35 for SBP, β = 2.24, 95% CI 1.88–2.61 for DBP, β = 0.52, 95% CI 0.14–0.91 for PR) using multi-pollutant model. Population-attributable fractions exposed to AD were 11.5% for those with elevated SBP (SBP ≥ 120 mmHg) and 23.7% for those with hypertension (SBP ≥ 140 mmHg or DBP ≥ 90 mmHg). This study showed a strong association between short-term AD exposure and increased SBP and DBP.


2006 ◽  
Vol 61 (11-12) ◽  
pp. 856-864 ◽  
Author(s):  
Daisuke Ogawa ◽  
Nobuyoshi Nakajima ◽  
Masanori Tamaoki ◽  
Mitsuko Aono ◽  
Akihiro Kubo ◽  
...  

Abstract O3 is the major component of photochemical oxidants and gives rise to visible injuries on plant leaves. In O3-exposed plants, O-2 is produced before the formation of the injury, but the role that O-2 plays in plant response to O3 exposure is still unknown. To clarify its role, we observed the behavior of plants during O3 exposure after pretreatment with tiron, which is an O-2 scavenger. When tiron-pretreated tobacco cv. Bel W3 was exposed to O3, leaf damage was attenuated. In O3-exposed tobacco, tiron inhibited increases in the levels of ethylene and salicylic acid, which promote leaf injury. Tiron pretreatment also suppressed increases in the expression of O3-induced genes. These results suggest that O-2 is involved in many plant responses induced by O3 exposure. Bel B, a tobacco cultivar that is genetically related to Bel W3, is reported to be more resistant to O3 than Bel W3, but the reason for this difference is unclear. We investigated the differences between the responses of Bel B and tiron-pretreated Bel W3 to O3 exposure, and we discuss the reasons for the resistance to O3 by comparing the phenotype of Bel B with that of tiron-pretreated Bel W3.


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