Boundedness in a chemotaxis-haptotaxis model with gradient-dependent flux limitation

2021 ◽  
pp. 107505
Author(s):  
Hui Wang ◽  
Pan Zheng ◽  
Jie Xing
Keyword(s):  
2019 ◽  
Vol 167 (1) ◽  
pp. 231-259 ◽  
Author(s):  
Yuka Chiyoda ◽  
Masaaki Mizukami ◽  
Tomomi Yokota

1984 ◽  
Vol 20 (2) ◽  
pp. 115-124 ◽  
Author(s):  
J.G. Wijmans ◽  
S. Nakao ◽  
C.A. Smolders

1983 ◽  
Vol 29 (3) ◽  
pp. 415-438 ◽  
Author(s):  
G. J. Pert

The structure of deflagrations in one-dimensional flow is examined in detail. It is shown that the rule that deflagrations be weak or Chapman-Jouget must be obeyed unless a non-hydrodynamic discontinuity occurs. Such flows are shown to be unique and stable, once the downstream expansion is specified. It is shown that non-hydrodynamic discontinuities, if strong, are accompanied by a compression leading to a weak termination. The application to plasmas produced by laser irradiation of a solid is investigated and the flow structure in the presence of flux limitation evaluated.


1985 ◽  
Vol 3 (3) ◽  
pp. 243-250 ◽  
Author(s):  
Tan Weihan ◽  
Gu Min

The physical mechanisms of thermal flux limitation and thermal conduction inhibition in laser plasmas are analyzed. A numerical calculation accounting for the non-negative electron distribution function and pressure gradient has been given.


1980 ◽  
Vol 23 (12) ◽  
pp. 2512 ◽  
Author(s):  
D. G. Colombant ◽  
Wallace M. Manheimer

Symmetry ◽  
2020 ◽  
Vol 12 (11) ◽  
pp. 1870
Author(s):  
Pawan Kumar ◽  
Christina Surulescu

We propose a model for glioma patterns in a microlocal tumor environment under the influence of acidity, angiogenesis, and tissue anisotropy. The bottom-up model deduction eventually leads to a system of reaction–diffusion–taxis equations for glioma and endothelial cell population densities, of which the former infers flux limitation both in the self-diffusion and taxis terms. The model extends a recently introduced (Kumar, Li and Surulescu, 2020) description of glioma pseudopalisade formation with the aim of studying the effect of hypoxia-induced tumor vascularization on the establishment and maintenance of these histological patterns which are typical for high-grade brain cancer. Numerical simulations of the population level dynamics are performed to investigate several model scenarios containing this and further effects.


Author(s):  
Michael Winkler

AbstractThe flux-limited Keller–Segel–Navier–Stokes system $$\begin{aligned} \left\{ \begin{array}{lcl} n_t + u\cdot \nabla n &{}=&{} \Delta n - \nabla \cdot \Big ( n f(|\nabla c|^2) \nabla c\Big ), \\ c_t + u\cdot \nabla c &{}=&{} \Delta c - c + n, \\ u_t + (u\cdot \nabla ) u &{}=&{} \Delta u + \nabla P + n\nabla \Phi , \qquad \nabla \cdot u=0, \end{array} \right. \qquad \qquad (\star ) \end{aligned}$$ n t + u · ∇ n = Δ n - ∇ · ( n f ( | ∇ c | 2 ) ∇ c ) , c t + u · ∇ c = Δ c - c + n , u t + ( u · ∇ ) u = Δ u + ∇ P + n ∇ Φ , ∇ · u = 0 , ( ⋆ ) is considered in a smoothly bounded domain $$\Omega \subset {\mathbb {R}}^2$$ Ω ⊂ R 2 . It is shown that whenever the suitably smooth function f models any asymptotically algebraic-type saturation of cross-diffusive fluxes in the sense that $$\begin{aligned} |f(\xi )| \le K_f\cdot (\xi +1)^{-\frac{\alpha }{2}} \end{aligned}$$ | f ( ξ ) | ≤ K f · ( ξ + 1 ) - α 2 holds for all $$\xi \ge 0$$ ξ ≥ 0 with some $$K_f>0$$ K f > 0 and $$\alpha >0$$ α > 0 , for any all reasonably regular initial data a corresponding no-flux/no-flux/Dirichlet problem admits a globally defined classical solution which is bounded, inter alia, in $$L^\infty (\Omega \times (0,\infty ))$$ L ∞ ( Ω × ( 0 , ∞ ) ) with respect to all its components. By extending a corresponding result known for a fluid-free counterpart of ($$\star $$ ⋆ ), this confirms that with regard to the possible emergence of blow-up phenomena, the choice $$f\equiv const.$$ f ≡ c o n s t . retains some criticality also in the presence of fluid interaction.


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