Should we pay attention to the aberrant nerve communication between the lingual and mylohyoid nerves?

2019 ◽  
Vol 57 (4) ◽  
pp. 317-322 ◽  
Author(s):  
C. Zhan ◽  
Z. Yuan ◽  
R. Qu ◽  
L. Zou ◽  
S. He ◽  
...  
Keyword(s):  
Nerve Communication

scholarly journals The functional analysis of mast cell-nerve communication.

2001 ◽  
Vol 41 (supplement) ◽  
pp. S219
Author(s):  
M. Hashimoto ◽  
R. Suzuki ◽  
T. Furuno ◽  
N. Hirashima ◽  
R. Teshima ◽  
...  
Keyword(s):  
Functional Analysis ◽  
Mast Cell ◽  
Nerve Communication

Surgically created fourth—third cranial nerve communication: temporary success in a child with bilateral third nerve hamartomas

1999 ◽  
Vol 90 (3) ◽  
pp. 542-545 ◽  
Author(s):  
Lars Frisén ◽  
Claes von Essen ◽  
Arne Roos
Keyword(s):  
Cranial Nerve ◽  
Nerve Repair ◽  
Trochlear Nerve ◽  
Upper Eyelid ◽  
Content Type ◽  
Direct Nerve ◽  
The Right ◽  
Neuroma Formation ◽  
Third Nerve ◽  
Nerve Communication

✓ Shortly after birth, an otherwise healthy infant developed eye deviation and ptosis due to a hamartomatous lesion of the interpeduncular segment of the right oculomotor nerve. The left nerve became similarly involved when the child was 1.5 years of age. Direct nerve repair was not possible. Instead, the trochlear nerve was divided and its proximal end was attached to the distal end of the third nerve. Elevation of the upper eyelid and partial adduction of the eye developed gradually over the ensuing 3 to 5 months. Both functions were lost after an additional 2 months, presumably as a result of tumor recurrence or neuroma formation. This case report shows that surgically created fourth—third cranial nerve communication is feasible and may merit consideration under similar circumstances.

Histamine sensitivity of mesenteric afferent nerves in the rat jejunum

1998 ◽  
Vol 275 (4) ◽  
pp. G675-G680 ◽  
Author(s):  
M. E. Kreis ◽  
W. Haupt ◽  
A. J. Kirkup ◽  
D. Grundy
Keyword(s):  
Mast Cell ◽  
Small Intestine ◽  
Mast Cells ◽  
Receptor Agonist ◽  
Rat Jejunum ◽  
Functional Interaction ◽  
Afferent Nerves ◽  
Afferent Discharge ◽  
Dose Dependent ◽  
Nerve Communication

The concept of functional interaction between mast cells and intestinal afferents is gaining support. We have therefore characterized the action of histamine on jejunal afferent discharge in the anesthetized rat. Whole nerve mesenteric afferent discharge was recorded in conjunction with intestinal pressure in response to a range of histamine agonists and antagonists. Histamine at 2, 4, and 8 μmol/kg (iv) evoked a dose-dependent biphasic increase in afferent discharge together with a biphasic rise in intestinal pressure. However, these two events were mediated independently, since nifedipine (1 mg/kg) substantially reduced the intestinal pressure increase but not the afferent discharge. These responses were completely inhibited by pyrilamine (5 mg/kg) but unaffected by ranitidine (5 mg/kg) or thioperamide (2 mg/kg). Neither the selective H2receptor agonist dimaprit nor the selective H3receptor agonist R-α-methylhistamine caused any modulation of afferent discharge. We conclude that histamine stimulates an H1receptor-mediated increase in mesenteric afferent discharge that is independent of intestinal motor events. This suggests that histamine potentially acts as a mediator in mast cell-to-afferent nerve communication in the small intestine.

Nerve communication model by bio-cells and optical dipole coupling effects

2013 ◽  
Vol 41 (6) ◽  
pp. 368-375 ◽  
Author(s):  
Farrah Dilla Zainol ◽  
Nopparat Thammawongsa ◽  
Somsak Mitatha ◽  
Jalil Ali ◽  
Preecha Yupapin
Keyword(s):  
Communication Model ◽  
Coupling Effects ◽  
Dipole Coupling ◽  
Nerve Communication
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