scholarly journals Reversible conduction failure on the deep tendon reflex response recording in early Guillain-Barré syndrome

2018 ◽  
Vol 3 ◽  
pp. 159-163
Author(s):  
Antonio García ◽  
María J. Sedano ◽  
Silvia Álvarez-Paradelo ◽  
José Berciano
Keyword(s):  
Deep Tendon Reflex ◽  
Guillain Barre Syndrome ◽  
Reflex Response ◽  
Tendon Reflex ◽  
Guillain Barré Syndrome ◽  
Conduction Failure ◽  
Guillain Barré

scholarly journals Para Infectious Guillain–Barre Syndrome (GBS) in Covid-19 – A Case Report

2021 ◽  
Vol 10 (9) ◽  
pp. 659-661
Author(s):  
Swapnil Lahole ◽  
Sourya Acharya ◽  
Nitin Raisinghani ◽  
Sunil Kumar ◽  
Aishwarya Ghule
Keyword(s):  
Deep Tendon Reflex ◽  
Guillain Barre Syndrome ◽  
Atypical Pneumonia ◽  
Cardiac Damage ◽  
Limb Weakness ◽  
Gastrointestinal Complications ◽  
Tendon Reflex ◽  
Immune Mediated ◽  
Guillain Barré Syndrome ◽  
Guillain Barré

Covid-19 is a disease caused by severe acute respiratory syndrome corona virus 2 (SARS-CoV-2). SARS-CoV-2 which affects respiratory, gastrointestinal and neurological systems. It not only causes atypical pneumonia with acute respiratory distress syndrome (ARDS), but also, acute cardiac damage, acute renal failure and gastrointestinal complications.1 It is a disorder that not only presents with fever and respiratory symptoms but can involve the nervous system with varied presentations in form of cerebrovascular accident, loss of taste, loss of smell, myelopathy, neuropathy, meningitis and encephalitis.2 Some cases of Guillain–Barre syndrome (GBS) associated with SARS-CoV-2 have been reported in the literature. GBS is acute immune mediated inflammatory polyradiculopathy.3 GBS presents as limb weakness or cranial nerve weakness, loss of deep tendon reflex, autonomic dysfunction due to peripheral nerve demyelination and sensory root demyelination.

An antibody to the GM1/GalNAc-GD1a complex correlates with development of pure motor Guillain–Barré syndrome with reversible conduction failure

2013 ◽  
Vol 254 (1-2) ◽  
pp. 141-145 ◽  
Author(s):  
Go Ogawa ◽  
Ken-ichi Kaida ◽  
Motoi Kuwahara ◽  
Fumihiko Kimura ◽  
Keiko Kamakura ◽  
...  
Keyword(s):  
Guillain Barre Syndrome ◽  
Guillain Barré Syndrome ◽  
Conduction Failure ◽  
Guillain Barré ◽  
Pure Motor

A Case of Peripheral Neuropathy Combined with Leukoencephalopathy after Injection with Intrathecal Methotrexate and Vincristine.

Blood ◽  
2007 ◽  
Vol 110 (11) ◽  
pp. 4348-4348
Author(s):  
Hae-sang Lee ◽  
Hyun Joo Jung ◽  
Jun Eun Park
Keyword(s):  
Peripheral Neuropathy ◽  
Acute Lymphocytic Leukemia ◽  
Deep Tendon Reflex ◽  
Lymphocytic Leukemia ◽  
Guillain Barre Syndrome ◽  
Sensory Loss ◽  
Intrathecal Methotrexate ◽  
Gradual Improvement ◽  
Guillain Barré Syndrome ◽  
Guillain Barré

Abstract Vincristine and methotrexate are the anchor drugs in the treatment of acute lymphocytic leukemia. The neurological complications caused by either vincristine or methotrexate have been well documented. A 10 year-old girl diagnosed acute lymphocytic leukemia without central nervous involvement and treated with CCG 1882 based therapy. She suffered from ascending paralysis from lower extremities without seizure or mental change three days after the fifth dose of vincristine and the fifth dose of intrathecal methotrexate during consolidation chemotherapy composed of 3-drug(vincristine, methotrexate, and L-asparaginase). Two days later, she developed dysarthria and respiratory discomfort. Her neurological examination showed a symmetric, flaccid, quadriparesis with loss of deep tendon reflex. But, she did not have sensory loss. We thought Guillain-Barre syndrome as first impression and carried out diagnostic evaluation. Viral study revealed no abnormality and cerebrospinal fluid study was not consistent with Guillain-Barre syndrome or infectious disease. Diffusion weighted magnetic resonance imaging showed a well-demarcated area of moderately high signal intensity in the white mater of the both parietal lobes. Neuromuscular conduction test revealed severe amplitude reduction in motor nerve action potential meaning on peripheral polyneuropathy. These findings were corresponded with drug-induced peripheral neuropathy and chemotherapy-related leukoencephalopathy that was detected incidentally. She did not receive further doses of vincristine and intrathecal methotrexate during the consolidation phase. There was gradual improvement of the weakness in the both lower and upper limbs for 3 months. She was recovery to walk with minimal support. We report peripheral neuropathy combined with subclinical leukoencephalopahty after injection with intrathecal methotrexate and vincristine.

Electrodiagnosis of reversible conduction failure in Guillain–Barré syndrome

2017 ◽  
Vol 56 (5) ◽  
pp. 919-924 ◽  
Author(s):  
Yee‐Cheun Chan ◽  
Aubrey M. Punzalan‐Sotelo ◽  
Therimadasamy A. Kannan ◽  
Nortina Shahrizaila ◽  
Thirugnanam Umapathi ◽  
...  
Keyword(s):  
Guillain Barre Syndrome ◽  
Guillain Barré Syndrome ◽  
Conduction Failure ◽  
Guillain Barré

Reversible conduction failure in pharyngeal-cervical-brachial variant of guillain-barré syndrome

2010 ◽  
Vol 42 (4) ◽  
pp. 608-612 ◽  
Author(s):  
Margherita Capasso ◽  
Francesca Notturno ◽  
Claudia Manzoli ◽  
Nobuhiro Yuki ◽  
Antonino Uncini
Keyword(s):  
Guillain Barre Syndrome ◽  
Guillain Barré Syndrome ◽  
Conduction Failure ◽  
Guillain Barré

scholarly journals Clinical features of Guillain–Barré syndrome patients with elevated serum creatine kinase levels

2020 ◽  
Author(s):  
Takafumi Hosokawa ◽  
Hideto Nakajima ◽  
Taiki Sawai ◽  
Yoshitsugu Nakamura ◽  
Eri Sano ◽  
...  
Keyword(s):  
Creatine Kinase ◽  
Clinical Features ◽  
Axonal Neuropathy ◽  
Elevated Serum ◽  
Serum Creatine Kinase ◽  
Guillain Barre Syndrome ◽  
Guillain Barré Syndrome ◽  
Conduction Failure ◽  
Electrophysiological Examination ◽  
Guillain Barré

Abstract Background: It is not well defined whether Guillain–Barré syndrome (GBS) patients with elevated serum creatine kinase (CK) levels have characteristic clinical features and are related to the subgroups of GBS. Methods: We retrospectively studied 51 consecutive patients with GBS, who visited our hospital, and compared clinical, laboratory and electrophysiological findings between patients with and without elevated CK levels. Results: Of 51 patients, 14 patients (27%) showed an elevation of serum CK levels. When compared with patients with the normal CK levels, the ratios of male, antecedent infections, and anti-GM1 antibody positivity were significantly higher in patients with elevated CK levels. The ratios of hypoesthesia, cranial nerve involvement, and urinary retention were significantly less in patients with elevated CK levels. There were no significant differences in disability at peak between two groups. In the electrophysiological examination, sensory nerve abnormalities were not observed. Although some patients with elevated CK levels showed prolongation of distal motor latencies (DMLs) and increase of durations in the initial examination, development of the prolongation of DMLs and increase of durations was not observed in the follow-up examinations. The findings were consistent with acute motor axonal neuropathy (AMAN) with reversible conduction failure (RCF) but not acute inflammatory demyelinating polyneuropathy (AIDP). Conclusions: The results suggest that the GBS patients with elevated CK levels represent not a group of AIDP but a group of AMAN with axonal degeneration or RCF even though the initial electrophysiological examination shows AIDP pattern. Key words: Creatine kinase, Guillain-Barré syndrome, AIDP, AMAN, reversible conduction failure

scholarly journals Limbic Encephalitis following Guillain-Barré Syndrome Associated with Mycoplasma Infection

2019 ◽  
Vol 11 (1) ◽  
pp. 17-23
Author(s):  
Miwa Yoshino ◽  
Jun Muneuchi ◽  
Eiko Terashi ◽  
Yu Yoshida ◽  
Yukitoshi Takahashi ◽  
...  
Keyword(s):  
Plasma Exchange ◽  
Limbic Encephalitis ◽  
Deep Tendon Reflex ◽  
Brain Magnetic Resonance Imaging ◽  
Immunoglobulin M ◽  
Guillain Barre Syndrome ◽  
High Dose ◽  
Mycoplasma Infection ◽  
Guillain Barré Syndrome ◽  
Guillain Barré

A 12-year-old girl was admitted to the authors’ hospital due to muscle weakness, gait disturbance, dysarthria, dysphagia, and diplopia. She experienced prodromal fever 10 days before admission. On examination, deep tendon reflex was absent in the extremities, and nerve conduction velocity was decreased in the ulnar nerve. She was diagnosed with Guillain-Barré syndrome (GBS). Despite steroid pulse therapy following administration of intravenous high-dose γ-globulin, clinical manifestations remained unchanged. Therefore, plasma exchange was performed on day 10 of the illness. The titer of serum Mycoplasma immunoglobulin M level was increased. Immunological testing was positive for serum anti-galactocerebroside C antibody. On day 18 of the illness, however, she developed generalized convulsion. Brain magnetic resonance imaging revealed high intensity in the medial temporal lobes, including the hippocampus and thalamus on T2-weighted intensity imaging, which was consistent with limbic encephalitis. Further immunological tests revealed positivity for anti-N-methyl-D-aspartate-type glutamate receptor antibody in the cerebrospinal fluid. She was treated with additional plasma exchange; however, she exhibited residual manifestations including short-term memory disorder, emotional incontinence, and convulsions. This article describes a notable case of limbic encephalitis following GBS associated with prodromal Mycoplasma infection. It is interesting that autoimmune encephalopathy is concomitant with autoimmune polyneuropathy subsequent to Mycoplasma infection.

PATTERNS OF CONDUCTION FAILURE IN THE GUILLAIN-BARRE SYNDROME

Brain ◽  
1988 ◽  
Vol 111 (2) ◽  
pp. 405-416 ◽  
Author(s):  
F.G.A. VAN DER MECHÉ ◽  
J. MEULSTEE ◽  
M. VERMEULEN ◽  
A. KIEVIT
Keyword(s):  
Guillain Barre Syndrome ◽  
Guillain Barré Syndrome ◽  
Conduction Failure ◽  
Guillain Barré

Comment on paraparetic Guillain-Barré syndrome: Non-demyelinating reversible conduction failure restricted to the lower limbs

2016 ◽  
Vol 55 (3) ◽  
pp. 445-446 ◽  
Author(s):  
José Berciano ◽  
Elena Gallardo ◽  
Pedro Orizaola ◽  
Enrique Marco de Lucas ◽  
Antonio García ◽  
...  
Keyword(s):  
Lower Limbs ◽  
Guillain Barre Syndrome ◽  
Guillain Barré Syndrome ◽  
Conduction Failure ◽  
Guillain Barré
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