Human Leukocyte Antigen-G Inhibits Neutrophil Adhesion to Coronary Artery Endothelial Cells Injured by Tumor Necrosis Factor Alpha

2013 ◽  
Vol 32 (4) ◽  
pp. S300 ◽  
Author(s):  
A.G. Mociornita ◽  
L.C. Tumiati ◽  
P.C. Papageorgiou ◽  
L. Grosman-Rimon ◽  
A. Ghashghai ◽  
...  
Keyword(s):  
Endothelial Cells ◽  
Coronary Artery ◽  
Tumor Necrosis ◽  
Human Leukocyte ◽  
Neutrophil Adhesion ◽  
Necrosis Factor Alpha ◽  
Leukocyte Antigen ◽  
Factor Alpha ◽  
Human Leukocyte Antigen G ◽  
Necrosis Factor

scholarly journals The Extracellular Adherence Protein from Staphylococcus aureus Inhibits Neutrophil Binding to Endothelial Cells

2004 ◽  
Vol 72 (10) ◽  
pp. 6164-6167 ◽  
Author(s):  
Axana Haggar ◽  
Cecilia Ehrnfelt ◽  
Jan Holgersson ◽  
Jan-Ingmar Flock
Keyword(s):  
Staphylococcus Aureus ◽  
Endothelial Cells ◽  
Tumor Necrosis Factor ◽  
Tumor Necrosis Factor Alpha ◽  
Tumor Necrosis ◽  
Neutrophil Adhesion ◽  
Necrosis Factor Alpha ◽  
Factor Alpha ◽  
Necrosis Factor ◽  
Adhesion Assays

ABSTRACT Extracellular adherence protein (Eap) from Staphylococcus aureus inhibits the adherence of neutrophils to nonstimulated and tumor necrosis factor alpha-stimulated endothelial cells in both static adhesion assays and flow adhesion assays. Consequently, Eap also impaired their transendothelial migration. During an S. aureus infection, Eap may thus serve to reduce inflammation by inhibiting neutrophil adhesion and extravasation.

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