scholarly journals 234 Migration of a Right Ventricular Pacing Lead Causing Left Haemothorax Via the Interventricular Septum and Left Ventricular Free Wall

2020 ◽  
Vol 29 ◽  
pp. S139
Author(s):  
M. MacPherson ◽  
A. Teh
Keyword(s):  
Right Ventricular ◽  
Interventricular Septum ◽  
Left Ventricular ◽  
Ventricular Free Wall ◽  
Free Wall ◽  
Ventricular Pacing ◽  
Left Ventricular Free Wall ◽  
Right Ventricular Pacing ◽  
Pacing Lead

Comparison of left ventricular free wall and septal diastolic compliance in the dog

1978 ◽  
Vol 234 (4) ◽  
pp. H392-H398 ◽  
Author(s):  
R. S. Kent ◽  
T. E. Carew ◽  
M. M. LeWinter ◽  
J. W. Covell
Keyword(s):  
Right Ventricular ◽  
Diastolic Pressure ◽  
Left Ventricular ◽  
Ventricular Free Wall ◽  
Segment Length ◽  
Free Wall ◽  
Left Ventricular Free Wall ◽  
Average Maximum ◽  
Diastolic Compliance ◽  
Regional Compliance

Septal to free wall dimensions are frequently employed for the analysis of diastolic compliance. However, the diastolic properties of these anatomically distinct regions of left ventricle are not well characterized. Regional compliance was studied in eight open-chest anesthetized dogs. Pairs of 2-mm-diameter piezoelectric crystals were implanted in the left ventricular free wall or septum 1.38 +/- 0.06 cm apart at a midwall location 58% +/- 1.9 of the left ventricular endocardial-epicardial or left ventricular endocardial-right ventricular endocardial distance. Left ventricular end-diastolic pressure was increased from an average of 8.1-21.0 mmHg, with a resulting average maximum end-diastolic strain of 11% (end-diastolic (ED) segment length/control ED length). Regional stiffness was assessed at all sites based on the relationship between left ventricular end-diastolic pressure and regional strain. Neither strain nor calculated stiffness coefficients differed significantly among the three sites. Septal transmural pressure (left ventricular end-diastolic pressure--right ventricular end-diastolic pressure) was nearly constant as left ventricular end-diastolic pressure increased during volume infusion and thus did not account for the observed septal strain.

scholarly journals Correlation between the periodontal treatment and ecocardiographic changes in adult dogs

Interação ◽  
2021 ◽  
Vol 21 (3) ◽  
pp. 43-57
Author(s):  
Daniel Paulino Júnior ◽  
Marina Queiroz Derruci ◽  
Leandro Zuccolotto Crivelenti ◽  
Brenda Faria Santos Gomes Parreira ◽  
Fernanda Gosuen Gonçalves Dias
Keyword(s):  
Left Ventricle ◽  
Periodontal Disease ◽  
Dental Treatment ◽  
Oral Treatment ◽  
Interventricular Septum ◽  
Left Ventricular ◽  
Periodontal Treatment ◽  
Ventricular Free Wall ◽  
Free Wall ◽  
Left Ventricular Free Wall

Periodontal disease is commonly diagnosed in dogs and has been associated with systemic lesions in several organs, however, studies correlating this oral condition with heart disease in the canine species are still scarce in the scientific literature. Thus, the objective of the present study was to evaluate such correlation in adult dogs, through serial echocardiographic exams and, nonetheless, to analyze the influence of dental treatment on cardiac function. For this, 60 dogs were used, distributed in two groups, the control (CG, n = 30: presence of periodontal disease, not submitted to oral treatment) and the treated (GT, n = 30: presence of periodontal disease and submitted to oral treatment). The animals of the CG were paired with those of the GT in terms of race, weight, age and degree of periodontal disease. Baseline echocardiographic examinations (D0) of the CG were performed and after 30 days (D30). In GT, examinations were also performed at D0 and D30, however, they were submitted to periodontal treatment after baseline measurements. The echocardiographic exams were performed at the Veterinary Cardiology Laboratory of the Veterinary Hospital of the University of Franca, to record and analyze the interventricular septum in diastole (SIVd), interventricular septum in systole (SIVs), diameter of the left ventricle in diastole (DVEd), left ventricular diameter in systole (EVDs), left ventricular free wall in diastole (PVEd) left ventricular free wall in systole (PVEs) shortening fraction of left ventricle (FS), final diastolic volume (VDF), final stroke volume (VSF), ejection fraction (EF), maximum pulmonary artery pressure gradient (max. GP AP) and left atrial and aortic artery diameter ratio (EA-AO). The results of the CG were statistically compared with those of the GT by means of simple analysis of variance (ANOVA). The data obtained regarding the SIVs, DVEd and PVEs parameters of the GT dogs at D30 showed a statistically significant decrease (p<0.05) when compared to the D30 of the CG. Thus, given the established methodology and the results found, it is possible to infer that periodontal treatment may benefit some cardiac parameters of adult dogs, improving their quality of life and survival.

Necrotic and apoptotic myocyte cell death in the aging heart of Fischer 344 rats

1996 ◽  
Vol 271 (3) ◽  
pp. H1215-H1228 ◽  
Author(s):  
J. Kajstura ◽  
W. Cheng ◽  
R. Sarangarajan ◽  
P. Li ◽  
B. Li ◽  
...  
Keyword(s):  
Cell Death ◽  
Right Ventricle ◽  
Interventricular Septum ◽  
Left Ventricular ◽  
Ventricular Free Wall ◽  
Terminal Deoxynucleotidyl Transferase ◽  
Free Wall ◽  
Fischer 344 Rats ◽  
Left Ventricular Free Wall ◽  
Fischer 344

To determine the effects of aging on myocyte cell death, Fischer 344 rats at 3, 7, 12, 16, and 24 mo of age were injected with myosin monoclonal antibody for the localization and quantification of necrotic myocyte cell death in the left ventricle, interventricular septum, and right ventricle. Conversely, the presence of DNA strand breaks in myocyte nuclei, indicative of programmed cell death, was evaluated by the terminal deoxynucleotidyl transferase assay and confirmed by DNA laddering. Myocyte necrosis, which involved nearly 1,000 myocytes in the left ventricular free wall at 3 mo, progressively increased with aging, reaching a value of 13,600 myocytes at 24 mo. Corre- sponding values in the interventricular septum were 300 and 9,400 myocytes. In the right ventricle, there were 270 necrotic myocytes at 3 mo and 9,000 at 24 mo. Programmed myocyte cell death was restricted to the left ventricular free wall and included 140 cells at 3 mo. This form of myocyte cell death increased at the subsequent age intervals, resulting in the involvement of 874 cells at 24 mo. The combination of necrosis and apoptosis in the left ventricular free wall was associated with 1,150 cells dying at 3 mo and 14,500 at 24 mo. In conclusion, myocyte cell death, apoptotic and necrotic in nature, constitutes an important determinant of the aging process, possibly mediating the occurrence of ventricular dysfunction and failure in the old heart.

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