A complement receptor for opsonized immune complexes on erythrocytes from Oncorhynchus mykiss but not Ictalarus punctatus

2006 ◽  
Vol 43 (10) ◽  
pp. 1595-1603 ◽  
Author(s):  
Barbara Schraml ◽  
M. Angeles Baker ◽  
Brian D. Reilly

1997 ◽  
Vol 82 (1) ◽  
pp. 49-59 ◽  
Author(s):  
Ronald P. Taylor ◽  
Polly J. Ferguson ◽  
Edward N. Martin ◽  
James Cooke ◽  
Kirsten L. Greene ◽  
...  


1980 ◽  
Vol 152 (4) ◽  
pp. 905-919 ◽  
Author(s):  
F M Griffin

The effects of ingestion of soluble immune complexes upon macrophage phagocytic function was studied. Ingestion of immune complexes severely impaired the macrophage's ability to ingest IgG-coated particles but did not alter its ability to interact with particles by means other than its Fc receptors. Treatment of macrophages that had ingested immune complexes with supernates containing the previously described lymphokine that augments macrophage complement receptor function failed to enhance the cells' interaction with either IgG-coated erythrocytes or zymosan particles but markedly enhanced their ability to phagocytize via their complement receptors. The possible significance of these findings in immunologically mediated inflammation is discussed.



1992 ◽  
Vol 24 (2) ◽  
pp. 101-106 ◽  
Author(s):  
M. Pascual ◽  
J.A. Schifferli


1979 ◽  
Vol 150 (3) ◽  
pp. 607-621 ◽  
Author(s):  
J Michl ◽  
M M Pieczonka ◽  
J C Unkeless ◽  
S C Silverstein

We have examined the Fc- and complement-receptor function of resident and thioglycollate-elicited mouse peritoneal macrophages plated on surfaces coated with rabbit antibody-antigen complexes and with complement. We derive four major conclusions from these studies. (a) The trypsin-resistant Fc receptors of resident and thioglycollate-elicited macrophages are completely modulated when these cells are plated on rabbit antibody-antigen complexes. Residual Fc receptor activity is a result of the incomplete modulation of trypsin-sensitive IgG2a receptors. (b) The complement receptors of thioglycollate-elicited macrophages, but not of resident macrophages, are modulated when these cells are plated on complement-coated surfaces. The capacity of the two cell types to modulate their complement receptors is correlated with their ability to ingest complement-coated erythrocytes. (c) The complement and Fc receptors of both types of macrophages move independently of one another. (d) Complement masks the Fc segments of IgG in immune complexes thereby rendering them ineffective as ligands for macrophage Fc receptors.



2002 ◽  
Vol 270 (2) ◽  
pp. 183-197 ◽  
Author(s):  
Andrea L Henderson ◽  
Margaret A Lindorfer ◽  
Adam D Kennedy ◽  
Patricia L Foley ◽  
Ronald P Taylor


2002 ◽  
Vol 105 (1) ◽  
pp. 36-47 ◽  
Author(s):  
Maria L. Craig ◽  
Alexander J. Bankovich ◽  
Ronald P. Taylor




1999 ◽  
Vol 36 (13-14) ◽  
pp. 827-835 ◽  
Author(s):  
Alessandra Nardin ◽  
Margaret A Lindorfer ◽  
Ronald P Taylor


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