Late postoperative cervical spinal cord herniation with pseudomeningocele

Author(s):  
Yulong Wang ◽  
Lian Zeng ◽  
Xiaodong Guo
2020 ◽  
Vol 137 ◽  
pp. 1-7 ◽  
Author(s):  
Andrew S. Jack ◽  
Jens R. Chapman ◽  
Praveen V. Mummaneni ◽  
Line G. Jacques ◽  
Carter S. Gerard

2014 ◽  
Vol 14 (5) ◽  
pp. 856-857 ◽  
Author(s):  
Stefan M. Iencean ◽  
Ion Poeata

2020 ◽  
Author(s):  
Anthony Diaz ◽  
S Shelby Burks ◽  
Richard Fisher ◽  
Allan D Levi

Abstract Spinal cord herniation (SCH) is a rare condition that is typically of idiopathic origin. Although SCH is mostly found in the thoracic region because of a dural defect, there are some reports of cervical SCH following surgery or trauma.1-3 Spinal cord tethering can be a result of SCH or as a standalone issue.4,5 These conditions can lead to progressive neurological deficits, including numbness, gait disturbances, and decreased muscle strength, requiring surgical correction. There are limited reports of surgical procedures for ventral SCHs. Several reports exist using a ventral approach for intradural tumors, but it is not commonly employed because of the inability to obtain adequate dural closure.6 Much of the literature on SCH comes from idiopathic and congenital cases in the thoracic spine.7,8 Posterior and posterolateral approaches for a ventral thoracic SCH have been described, as well as an anterior approach for a ventral cervical SCH.9-12 In this video, we describe a posterior approach for a ventral cervical SCH. A 38-yr-old male presented with progressive cervical myelopathy 9 yr after a C2-C3 schwannoma resection requiring an anterior approach and corpectomy of C3 with partial corpectomies of C2 and C4. A preoperative magnetic resonance imaging showed a ventrally herniated spinal cord at the top of the C3 vertebral body and below the C4 vertebral body. Informed consent was obtained. The posterior surgical approach involved a C1-C5 laminectomy, sectioning the dentate ligament, ventral cord untethering, removal of residual tumor, and placement of a ventral sling. A significant improvement in sensory and motor function was observed postoperatively.


Cureus ◽  
2019 ◽  
Author(s):  
Nicolas K Khattar ◽  
Andrew M Donovan ◽  
Brent G Oxford ◽  
Shawn W C Adams ◽  
Thomas J Altstadt

2016 ◽  
Vol 16 (8) ◽  
pp. e507-e508 ◽  
Author(s):  
C. Rory Goodwin ◽  
Nancy Abu-Bonsrah ◽  
Shamsudini Hashi ◽  
Akwasi Ofori Boah ◽  
Daniel M. Sciubba

Author(s):  
L. Vacca-Galloway ◽  
Y.Q. Zhang ◽  
P. Bose ◽  
S.H. Zhang

The Wobbler mouse (wr) has been studied as a model for inherited human motoneuron diseases (MNDs). Using behavioral tests for forelimb power, walking, climbing, and the “clasp-like reflex” response, the progress of the MND can be categorized into early (Stage 1, age 21 days) and late (Stage 4, age 3 months) stages. Age-and sex-matched normal phenotype littermates (NFR/wr) were used as controls (Stage 0), as well as mice from two related wild-type mouse strains: NFR/N and a C57BI/6N. Using behavioral tests, we also detected pre-symptomatic Wobblers at postnatal ages 7 and 14 days. The mice were anesthetized and perfusion-fixed for immunocytochemical (ICC) of CGRP and ChAT in the spinal cord (C3 to C5).Using computerized morphomety (Vidas, Zeiss), the numbers of IR-CGRP labelled motoneurons were significantly lower in 14 day old Wobbler specimens compared with the controls (Fig. 1). The same trend was observed at 21 days (Stage 1) and 3 months (Stage 4). The IR-CGRP-containing motoneurons in the Wobbler specimens declined progressively with age.


Sign in / Sign up

Export Citation Format

Share Document