scholarly journals CEREBRAL VASOSPASM AND UNRUPTURED ANEURYSM IN THUNDERCLAP HEADACHE

The Lancet ◽  
1988 ◽  
Vol 332 (8618) ◽  
pp. 1020 ◽  
Author(s):  
E.F.M. Wijdicks ◽  
H. Kerkhoff ◽  
J. Van Gijn
Cephalalgia ◽  
2003 ◽  
Vol 23 (9) ◽  
pp. 854-859 ◽  
Author(s):  
Yi-Chu Liao ◽  
Jong-Ling Fuh ◽  
Jiing-Feng Lirng ◽  
Shiang-Ru Lu ◽  
Zin-An Wu ◽  
...  

Bathing headache is rarely described in literature. We report four middle-aged Taiwanese women who developed severe throbbing headache with maximum intensity of onset during bathing. Diffuse cerebral vasospasm was demonstrated in one of them. All their headaches resolved spontaneously ( n = 1) or after nimodipine treatment ( n = 3). Except for one patient with vasospasm in whom reversible posterior leukoencephalopathy and an asymptomatic cerebellar infarction developed, the others recovered without any complications. The clinical profile of bathing headache points to idiopathic thunderclap headache. It may not be as benign as previously reported. Nimodipine might be effective in treatment of this special headache syndrome.


Neurosurgery ◽  
1985 ◽  
Vol 17 (5) ◽  
pp. 792-796 ◽  
Author(s):  
Stephen M. Bloomfield ◽  
V. K. H. Sonntag

Abstract We present the case of a patient who developed significant vasospasm on the 9th postoperative day after the uneventful clipping of an unruptured asymptomatic berry aneurysm. We discuss the literature and potential mechanisms for vasospasm in the absence of subarachnoid hemorrhage.


2020 ◽  
pp. 1-6 ◽  
Author(s):  
Pietro Fiaschi ◽  
Anania Pasquale ◽  
Ceraudo Marco ◽  
D’Andrea Alessandro ◽  
Pietro Fiaschi ◽  
...  

Background and Importance: Angiographic-proven and clinically-evident cerebral vasospasm (CVS) after uneventful elective clipping of unruptured intracranial aneurysm (UIA) is a very rare and often underestimated event. To date, the knowledge of risk factors, pathophysiology, and demographic characteristics of these conditions are solely relegated to few case reports. With the aim of better characterize shared features and mechanism that could be involved in such event we also performed a review of the present literature and analyzed aneurysm’s features, surgical factors, treatments, recovery and of all reported cases of CVS after elective clipping. Clinical Presentation: We report a case of a cerebral vasospasm following elective clipping of a middle cerebral artery (MCA) bifurcation aneurysm in a 59-year-old woman who smoked next days after treatment, despite medical advice. We found ten cases comparable to ours with angiographic-proven and clinically evident cerebral vasospasm after uneventful elective clipping. Conclusion: Classic mechanisms of CVS following SAH have been widely studied. In all the cases we analyzed, no subarachnoid bleeding occurred, as demonstrated in pre and postoperative CT scans and intraoperatively. Various theories on the possible mechanism have been advanced. It seems reasonable that CVS following elective clipping of unruptured aneurysm is a multifactorial phenomenon. Although its pathogenesis is unclear, clinicians should keep in mind the existence of this event, that is rare, but it could be seen in the clinical practice of every neurosurgery ward. In our opinion, it’s worth to know this possible post-operative complication because, when suspected clinical signs and symptoms of delayed ischemic neurological deficit (DIND) arise after elective clipping, it’s important to make an early diagnosis of CVS owing to early treatments are critical to improve clinical outcome


Cephalalgia ◽  
2006 ◽  
Vol 26 (5) ◽  
pp. 530-536 ◽  
Author(s):  
S-P Chen ◽  
J-L Fuh ◽  
J-F Lirng ◽  
S-J Wang

Primary thunderclap headache (TCH) is sometimes associated with cerebral vasospasm. However, the role of vasospasm in relation to the development of reversible or irreversible posterior leukoencephalopathy among patients with primary TCH has never been fully addressed. This paper includes a report on a 51-year-old woman with primary TCH complicated with posterior leukoencephalopathy and a literature review of 16 further patients with the same illness. Their magnetic resonance or conventional angiographic findings were clearly described. Our review found that all these 17 patients showed evidence of cerebral vasospasm. Eleven (65%) of them developed permanent ischaemic infarctions, almost exclusively located at the watershed zones. We suggest that the presence of vasospasm might be requisite for posterior leukoencephalopathy as well as for permanent infarctions in these patients. Therefore, searching for any clue of vasospasm is mandatory in treatment of patients with primary TCH. Absence of an accompanying vasospasm might predict a good outcome.


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