Effect of PGE in multiple experimental models IX: in vivo effects of PGE on immune response of leucocytes to wounds

Burns ◽  
1994 ◽  
Vol 20 (2) ◽  
pp. 132-135 ◽  
Author(s):  
Y.-L. Dong ◽  
D.N. Herndon ◽  
T.Z. Yan ◽  
J.P. Waymack
1993 ◽  
Vol 15 (4) ◽  
pp. 415-428 ◽  
Author(s):  
Vito Covelli ◽  
Angela B. Maffione ◽  
Beatrice Greco ◽  
Bruno Cannuscio ◽  
Rosa Calvello ◽  
...  

1996 ◽  
Vol 3 (4) ◽  
pp. 381-386 ◽  
Author(s):  
Richard J. Barth ◽  
Michael A. Coppola ◽  
William R. Green

1988 ◽  
Vol 48 (2) ◽  
pp. 238-246 ◽  
Author(s):  
A. Scordamaglia ◽  
G. Ciprandi ◽  
S. Ruffoni ◽  
Maria Caria ◽  
Francesca Paolieri ◽  
...  

2007 ◽  
Vol 84 (1) ◽  
pp. 92-102 ◽  
Author(s):  
Beatrice Gagnaire ◽  
Melanie Gay ◽  
Arnaud Huvet ◽  
Jean-Yves Daniel ◽  
Denis Saulnier ◽  
...  

1995 ◽  
Vol 6 (4) ◽  
pp. 1143-1150 ◽  
Author(s):  
M H Sayegh ◽  
L A Turka

It is now accepted that T cells need two signals for full activation. The first is the foreign antigen itself presented by self-major histocompatibility complex and thus provides antigen specificity to the immune response. The second is a "costimulatory" signal, the best-characterized of which is provided through the T cell accessory molecule CD28. In vitro, the blockade of costimulatory signals inhibits T cell activation and induces a state of antigen-specific unresponsiveness. In vivo, agents that block CD28-mediated costimulation have proved extremely effective in inhibiting the immune response in experimental models of transplantation and autoimmune disease, providing novel strategies for use in clinical trials in the near future.


1994 ◽  
Vol 16 (3) ◽  
pp. 205-215 ◽  
Author(s):  
R.U. Chukwuocha ◽  
E. Reyes ◽  
S. Tokuda

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