scholarly journals Involvement of parental imprinting in the antisense regulation of onco-miR-372-373

2013 ◽  
Vol 4 (1) ◽  
Author(s):  
Yonatan Stelzer ◽  
Ido Sagi ◽  
Nissim Benvenisty
Keyword(s):  
Parental Imprinting ◽  
Antisense Regulation

Antisense Regulation of Oncogenes in Human Cancer

1996 ◽  
Vol 7 (3-4) ◽  
pp. 151-190 ◽  
Author(s):  
Tapas Mukhopadhyay ◽  
Jack A. Roth
Keyword(s):  
Human Cancer ◽  
Antisense Regulation

scholarly journals Immunocytochemical Localization of Methylated DNA in Human Pachytene Oocytes and Questions of Parental Imprinting

Caryologia ◽  
1988 ◽  
Vol 41 (3-4) ◽  
pp. 189-200 ◽  
Author(s):  
Georgiana M. Jagiello ◽  
Jye-Siung Fang ◽  
Umadevi Tantravahi ◽  
Mercedes B. Ducayen ◽  
Bernard F. Erlanger
Keyword(s):  
Immunocytochemical Localization ◽  
Methylated Dna ◽  
Parental Imprinting

Evidence for a new form of dominant negative imprinting at the ovine callipyge locus

1996 ◽  
Vol 1996 ◽  
pp. 51-51
Author(s):  
N.E. Cockett ◽  
S.P. Jackson ◽  
T.L. Shay ◽  
F. Farnir ◽  
S. Berghmans ◽  
...  
Keyword(s):  
Strong Evidence ◽  
Distal Part ◽  
Segregation Ratio ◽  
Dominant Negative ◽  
Chromosome 18 ◽  
Dominant Mutation ◽  
Independent Segregation ◽  
Parental Imprinting ◽  
New Form ◽  
Unusual Phenotype

In 1983, the occurrence of a ram exhibiting an exceptional muscular development was reported in a flock of Dorset sheep. This unusual phenotype was transmitted to some of the offspring, and subsequent matings involving hypermuscled male descendents of the founder ram and normal ewes clearly demonstrated a 50%-50%, sex-independent segregation ratio of the trait. It was therefore postulated that a dominant mutation (CLPG versus the normal clpg allele) at the autosomal “callipyge“ locus was responsible for this hypertrophy. Using the same family material, the callipyge locus was unambiguously mapped to the distal part of ovine chromosome 18 (Cockett et al., 1994).In this paper, we present very strong evidence that the callipyge locus is subjected to a previously undescribed form of parental imprinting. It is indeed demonstrated that animals only express the callipyge phenotype if they have inherited the CLPG mutation from their sire, not if it is transmitted by the dam.

Photodynamic Antisense Regulation of Human Cervical Carcinoma Cell

2005 ◽  
pp. 389-396
Author(s):  
Akira Murakami ◽  
Asako Yamayoshi ◽  
Reiko Iwase ◽  
Jun-ichi Nishida ◽  
Tetsuji Yamaoka ◽  
...  
Keyword(s):  
Cervical Carcinoma ◽  
Carcinoma Cell ◽  
Human Cervical Carcinoma ◽  
Antisense Regulation

Parental imprinting and epigenetic programming of the mouse genome: long lasting consequences for development and phenotype

1993 ◽  
pp. 367-376
Author(s):  
W. Reik ◽  
H. Sasaki ◽  
A. Ferguson-Smith ◽  
R. Feil ◽  
L. Bowden ◽  
...  
Keyword(s):  
Mouse Genome ◽  
Epigenetic Programming ◽  
Parental Imprinting

scholarly journals Extensive, clustered parental imprinting of protein-coding and noncoding RNAs in developing maize endosperm

2011 ◽  
Vol 108 (50) ◽  
pp. 20042-20047 ◽  
Author(s):  
M. Zhang ◽  
H. Zhao ◽  
S. Xie ◽  
J. Chen ◽  
Y. Xu ◽  
...  
Keyword(s):  
Noncoding Rnas ◽  
Maize Endosperm ◽  
Protein Coding ◽  
Parental Imprinting

A novel pleckstrin homology-related gene family defined by Ipl/Tssc3, TDAG51, and Tih1: tissue-specific expression, chromosomal location, and parental imprinting

1999 ◽  
Vol 10 (12) ◽  
pp. 1150-1159 ◽  
Author(s):  
Dale Frank ◽  
Cathy L. Mendelsohn ◽  
Emilio Ciccone ◽  
Kristian Svensson ◽  
Rolf Ohlsson ◽  
...  
Keyword(s):  
Gene Family ◽  
Chromosomal Location ◽  
Related Gene ◽  
Pleckstrin Homology ◽  
Specific Expression ◽  
Tissue Specific ◽  
Tissue Specific Expression ◽  
Parental Imprinting

scholarly journals GCSH antisense regulation determines breast cancer cells’ viability

2018 ◽  
Vol 8 (1) ◽  
Author(s):  
Anna Adamus ◽  
Petra Müller ◽  
Bente Nissen ◽  
Annika Kasten ◽  
Stefan Timm ◽  
...  
Keyword(s):  
Breast Cancer ◽  
Cancer Cells ◽  
Breast Cancer Cells ◽  
Antisense Regulation

scholarly journals Parental imprinting effect at the INS-IGF2 diabetes susceptibility locus

Diabetologia ◽  
1995 ◽  
Vol 38 (6) ◽  
pp. 715-719 ◽  
Author(s):  
C. Polychronakos ◽  
A. Kukuvitis ◽  
N. Giannoukakis ◽  
E. Colle
Keyword(s):  
Susceptibility Locus ◽  
Diabetes Susceptibility ◽  
Parental Imprinting
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