Differential Diagnosis of Acute Facial Paralysis

2014 ◽  
1981 ◽  
Vol 89 (5) ◽  
pp. 841-848 ◽  
Author(s):  
Mark May ◽  
Thomas J. Fria ◽  
Frank Blumenthal ◽  
Hugh Curtin

The differential diagnosis in 170 patients between birth and 18 years of age is reviewed. There are a number of obvious physical findings and historical features that allow one to make a diagnosis rather quickly. Pain, vesicles, a red pinna, vertigo, and sensorineural hearing loss suggest herpes zoster oticus. Slow progression beyond three weeks, recurrent facial paralysis involving the same side, facial twitching, weakness, or no return of function after six months indicate a neoplasm. Bilateral simultaneous facial paralysis indicates a cause other than Bell's palsy, such as Guillain-Barré syndrome, pseudobulbar palsy, sarcoidosis, and leukemia. Recurrent facial paralysis associated with a fissured tongue, facial edema, and a positive family history should suggest Melkersson-Rosenthal syndrome.


PEDIATRICS ◽  
1972 ◽  
Vol 50 (1) ◽  
pp. 171-171
Author(s):  
Saul D. Roskes

The review paper by Drs. Manning and Adour on facial paralysis in childhood was illuminating in terms of differential diagnosis. However, one very important consideration in the assessment of children who present with peripheral facial paralysis is the possibility that it may be the initial manifestation of severe hypertension in children. It is presumed that the paralysis is due to hemorrhage within the facial canal. Because hypertension in childhood very frequently has an etiology which is surgically or medically remediable the index of suspicion for its presence must be maintained at a high level.


2016 ◽  
Vol 22 (4) ◽  
pp. 196-198 ◽  
Author(s):  
Nihan Hande Akçakaya ◽  
Meltem Hale Alpsan Gökmen ◽  
Yeşim Gülşen Parman ◽  
Feza Deymeer ◽  
Piraye Oflazer

1988 ◽  
Vol 98 (1) ◽  
pp. 53-59 ◽  
Author(s):  
Carl Snyderman ◽  
Mark May ◽  
Michael A. Berman ◽  
Hugh D. Curtin

Traumatic neuromas (TN) are benign proliferations of neural tissue that may occur without disruption of the facial nerve. The clinical presentation, as well as the radiographic appearance, may suggest neoplastic involvement of the facial nerve. Histologically, they may closely resemble neurilemomas (Schwannomas) or neurofibromas. Three cases of TN of the facial nerve associated with facial paralysis are presented here. Unlike previously reported cases, these tumors were not associated with chronic inflammatory middle ear disease. TN must be considered in the differential diagnosis and treatment of facial paralysis.


1986 ◽  
Vol 100 (10) ◽  
pp. 1187-1193
Author(s):  
W. H. Moesker ◽  
R. A. Tange

SummaryWe present two patients, mother and daughter, with a skeletal disorder of the skull, hearing loss and in one of them recurrent facial paralysis. In one patient middle ear exploration was performed. The malleus and incus were found to be fixed in the epitympanum.The differential diagnosis is discussed. We reviewed all known hereditary conditions with hearing loss and musculoskeletal disease but we could not accept any of these diagnoses for our patients.


1994 ◽  
pp. 367-367
Author(s):  
A. Kumar ◽  
M. Mafee ◽  
M. Dailey

2021 ◽  
Vol 38 (4) ◽  
pp. 693-695
Author(s):  
Kemal KEF

Ramsay-Hunt Syndrome (Herpes zoster oticus) is often characterized by severe ear pain, vesicles on external auditory canal or auricula and peripheral facial paralysis. However, the patient in this case presented to the clinic with dizziness, pain in the ear and vomiting. During the physical examination there was no vesicles and no evidence of skin changes found around the auricula that were typical for Ramsay Hunt syndrome. The patient did not have facial paralysis. If there is an unexpected severe ear pain accompanying vertigo, Ramsay Hunt syndrome should be considered in the differential diagnosis even if there is no rash or facial paralysis.


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