Prognostic Importance of Elevated Jugular Venous Pressure and a Third Heart Sound in Patients with Heart Failure

2001 ◽  
Vol 345 (26) ◽  
pp. 1912-1913 ◽  
e-CliniC ◽  
2016 ◽  
Vol 4 (2) ◽  
Author(s):  
Devina E. Haris ◽  
Starry H. Rampengan ◽  
Edmond L. Jim

Abstract: Acute Heart Failure is marked by a fast/rapid attack or sudden changes in symptoms or signs of Heart Failure. Heart failure has become a main issue in cardiology, because of an increasing number of Heart Failure patients and frequent re-hospitalization and death and disability. The most common cause of heart failure is coronary artery disease and hypertension. Patients with heart failure have typical symptoms, such as shortness of breath at rest or activity, easily tired, leg edema, and also tachycardia, tachypnea, pulmonary ronkhi, pleural effusion, increased jugular venous pressure, peripheral edema, hepatomegaly, and structural abnormalities or functional heart symptoms at rest, such as cardiomegaly, third heart sound, and increased levels of natriuretic peptides.Keywords: acute heart failure, inpatients care Abstrak: Gagal jantung akut adalah serangan yang cepat/rapid onset atau terjadinya perubahan mendadak dari gejala atau tanda gagal jantung. Gagal jantung telah menjadi masalah yang utama pada bidang kardiologi, karena bertambahnya jumlah penderita gagal jantung dan seringnya terjadi rawat ulang serta kematian dan kecacatan. Penyebab tersering gagal jantung di adalah penyakit arteri koroner dan hipertensi. Pasien yang mengalami gagal jantung memiliki gejala yang khas yaitu sesak napas saat istirahat atau aktifitas, mudah lelah, edema tungkai, dan terdapat juga tanda-tanda khas yaitu takikardi, takipnea, ronkhi paru, efusi pleura, peningkatan tekanan vena jugularis, edema perifer, hepatomegali, dan terdapat kelainan struktural atau fungsional jantung saat pasien istirahat yaitu kardiomegali, suara jantung ketiga, meningkatnya kadar peptida natriuretik. Kata kunci: gagal jantung akut, rawat inap


2020 ◽  
Vol 125 (10) ◽  
pp. 1524-1528
Author(s):  
Kenichi Kasai ◽  
Tatsuya Kawasaki ◽  
Shingo Hashimoto ◽  
Shiho Inami ◽  
Atsushi Shindo ◽  
...  

Circulation ◽  
2020 ◽  
Vol 142 (Suppl_3) ◽  
Author(s):  
Estefania Oliveros ◽  
Ashish A Correa ◽  
Aditya Parikh ◽  
Robert Leber ◽  
Soheila Talebi ◽  
...  

Introduction: High-output heart failure (HF) develops in the setting of excessive cardiac output. Case Presentation: 65-year-old male with HF (EF 40%), severe right ventricular dysfunction, and emphysema presented with dyspnea and anasarca. On admission, physical examination showed a BP 97/66mmHg, heart rate 109bpm, temperature 97.2F, respiratory rate of 19rpm, 93% on 2-Liters of oxygen. Jugular venous pressure was 20 cm H20 with large V waves. The rhythm was irregular, with a loud second heart sound, audible third heart sound, parasternal heave, and left lower sternal border murmur. Lung exam demonstrated basilar crackles and prolonged expiratory phase. The abdomen was distended with a pulsatile liver and the lower extremities were cool with 3+ pitting edema. There was an audible bruit with thrill at the right groin. Laboratory testing showed sodium=123mEq/L, creatinine=1.25mg/dL, bilirubin=2.2, ALT=135U/L, AST=146u/L, troponin-I=0.097ng/mL, BNP=1528pg/mL. CT and VQ scan were negative for acute/chronic pulmonary embolism. Lower extremity Doppler ultrasound revealed a right common femoral arteriovenous (AV) fistula (Fig.1). After diuretics and milrinone, a left heart catheterization demonstrated known three vessel disease, but without limitations in instant flow reserve. Right heart catheterization demonstrated RA=15mmHg, RV=50/16mmHg, PA=50/24(34)mmHg, PCWP=11mmHg, CO=5.4L/min, PVR=4.25WU, after which the fistula was ligated. On post-op day 2, repeat hemodynamics off inotropes showed an RA=4mmHg, PA=40/18mmHg, PCWP=18 mmHg, CO=4.4L/min and normalization of end-organ function. He was maximized on GDMT. Echocardiogram 5 months later showed improvement of RV function and he was able to go back to work with minimal symptoms. Conclusion: AV fistulas can lead to high-output HF if undiagnosed. A multidisciplinary approach and comprehensive hemodynamic assessment proved essential in allowing improvement of symptoms and resulting outcomes.


ESC CardioMed ◽  
2018 ◽  
pp. 1758-1762
Author(s):  
Pardeep S. Jhund

The clinical history aims to elicit the symptoms that are commonly experienced by patients with heart failure such as dyspnoea, paroxysmal nocturnal dyspnoea, orthopnoea, and fatigue. The history may also provide clues as to the aetiology of heart failure and symptoms that suggest alternative diagnoses. Similarly, signs that are elicited on clinical examination are used to support the diagnosis of heart failure. In addition, they can be used to determine prognosis and assess response to treatment or the need for more intensive treatment. Common signs such as peripheral oedema, jugular venous distension, and pulmonary crackles are less specific for the diagnosis of heart failure than others such as a third heart sound. The presence of some clinical signs may help determine the aetiology of heart failure and indicate other potential diagnoses that may present like heart failure but require very different treatment. While signs and symptoms are used in conjunction with imaging evidence and raised natriuretic peptides to make the diagnosis of heart failure, the electrocardiogram (ECG) still plays a central role. The ECG can be used to determine underlying aetiology (such as evidence of prior myocardial infarction) and guide therapeutic decision-making such as the need for cardiac resynchronization therapy in those with bundle branch block. Information on symptoms, signs, and investigations such as the ECG need to be integrated to ensure the accurate diagnosis and optimal treatment of patients with heart failure.


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