Analysis of antibody response to an epitope in the haemagglutinin subunit 2 of avian influenza virus H5N1 for differentiation of infected and vaccinated chickens

2019 ◽  
Vol 49 (2) ◽  
pp. 161-170
Author(s):  
Khrisdiana Putri ◽  
Michael Haryadi Wibowo ◽  
Simson Tarigan ◽  
Nadeeka Wawegama ◽  
Jagoda Ignjatovic ◽  
...  
2007 ◽  
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pp. 10 ◽  
Author(s):  
Navin Horthongkham ◽  
Tananun Srihtrakul ◽  
Niracha Athipanyasilp ◽  
Sontana Siritantikorn ◽  
Wannee Kantakamalakul ◽  
...  

2008 ◽  
Vol 14 (2) ◽  
pp. 308-310 ◽  
Author(s):  
Matthias Giese ◽  
Timm C. Harder ◽  
Jens P. Teifke ◽  
Robert Klopfleisch ◽  
Angele Breithaupt ◽  
...  

2018 ◽  
Vol 48 (1) ◽  
pp. 73-79 ◽  
Author(s):  
Mohammed Nooruzzaman ◽  
Md. Enamul Haque ◽  
Emdadul Haque Chowdhury ◽  
Mohammad Rafiqul Islam

2011 ◽  
Vol 64 (2) ◽  
pp. 205-211 ◽  
Author(s):  
Alexander C.K. Lai ◽  
Wai-lan Wu ◽  
Siu-ying Lau ◽  
Yi Guan ◽  
Honglin Chen

2011 ◽  
Vol 2011 ◽  
pp. 1-10 ◽  
Author(s):  
Yi Jin ◽  
Yanxin Hu ◽  
Deping Han ◽  
Ming Wang

Chronic heat stress (CHS) can negatively affect immune response in animals. In this study we assessed the effects of CHS on host innate immunity and avian influenza virus H5N1 infection in mice. Mice were divided into two groups: CHS and thermally neutral (TN). The CHS treatment group exhibited reduced local immunity in the respiratory tract, including the number of pulmonary alveolar macrophages and lesions in the nasal mucosa, trachea, and lungs. Meanwhile, CHS retarded dendritic cells (DCs) maturation and reduced the mRNA levels of IL-6 and IFN-βsignificantly (P<.05). After the CHS treatment, mice were infected with H5N1 virus. The mortality rate and viral load in the lungs of CHS group were higher than those of TN group. The results suggest that the CHS treatment could suppress local immunity in the respiratory tract and innate host immunity in mice significantly and moderately increased the virulence in H5N1-infected mice.


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