Effect of Corynebacterium parvum , Methanol-Extraction Residue of BCG, and Levamisole on Macrophage Random Migration, Chemotaxis, and Pinocytosis

1977 ◽  
Vol 58 (6) ◽  
pp. 1753-1757 ◽  
Author(s):  
Neal A. Sher ◽  
David G. Poplack ◽  
R. Michael Blaese ◽  
Teresa M. Brown ◽  
Sotiros D. Chaparas
Keyword(s):  
Corynebacterium Parvum ◽  
Random Migration ◽  
Methanol Extraction

Effect of Immunotherapy With Corynebacterium Parvum and Methanol Extraction Residue of BCG Administered Intravenously on Host Defense Function in Cancer Patients2, 3

1981 ◽  
Vol 66 (6) ◽  
pp. 993-1002 ◽  
Author(s):  
Evan M. Hersh ◽  
Samuel G. Murphy ◽  
Jorge R. Quesada ◽  
Jordan U. Gutterman ◽  
Charles R. Gachwind ◽  
...  
Keyword(s):  
Host Defense ◽  
Corynebacterium Parvum ◽  
Methanol Extraction ◽  
Defense Function ◽  
Host Defense Function

Epithelial dendritic cells and connective tissue macrophages in oral corcinogenesis and the effects of systemic Corynebacterium parvum

1989 ◽  
Vol 10 (3) ◽  
pp. 501-507 ◽  
Author(s):  
A. Pitigala-Arachchi ◽  
J.B. Matthews ◽  
C. Scully ◽  
S.S. Prime
Keyword(s):  
Dendritic Cells ◽  
Connective Tissue ◽  
Corynebacterium Parvum

The importance of parasite load in the killing ofPlasmodium vinckeiin mice treated withCorynebacterium parvumor alloxan monohydrate

Parasitology ◽  
1984 ◽  
Vol 89 (3) ◽  
pp. 417-424 ◽  
Author(s):  
F. E. G. Cox ◽  
Stephanie M. Millott
Keyword(s):  
Red Blood Cells ◽  
Blood Cells ◽  
Parasite Load ◽  
Corynebacterium Parvum ◽  
Specific Immunity ◽  
Pre Treatment ◽  
Alloxan Monohydrate ◽  
High Parasitaemias ◽  
Plasmodium Vinckei

SUMMARYMice pre-treated withCorynebacterium parvumand later challenged withPlasmodium vinckeibecome infected but do not die whereas control mice do. When pre-treated mice were challenged with 1, 10, 1 × 102, 1 × 104, 1 × 105or 1 × 106parasites, the pre-patent periods correlated directly with the number of parasites injected, but the subsequent parasitaemias reached similar levels. This suggests that parasite killing, resulting from pre-treatment withC. parvum, is not triggered until the parasite load has reached a particular threshold. The injection of alloxan monohydrate, which brings about the release of toxic oxygen inter mediates thought to be involved in non-specific immunity, has little effect onP. vinckeiinfections until the parasitaemia is relatively high. This indicates that oxygen-mediated parasite killing also does not occur until the parasitaemia has reached a particular threshold. It is suggested that it is only at relatively high parasitaemias that the factors involved in parasite killing are able to enter the infected red blood cells.

scholarly journals AN ANALYSIS OF THE INHIBITORY EFFECT OF CORYNEBACTERIUM PARVUM ON GRAFT-VERSUS-HOST DISEASE

1967 ◽  
Vol 5 (6) ◽  
pp. 1510-1524 ◽  
Author(s):  
J. G. HOWARD ◽  
G. BIOZZI ◽  
C. STIFFEL ◽  
D. MOUTON ◽  
P. LIACOPOULOS
Keyword(s):  
Graft Versus Host Disease ◽  
Inhibitory Effect ◽  
Corynebacterium Parvum ◽  
Host Disease ◽  
Graft Versus Host

Subcutaneous Corynebacterium parvum in Bladder Cancer

1979 ◽  
Vol 51 (4) ◽  
pp. 278-282 ◽  
Author(s):  
ELIZABETH C. PURVES ◽  
M. SNELL ◽  
W. A. COPE ◽  
I. E. ADDISON ◽  
R.F.P. COPLAND ◽  
...  
Keyword(s):  
Bladder Cancer ◽  
Corynebacterium Parvum

TOXICITY OF INTRAVENOUS CORYNEBACTERIUM PARVUM

The Lancet ◽  
1975 ◽  
Vol 306 (7944) ◽  
pp. 1090-1091 ◽  
Author(s):  
R OSSORIO
Keyword(s):  
Corynebacterium Parvum
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