scholarly journals Lung fluid balance in patients with heart failure: effect of beta‐2 adrenergic‐receptor stimulation

2011 ◽  
Vol 25 (S1) ◽  
Author(s):  
Bryan Taylor ◽  
Maile Ceridon ◽  
Eric Snyder ◽  
Alex Carlson ◽  
Minelle Hulsebus ◽  
...  
Keyword(s):  
Heart Failure ◽  
Fluid Balance ◽  
Adrenergic Receptor ◽  
Receptor Stimulation ◽  
Lung Fluid ◽  
Lung Fluid Balance ◽  
Patients With Heart Failure ◽  
Beta 2

scholarly journals The effect of β 2 adrenergic‐receptor stimulation on lung fluid balance in heart failure

2012 ◽  
Vol 26 (S1) ◽  
Author(s):  
Bryan Joseph Taylor ◽  
Maile Ceridon ◽  
Eric Snyder ◽  
Thomas Olson ◽  
Bruce David Johnson
Keyword(s):  
Heart Failure ◽  
Fluid Balance ◽  
Adrenergic Receptor ◽  
Receptor Stimulation ◽  
Lung Fluid ◽  
Lung Fluid Balance

scholarly journals Hypoxia induced changes in lung fluid balance in humans is associated with beta-2 adrenergic receptor density on lymphocytes

2012 ◽  
Vol 183 (2) ◽  
pp. 159-165 ◽  
Author(s):  
Micah W. Johnson ◽  
Bryan J. Taylor ◽  
Minelle L. Hulsebus ◽  
Bruce D. Johnson ◽  
Eric M. Snyder
Keyword(s):  
Fluid Balance ◽  
Adrenergic Receptor ◽  
Receptor Density ◽  
Lung Fluid ◽  
Lung Fluid Balance ◽  
Beta 2 ◽  
Induced Changes

scholarly journals Beta-2 adrenergic receptor gene polymorphisms Gln27Glu, Arg16Gly in patients with heart failure

2009 ◽  
Vol 9 (1) ◽  
Author(s):  
Alfredo José Mansur ◽  
Rosana Seleri Fontes ◽  
Regina Airoldi Canzi ◽  
Raphael Nishimura ◽  
Airlane Pereira Alencar ◽  
...  
Keyword(s):  
Heart Failure ◽  
Adrenergic Receptor ◽  
Gene Polymorphisms ◽  
Receptor Gene ◽  
Patients With Heart Failure ◽  
Beta 2 ◽  
Receptor Gene Polymorphisms ◽  
Adrenergic Receptor Gene

scholarly journals Effect of β 2 -adrenergic receptor stimulation on lung fluid in stable heart failure patients

2017 ◽  
Vol 36 (4) ◽  
pp. 418-426 ◽  
Author(s):  
Bryan J. Taylor ◽  
Eric M. Snyder ◽  
Maile L. Richert ◽  
Courtney M. Wheatley ◽  
Steven C. Chase ◽  
...  
Keyword(s):  
Heart Failure ◽  
Adrenergic Receptor ◽  
Receptor Stimulation ◽  
Heart Failure Patients ◽  
Lung Fluid

Sodium transport and fluid balance in lungs from normal and dystrophic hamsters

1994 ◽  
Vol 77 (4) ◽  
pp. 1750-1754 ◽  
Author(s):  
W. F. Waltz ◽  
J. A. Burbach ◽  
E. H. Schlenker ◽  
B. E. Goodman
Keyword(s):  
Fluid Balance ◽  
Sodium Transport ◽  
Fluorescein Isothiocyanate ◽  
Dry Weight ◽  
Apparent Permeability ◽  
Lung Fluid ◽  
Lung Fluid Balance ◽  
Permeability Surface ◽  
Lower Permeability ◽  
Sodium Clearance

Gravimetric and sodium transport characteristics of lungs from BIO 14.6 (dystrophic) hamsters were compared with those of lungs from golden Syrian (normal) hamsters at 30 and 150 days of age. Isolated perfused lungs were used to determine lung permeability and fluid balance differences between normal and dystrophic animals at both ages. Apparent permeability-surface area products for air space-to-vascular space sodium, sucrose, and fluorescein isothiocyanate-labeled dextran fluxes were compared in the four groups of hamsters. Morphometric analysis of fixed lungs of representative hamsters from each group was also performed. Dystrophic hamsters exhibited higher lung wet-to-dry weight ratios than normal hamsters at both ages. Lungs from dystrophic hamsters were less sensitive to inhibition of sodium transport by amiloride than lungs from age-matched normal hamsters. Dystrophic hamster lungs had higher absolute permeabilities of the passively transported solutes, lower permeability values for sodium, and only one-half of the amiloride-sensitive sodium transport of lungs from age-matched normal hamsters. Differences in lung fluid balance between dystrophic and normal hamsters may be related to differences in sodium clearance.

Inspiratory airway obstruction does not affect lung fluid balance in lambs

1985 ◽  
Vol 58 (4) ◽  
pp. 1314-1318 ◽  
Author(s):  
T. N. Hansen ◽  
A. L. Gest ◽  
S. Landers
Keyword(s):  
Airway Obstruction ◽  
Endotracheal Tube ◽  
Fluid Balance ◽  
Left Atrial ◽  
Lymph Flow ◽  
Lung Fluid ◽  
Lung Fluid Balance ◽  
Pulmonary Arterial ◽  
Lung Lymph ◽  
Lung Lymph Flow

The purpose of this study was to examine the effects of inspiratory airway obstruction on lung fluid balance in newborn lambs. We studied seven 2- to 4-wk-old lambs that were sedated with chloral hydrate and allowed to breathe 30–40% O2 spontaneously through an endotracheal tube. We measured lung lymph flow, lymph and plasma protein concentrations, pulmonary arterial and left atrial pressures, mean and phasic pleural pressures and airway pressures, and cardiac output during a 2-h base-line period and then during a 2- to 3-h period of inspiratory airway obstruction produced by partially occluding the inspiratory limb of a nonrebreathing valve attached to the endotracheal tube. During inspiratory airway obstruction, both pleural and airway pressures decreased 5 Torr, whereas pulmonary arterial and left atrial pressures each decreased 4 Torr. As a result, calculated filtration pressure remained unchanged. Inspiratory airway obstruction had no effect on steady-state lung lymph flow or the lymph protein concentration relative to that of plasma. We conclude that in the spontaneously breathing lamb, any decrease in interstitial pressure resulting from inspiratory airway obstruction is offset by a decrease in microvascular hydrostatic pressure so that net fluid filtration remains unchanged.

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