Sudden decrease in spectral entropy associated with severe carotid artery spasm

2009 ◽  
Vol 26 (1) ◽  
pp. 84-86 ◽  
Author(s):  
Sriganesh Kamath ◽  
Puthuvassery Raman Suneel ◽  
Subrata Kumar Singha
Keyword(s):  
Carotid Artery ◽  
Spectral Entropy ◽  
Sudden Decrease

scholarly journals Postoperative stroke in head and neck cancer patients submitted to surgery

2018 ◽  
Vol 4 (4) ◽  
pp. 886
Author(s):  
Diana P. Silva ◽  
Joaquim C. Silva ◽  
Eurico Monteiro
Keyword(s):  
Carotid Artery ◽  
Head And Neck ◽  
Carotid Artery Stenosis ◽  
Neck Surgery ◽  
Primary Treatment ◽  
Artery Stenosis ◽  
Artery Ligation ◽  
Neurologic Recovery ◽  
Postoperative Stroke ◽  
Sudden Decrease

<p class="abstract"><strong>Background:</strong> Stroke is a highly morbid complication after head and neck surgery (HNS). Our purpose was to report the casuistic of postoperative stroke (POS) in ENT Department of Oporto Cancer Institute, discuss predictable causes and prognosis.</p><p class="abstract"><strong>Methods:</strong> Retrospective study that included cases of POS in our Department, between 2012 and 2017. Medical files were reviewed and perioperative aspects were analysed.  </p><p class="abstract"><strong>Results:</strong> We identified 8 cases of POS in 293 HNS performed. All underwent to HNS including bilateral ND as primary treatment. Several cardiovascular risk factors (CVRF) were present, especially carotid artery stenosis (CAS). Complete internal carotid artery (ICA) encasement was present in 2 patients, in whom artery ligation was performed. Acute drop of bispectral index (BIS) occurred in 1 patient. Stroke occurred at 2.8 post-operatory day (in mean) and was ischemic in 7 patients and haemorrhagic in 1. The most affected vessel was cerebral media artery (87.5%). Hemiparesis was the main clinical manifestation (63%). Incidence of POS was 2.7%. Functional and neurologic recovery occurred in 50%. Mortality rate was 37.5%.</p><p><strong>Conclusions:</strong> Our findings suggest that worst outcomes were observed in patients with advanced tumour stages, vessel tumour involvement or carotid artery stenosis presence with requirement ligation of ICA, higher number of CVRF, and low or sudden decrease in BIS values during surgery. According to our results we recommend to screen, select and optimize patients to minimise the incidence and severity of this complication.</p>

Endotoxin Alteration of the Mucopolysaccharide Blood Vessel Coating in Rats

1974 ◽  
Vol 32 ◽  
pp. 148-149
Author(s):  
D. E. Philpott ◽  
A. Takahashi
Keyword(s):  
Skeletal Muscle ◽  
Endothelial Cells ◽  
Salivary Gland ◽  
Carotid Artery ◽  
Basement Membrane ◽  
Coronary Vessels ◽  
Ruthenium Red ◽  
E Coli ◽  
Old Rats ◽  
The Brain

Two month, eight month and two year old rats were treated with 10 or 20 mg/kg of E. Coli endotoxin I. P. The eight month old rats proved most resistant to the endotoxin. During fixation the aorta, carotid artery, basil arartery of the brain, coronary vessels of the heart, inner surfaces of the heart chambers, heart and skeletal muscle, lung, liver, kidney, spleen, brain, retina, trachae, intestine, salivary gland, adrenal gland and gingiva were treated with ruthenium red or alcian blue to preserve the mucopolysaccharide (MPS) coating. Five, 8 and 24 hrs of endotoxin treatment produced increasingly marked capillary damage, disappearance of the MPS coating, edema, destruction of endothelial cells and damage to the basement membrane in the liver, kidney and lung.

Oxidative injury associated with stenting of asymptomatic carotid artery stenosis

VASA ◽  
2017 ◽  
Vol 46 (4) ◽  
pp. 268-274
Author(s):  
Erhan Saraçoğlu ◽  
Ertan Vuruşkan ◽  
Yusuf Çekici ◽  
Salih Kiliç ◽  
Halil Ay ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Carotid Artery ◽  
Oxidative Damage ◽  
Carotid Artery Stenosis ◽  
Artery Stenosis ◽  
Oxidative Stress Marker ◽  
Asymptomatic Carotid ◽  
Imaging Methods ◽  
Neurological Findings ◽  
Asymptomatic Carotid Artery Stenosis

Abstract. Background: After carotid artery stenting (CAS), neurological complications that cannot be explained with imaging methods may develop. In our study we aimed to show, using oxidative stress markers, isolated oxidative damage and resulting neurological findings following CAS in patients with asymptomatic carotid artery stenosis. Patients and methods: We included 131 neurologically asymptomatic patients requiring CAS. The neurological findings were evaluated using the modified Rankin Scale (mRS) prior to the procedure, one hour post-procedure, and two days after. Patients with elevated mRS scores but with or without typical hyperintense lesions observed on an MRI and with changes of oxidative stress marker levels at the time (Δtotal-thiol, Δtotal antioxidative status [TAS], and Δtotal oxidant status [TOS]) were evaluated. Results: In the neurological examination carried out one hour prior to the procedure, there were 92 patients with mRS = 0, 20 with mRS = 1, and 12 with mRS = 2. When Δtotal-thiol, ΔTAS, and ΔTOS values and the mRS were compared, it was observed that as the difference in oxidative parameters increased, clinical deterioration also increased proportionally (p = 0.001). Conclusions: We demonstrate a possible correlation between oxidative damage and neurological findings after CAS which could not be explained by routine imaging methods.

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