EXTERNAL SUBRETINAL DRAINAGE, BEVACIZUMAB, AND SCLERAL BUCKLING FOR COMPLETE EXUDATIVE RETINAL DETACHMENT AFTER PHOTOCOAGULATION IN RETINOPATHY OF PREMATURITY

2014 ◽  
Vol 8 (1) ◽  
pp. 33-36 ◽  
Author(s):  
Felix Armada-Maresca ◽  
Jesus Peralta-Calvo ◽  
Natalia Pastora-Salvador ◽  
Anna Grabowska ◽  
Jose Vallejo-Garcia
2018 ◽  
Vol 53 (2) ◽  
pp. e74-e77 ◽  
Author(s):  
Parveen Sen ◽  
Hatim Yusufali ◽  
Vikas Khetan ◽  
Chetan Rao ◽  
Aniruddha Banerjee

PEDIATRICS ◽  
1982 ◽  
Vol 69 (3) ◽  
pp. 380-381
Author(s):  
William A. Silverman

In 1980, a review appeared in the ophthalmology literature1 that summarized attempts at active treatment of the neovascular changes in retinopathy of prematurity (ROP) (referred to in the past as "proliferative retrolental fibroplasia [RLF]") and of retinal detachment which may occur in the disorder. The author of the survey observed (among a number of treatments tried) that the effectiveness of transscleral cryotherapy for treatment of proliferative changes was difficult to evaluate and he pointed out that "no prospective clinical trial has demonstrated the value [of this approach]." In respect to scleral buckling, as treatment for detachments in ROP, he remarked that although the approach seemed to be rational, the "ultimate fate of [treated] eyes remains uncertain."


1990 ◽  
Vol 201 (2) ◽  
pp. 79-82 ◽  
Author(s):  
Toshihiko Matsuo ◽  
Koji Eguchi ◽  
Nobuhiko Matsuo

2021 ◽  
Vol 11 (1) ◽  
pp. 16-21
Author(s):  
Rubina Shah

Coats disease is the potential leading cause of blindness which is an idiopathic disorder characterized by progressive deposition of intraretinal or subretinal exudates and abnormal development of retinal vasculatures known as telangiectasia that leads to exudative retinal detachment. The case shows unilateral involvement of the eye and primarily at an early age of 12 years. The cause of the disease is idiopathic and if associated with other genetic disorders emphasizes the deposition of a genetic component. The underlying histopathological problem is thought due to the abnormal permeability of capillary endothelial cells of the retina. The complications of long-standing disease may progress to total retinal detachment, leukocoria, painful Glaucoma and can be hard to differentiate from retinoblastoma. The diagnostic tools are indirect ophthalmoscopy, fluorescein angiography, CT scan, and MRI. The treatment depends on the closure of abnormal leaking vessels of the retina which is laser therapy and anti-VEGF at its early stage. The advanced stage requires surgical therapy such as scleral buckling, pars plana vitrectomy, and anterior vitrectomy for reattachment of the retina or pre retinal membrane. A careful selection therapy showed a 70% improvement in the clinical course of the disease


2020 ◽  
pp. 112067212094401
Author(s):  
Francesco Barca ◽  
Giulio Vicini ◽  
Cristina Nicolosi ◽  
Giulia Pieretti ◽  
Tomaso Caporossi ◽  
...  

Introduction: We report about a large retinal capillary hemangioma (RCH) with exudative retinal detachment and a macular fold, treated with Ruthenium-106 brachytherapy (Ru-106 BT) and scleral buckling surgery, followed by pars plana vitrectomy (PPV), for the removal of macular tractions. Case description: A 17-year-old boy was referred to our Ocular Oncology Unit for a large RCH in the left eye. BCVA was hand motion. The RCH measured 4.9 × 6.85 mm in basal diameters and 4.0 mm in thickness and was located in the mid-peripheral temporal retina. It was surrounded by extensive subretinal exudation, forming an exudative retinal detachment, with a retinal fold that extended from the lesion to the optic disc. We performed Ru-106 BT and at the moment of the plaque removal we placed a radial buckle with the aim to unbend the retinal fold. At 3-months follow-up the exudation decreased, we achieved the opening of the peripheral side of the retinal fold, but the macula was still detached. We decided to perform a lens sparing PPV, macular peeling and air tamponade, to remove the vitreoretinal tractions ab interno and to try to complete the opening of the macular fold. After 1-month BCVA was counting fingers, the retina appeared attached, also in the macular area, but the retinal fold remained partially close in the macular side. After 6 months the tumor was inactivated, the macula remained attached, unfortunately, the macular fold remained partially close. Conclusion: Ru-106 BT and scleral buckling concurrent approach can be an effective treatment modality in selected cases of large RCHs, followed by PPV to remove eventual vitreo-retinal tractions.


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