Myocardial ischemic injury derived from multiple thromboemboli due to eosinophilic endomyocarditis (Löffler endocarditis) causing right ventricular rupture

2021 ◽  
Author(s):  
Hidenori Yoshizawa ◽  
Yoshihiko Ikeda ◽  
Kinta Hatakeyama ◽  
Ken‐ichi Yoshida
Keyword(s):  
Right Ventricular ◽  
Ischemic Injury ◽  
Ventricular Rupture ◽  
Myocardial Ischemic Injury

Precordial S-T segment elevation mapping: An atraumatic method for assessing alterations in the extent of myocardial ischemic injury

1972 ◽  
Vol 29 (2) ◽  
pp. 223-230 ◽  
Author(s):  
Peter R. Maroko ◽  
Peter Libby ◽  
James W. Covell ◽  
Burton E. Sobel ◽  
John Ross ◽  
...  
Keyword(s):  
Ischemic Injury ◽  
Myocardial Ischemic Injury

Repair of right ventricular rupture complicating mediastinitis

1999 ◽  
Vol 68 (5) ◽  
pp. 1892-1893 ◽  
Author(s):  
Tamás Szerafin ◽  
Osama Jaber ◽  
Árpád Péterffy
Keyword(s):  
Right Ventricular ◽  
Ventricular Rupture

A Novel nonpeptidic caspase-3/7 inhibitor, (S)-(+)-5-[1-(2-methoxymethylpyrrolidinyl)sulfonyl]isatin reduces myocardial ischemic injury

2002 ◽  
Vol 456 (1-3) ◽  
pp. 59-68 ◽  
Author(s):  
Justin G Chapman ◽  
William P Magee ◽  
Hans A Stukenbrok ◽  
Gretchen E Beckius ◽  
Anthony J Milici ◽  
...  
Keyword(s):  
Caspase 3 ◽  
Ischemic Injury ◽  
Myocardial Ischemic Injury

Right Ventricular Rupture After Extubation of a Patient With an Open Chest

2018 ◽  
Vol 32 (1) ◽  
pp. 461-463
Author(s):  
Robert Doody ◽  
Brett Cronin ◽  
E. Orestes O’Brien
Keyword(s):  
Right Ventricular ◽  
Open Chest ◽  
Ventricular Rupture

Osmotic shock: modulation of contractile function, pHi, and ischemic damage in perfused guinea pig heart

1999 ◽  
Vol 276 (4) ◽  
pp. H1236-H1244 ◽  
Author(s):  
Douglas E. Befroy ◽  
Trevor Powell ◽  
George K. Radda ◽  
Kieran Clarke
Keyword(s):  
Nmr Spectroscopy ◽  
Guinea Pig ◽  
Intracellular Ph ◽  
Contractile Function ◽  
Osmotic Shock ◽  
Ischemic Injury ◽  
Ischemic Damage ◽  
Guinea Pig Heart ◽  
Myocardial Ischemic Injury ◽  
Developed Pressure

To determine the contribution of changes in extracellular osmolarity to ischemic injury, isolated guinea pig hearts were perfused with hyposmotic (220 mosM) or hyperosmotic (380 mosM) buffer.31P NMR spectroscopy was used to follow changes in intracellular pH (pHi) and energetics. Hyposmotic buffer decreased myocardial developed pressure by 30 ± 2% and pHi by 0.02 ± 0.01 unit, whereas hyperosmotic buffer increased myocardial developed pressure by 34 ± 1% and pHi by 0.14 ± 0.01 unit. All hearts recovered to control values on restoration of isosmotic (300 mosM) buffer. The hyperosmolar-induced intracellular alkalosis and developed pressure increase were not prevented by inhibition of Na+/H+exchange with use of 1 μM HOE-642 but were abolished with use of bicarbonate-free buffers. After 20 min of total global ischemia, hearts perfused with hyposmotic buffer showed significantly greater recoveries of developed pressure, phosphocreatine, and ATP than control hearts, but hearts perfused with hyperosmotic buffer did not recover after ischemia. In conclusion, buffer osmolarities between 220 and 380 mosM alter myocardial pHi and developed pressure but are not deleterious during perfusion. However, buffer osmolarity significantly alters the extent of myocardial ischemic injury.

scholarly journals Right Ventricular Rupture Caused by Prolonged Cardiopulmonary Resuscitation after Sudden Cardiac Arrest

2015 ◽  
Vol 88 (4) ◽  
pp. 434
Author(s):  
Hee Jeong Lee ◽  
Seong Soon Kwon ◽  
Hye Ran Kang ◽  
Duk Won Bang ◽  
Byoung Won Park ◽  
...  
Keyword(s):  
Cardiac Arrest ◽  
Cardiopulmonary Resuscitation ◽  
Right Ventricular ◽  
Sudden Cardiac Arrest ◽  
Ventricular Rupture
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