Om tuis te gaan in styl: Territoriumskepping in Breyten Breytenbach se ’n Seisoen in die paradys (1976) en Dog Heart (1998)

This article is a study of how Breyten Breytenbach deals with the idea of home in his autobiographical prose, how he experiences home and how he constructs it. Although I hope that the argument will serve to give a new perspective on any of Breytenbach’s texts that deal with questions about the nature of home and the construction thereof, I only refer to ’n Seisoen in Paradys (A Season in Paradise, 1976) and Dog Heart (1998). The article uses concepts of Deleuze and Guattari to show how Breytenbach writes his home, and especially how his constructions of a home in his self-writings are products of a certain style of writing.

9. PREVALENSI DIROFILARIASIS PADA ANJING LOKAL (Canis domestica) DI KECAMATAN LHOKNGA ACEH BESAR SECARA PATOLOGI ANATOMIS Prevalence of Dirofilariasis on Local Dogs (Canis domestica) in Lhoknga Aceh Besar Using Phatological Anatomy Method

The aim of this research was to find out the prevalence of Dirofilaria immitis on local dog in Lhoknga Aceh Besar. This study used 30 local dogs collected from 5 villages in Lhoknga Aceh Besar. Dogs were injected intravenous with phenobarbital at dose of 2-3 mg/kg body weight, then necropsied to find out Dirofilaria immitis in dog heart. The result showed that 3 out of 30 dogs were positive dirofilariasis, two dogs from Nusa village and one dog from Lampaya village. In conclusion, the prevalence of dirofilariasis on local dogs in Lhoknga Aceh Besar is 10%. ____________________________________________________________________________________________________________________ Key words: prevalence, Dirofilaria immitis, dogs, necropsy

The Formation of Aberrant Collateral Vessels during Coronary Arteriogenesis in Dog Heart

We previously reported excessive growth of collateral vessels in the dog heart during arteriogenesis induced by implantation of an ameroid constrictor around the circumflex branch of the left coronary artery. In the present study, using histology and immunocofocal microscopy, we further investigated how these aberrant collateral vessels form. By comparison with mature collateral vessels the following findings were made: perivascular space was very narrow where damage of the perivascular myocardium occurred; the neointima was very thick, resulting in a very small lumen; elastica van Gieson staining revealed the absence of the internal elastic lamina and of elastic fibers in the adventitia, but abundant collagen in the adventitia as well as in the neointima; smooth muscle cells of the neointima expressed less α-SM actin and little desmin; expression of the fibroblast growth factors aFGF, bFGF and platelet-derived growth factor (PDGF)-AB was observed mainly in the endothelial cells and abluminal region, but transforming growth factor-β1 was only present in the adventitia and damaged myocardium; angiogenesis in the neointima was observed in some collateral vessels expressing high levels of eNOS, and cell proliferation was mainly present in the abluminal region, but apoptosis was in the deep neointima. In conclusion, these data for the first time reveal that the formation of the aberrant collateral vessels in the dog heart involves active extracellular proteolysis and a special expression profile of growth factors, eNOS, cell proliferation and apoptosis. The finding of a narrow perivascular space and perivascular myocardial damage suggests that anatomical constraint is most likely the cause for exacerbated inward remodeling in aberrant collateral vessels in dog heart.