scholarly journals Apoptosis is a major mode of cell death caused by ischaemia and ischaemia/reperfusion injury to the rat intestinal epithelium

Gut ◽  
1998 ◽  
Vol 42 (4) ◽  
pp. 530-537 ◽  
Author(s):  
H Ikeda ◽  
Y Suzuki ◽  
M Suzuki ◽  
M Koike ◽  
J Tamura ◽  
...  
Keyword(s):  
Cell Death ◽  
Reperfusion Injury ◽  
Intestinal Epithelium ◽  
Ischaemia Reperfusion Injury ◽  
Ischaemia Reperfusion ◽  
Major Mode

Urocortin inhibits Beclin1-mediated autophagic cell death in cardiac myocytes exposed to ischaemia/reperfusion injury

2006 ◽  
Vol 40 (6) ◽  
pp. 846-852 ◽  
Author(s):  
Lauren Valentim ◽  
Kevin M. Laurence ◽  
Paul A. Townsend ◽  
Christopher J. Carroll ◽  
Surinder Soond ◽  
...  
Keyword(s):  
Cell Death ◽  
Reperfusion Injury ◽  
Cardiac Myocytes ◽  
Autophagic Cell Death ◽  
Ischaemia Reperfusion Injury ◽  
Ischaemia Reperfusion

Polydatin post-treatment alleviates myocardial ischaemia/reperfusion injury by promoting autophagic flux

2016 ◽  
Vol 130 (18) ◽  
pp. 1641-1653 ◽  
Author(s):  
Yuanna Ling ◽  
Guiming Chen ◽  
Yi Deng ◽  
Huixiong Tang ◽  
Long Ling ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Cell Death ◽  
Reperfusion Injury ◽  
Myocardial Ischaemia ◽  
Reactive Oxygen ◽  
Post Treatment ◽  
Autophagic Flux ◽  
Oxygen Species ◽  
Ischaemia Reperfusion Injury ◽  
Ischaemia Reperfusion

The present paper provides evidence that polydatin (PD) post-treatment alleviates myocardial ischaemia/reperfusion (I/R) injury by promoting autophagic flux to clear damaged mitochondria to reduce reactive oxygen species (ROS) and cell death.

Why is it worth testing the ability of zinc to protect against ischaemia reperfusion injury for human application

Metallomics ◽  
2019 ◽  
Vol 11 (8) ◽  
pp. 1330-1343 ◽  
Author(s):  
Joseph Ischia ◽  
Damien M. Bolton ◽  
Oneel Patel
Keyword(s):  
Cell Death ◽  
Reperfusion Injury ◽  
Organ Dysfunction ◽  
Oxygen Supply ◽  
Tissue Injury ◽  
Blood Oxygen ◽  
Ischaemia Reperfusion Injury ◽  
Ischaemia Reperfusion

Ischaemia (interruption in the blood/oxygen supply) and subsequent damage induced by reperfusion (restoration of blood/oxygen supply) ultimately leads to cell death, tissue injury and permanent organ dysfunction.

scholarly journals Activation of volume-sensitive Cl− channel mediates autophagy-related cell death in myocardial ischaemia/reperfusion injury

Oncotarget ◽  
2016 ◽  
Vol 7 (26) ◽  
pp. 39345-39362 ◽  
Author(s):  
Yuesheng Xia ◽  
Yan Liu ◽  
Tong Xia ◽  
Xing Li ◽  
Cong Huo ◽  
...  
Keyword(s):  
Cell Death ◽  
Reperfusion Injury ◽  
Myocardial Ischaemia ◽  
Ischaemia Reperfusion Injury ◽  
Cl Channel ◽  
Ischaemia Reperfusion ◽  
Related Cell

Differential bradykinin B1 and B2 receptor regulation in cell death induced by hepatic ischaemia/reperfusion injury

2014 ◽  
Vol 127 (6) ◽  
pp. 405-413 ◽  
Author(s):  
Mayra A. Paio ◽  
Maria Kouyoumdjian ◽  
Durval R. Borges ◽  
Marcia R. Nagaoka
Keyword(s):  
Cell Death ◽  
Reperfusion Injury ◽  
Apoptotic Cell ◽  
Receptor Regulation ◽  
Necrotic Cell Death ◽  
Necrotic Cell ◽  
Ischaemia Reperfusion Injury ◽  
B1 Receptor ◽  
Ischaemia Reperfusion ◽  
Kinin B1 Receptor

In the present study, we have demonstrated that the kinin B1 receptor may participate in apoptotic cell death signalling, whereas the B2 receptor may be involved in necrotic cell death during IRI.

scholarly journals Effect of intracellular lipid droplets on cytosolic Ca2+and cell death during ischaemia-reperfusion injury in cardiomyocytes

2009 ◽  
Vol 587 (6) ◽  
pp. 1331-1341 ◽  
Author(s):  
Ignasi Barba ◽  
Laia Chavarria ◽  
Marisol Ruiz-Meana ◽  
Maribel Mirabet ◽  
Esperanza Agulló ◽  
...  
Keyword(s):  
Cell Death ◽  
Reperfusion Injury ◽  
Lipid Droplets ◽  
Intracellular Lipid ◽  
Ischaemia Reperfusion Injury ◽  
Ischaemia Reperfusion
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