scholarly journals Radiofrequency ablation therapy in idiopathic left ventricular tachycardia with no obvious structural heart disease.

Circulation ◽  
1994 ◽  
Vol 89 (4) ◽  
pp. 1690-1696 ◽  
Author(s):  
M S Wen ◽  
S J Yeh ◽  
C C Wang ◽  
F C Lin ◽  
I C Chen ◽  
...  
Keyword(s):  
Heart Disease ◽  
Ventricular Tachycardia ◽  
Radiofrequency Ablation ◽  
Structural Heart Disease ◽  
Left Ventricular ◽  
Ablation Therapy ◽  
Idiopathic Left Ventricular Tachycardia ◽  
Radiofrequency Ablation Therapy

scholarly journals Echocardiographic Predictors of Ventricular Tachycardia

2014 ◽  
Vol 8s4 ◽  
pp. CMC.S18499 ◽  
Author(s):  
John N. Catanzaro ◽  
John N. Makaryus ◽  
Amgad N. Makaryus ◽  
Cristina Sison ◽  
Christos Vavasis ◽  
...  
Keyword(s):  
Heart Disease ◽  
Ventricular Tachycardia ◽  
Left Atrial ◽  
Structural Heart Disease ◽  
Left Ventricular ◽  
Pharmacologic Therapy ◽  
Cardioverter Defibrillator ◽  
First Occurrence ◽  
Echocardiographic Parameters

Background Patients with structural heart disease are prone to ventricular tachycardia (VT) and ventricular fibrillation (VF), which account for the majority of sudden cardiac deaths (SCDs). We sought to examine echocardiographic parameters that can predict VT as documented by implantable cardioverter-defibrillator (ICD) appropriate discharge. We examine echocardiographic parameters other than ejection fraction that may predict VT as recorded via rates of ICD discharge. Methods Analysis of 586 patients (469 males; mean age = 68 ± 3 years; mean follow-up time of 11 ± 14 months) was undertaken. Echo parameters assessed included left ventricular (LV) internal end diastolic/systolic dimension (LVIDd, LVIDs), relative wall thickness (RWT), and left atrial (LA) size. Results The incidence of VT was 0.22 (114 VT episodes per 528 person-years of follow-up time). Median time-to-first VT was 3.8 years. VT was documented in 79 patients (59 first VT incidence, 20 multiple). The echocardiographic parameter associated with first VT was LVIDs >4 cm ( P = 0.02). Conclusion The main echocardiographic predictor associated with the first occurrence of VT was LVIDs >4 cm. Patients with an LVIDs >4 cm were 2.5 times more likely to have an episode of VT. Changes in these echocardiographic parameters may warrant aggressive pharmacologic therapy and implantation of an ICD.

Premature ventricular contraction-induced cardiomyopathy in children

2015 ◽  
Vol 26 (4) ◽  
pp. 711-717 ◽  
Author(s):  
Zebulon Z. Spector ◽  
Stephen P. Seslar
Keyword(s):  
Ventricular Tachycardia ◽  
Radiofrequency Ablation ◽  
Premature Ventricular Contraction ◽  
Structural Heart Disease ◽  
Left Ventricular ◽  
Ventricular Contraction ◽  
Premature Ventricular Contractions ◽  
Multi Centre Study ◽  
History Of

AbstractBackgroundAdults with high premature ventricular contraction burden can develop left ventricular dilation, dysfunction, and strain, consistent with a cardiomyopathy, which is reversible with radiofrequency ablation of the premature ventricular contractions. Evidence in children with similar ectopy burden is limited. We performed a single-centre retrospective review to examine the prevalence of premature ventricular contraction-induced cardiomyopathy, natural history of ventricular ectopy, and progression to ventricular tachycardia in children with frequent premature ventricular contractions.MethodsChildren aged between 6 months and 18 years, with premature ventricular contractions comprising at least 20% of rhythm on 24-hour Holter monitor, were included in our study. Those with significant structural heart disease, ventricular tachycardia greater than 1% of rhythm at the time of premature ventricular contraction diagnosis, or family history of cardiomyopathy – except tachycardia-induced – were excluded. Cardiomyopathy was defined by echocardiographic assessment.ResultsA total of 36 children met the study criteria; seven patients (19.4%, 95% CI 6.2–32.6%) met the criteria for cardiomyopathy, mostly at initial presentation. Ectopy decreased to <10% of beats without intervention in 16.7% (95% CI 4.3–29.1%) of the patients. No patient progressed to having ventricular tachycardia as more than 1% of beats on follow-up Holter. Radiofrequency ablation was performed in three patients without cardiomyopathy.ConclusionsOur study demonstrates a higher prevalence of cardiomyopathy among children with high premature ventricular contraction burden than that previously shown. Ectopy tended to persist throughout follow-up. These trends suggest the need for a multi-centre study on frequent premature ventricular contractions in children. In the interim, regular follow-up with imaging to evaluate for cardiomyopathy is warranted.

Radiofrequency Ablation of Idiopathic Left Ventricular Tachycardia with Changing EGG Morphology

1998 ◽  
Vol 21 (8) ◽  
pp. 1668-1671 ◽  
Author(s):  
YI-JEN CHEN ◽  
SHIH-ANN CHEN ◽  
CHING-TAI TAI ◽  
CHERN-EN CHIANG ◽  
SHIH-HUANG LEE ◽  
...  
Keyword(s):  
Ventricular Tachycardia ◽  
Radiofrequency Ablation ◽  
Left Ventricular ◽  
Idiopathic Left Ventricular Tachycardia ◽  
Egg Morphology

A 52-year-old woman with ventricular tachycardia

Heart ◽  
2018 ◽  
Vol 104 (24) ◽  
pp. 2025-2043 ◽  
Author(s):  
Ana Fidalgo ◽  
Leticia Fernandez-Friera ◽  
Jorge Solis
Keyword(s):  
Heart Disease ◽  
Ventricular Tachycardia ◽  
Pulse Sequence ◽  
Electrophysiological Study ◽  
Structural Heart Disease ◽  
Left Ventricular ◽  
Left Ventricular Cavity ◽  
List Type ◽  
Oblique View ◽  
Cardiovascular Mr

Clinical introductionA 52-year-old woman with shortness of breath and palpitations was referred to a cardiologist. A 24-hour Holter demonstrated high density (37%) of ventricular premature beats (VPBs) and long runs of non-sustained (eventually sustained) monomorphic ventricular tachycardia (VT) with the same morphology as several VPBs detected in a 12-lead ECG (figure 1A). A transthoracic echocardiogram was performed, and the patient’s evaluation was completed with a functional and gadolinium-enhanced cardiovascular MR (CMR) study (figure 1B,C) to assess structural heart disease. In a follow-up visit, an electrophysiological study (EPS) was performed to identify the origin of VPBs and VT (figure 1D).Figure 1(A) A 12-lead ECG. (B) Cine CMR-SSFP (steady-state-free-precession) sequence on a three-chamber view. (C) Inversion-recovery gradient echo CMR pulse sequence for delayed enhancement assessment. (D) Three-dimensional electroanatomic voltage mapping of the left ventricular cavity (cranial left anterior oblique view). CMR, cardiovascular MR.QuestionWhat is the most likely cause of VPBs and VT?Idiopathic VT in the absence of structural heart disease.Bileaflet mitral valve prolapse (MVP).Dilated cardiomyopathy.Left ventricular non-compaction cardiomyopathy.Ischaemic cardiomyopathy.

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