scholarly journals Aldose Reductase Inhibition Prevents Development of Posterior Capsular Opacification in an In Vivo Model of Cataract Surgery

2018 ◽  
Vol 59 (8) ◽  
pp. 3591 ◽  
Author(s):  
Leonid M. Zukin ◽  
Michelle G. Pedler ◽  
Sergio Groman-Lupa ◽  
Mina Pantcheva ◽  
David A. Ammar ◽  
...  
Keyword(s):  
Cataract Surgery ◽  
Aldose Reductase ◽  
Posterior Capsular Opacification ◽  
In Vivo Model ◽  
Aldose Reductase Inhibition

scholarly journals Prevention of Posterior Capsular Opacification through Aldose Reductase Inhibition

2009 ◽  
Vol 50 (2) ◽  
pp. 752 ◽  
Author(s):  
Umesh C. S. Yadav ◽  
Farshid Ighani-Hosseinabad ◽  
Frederik J. G. M. van Kuijk ◽  
Satish K. Srivastava ◽  
Kota V. Ramana
Keyword(s):  
Aldose Reductase ◽  
Posterior Capsular Opacification ◽  
Aldose Reductase Inhibition

Panel Discussion II: Usefulness of in vitro and in vivo Model Systems for Predicting the Adverse Public Health Outcome for Antimicrobial Drug Residues in Food

2000 ◽  
Vol 12 (3) ◽  
pp. 45-53
Author(s):  
Andrew Beaulieu ◽  
Jacques Boisseau ◽  
Carl Cerniglia ◽  
Denis Corpet ◽  
A. Haydée Fernández ◽  
...  
Keyword(s):  
Public Health ◽  
Health Outcome ◽  
Panel Discussion ◽  
Antimicrobial Drug ◽  
Model Systems ◽  
In Vivo Model ◽  
Drug Residues ◽  
Public Health Outcome

P1735 Arginine supplementation and oxidative processes: in vivo model

2003 ◽  
Vol 24 (5) ◽  
pp. 330
Author(s):  
A KOPFF
Keyword(s):  
In Vivo Model ◽  
Oxidative Processes ◽  
Arginine Supplementation

Establishment of an in vivo model for KCNJ5 dependent hyperaldosteronism

2015 ◽  
Vol 122 (03) ◽  
Author(s):  
U Lichtenauer ◽  
PL Schmid ◽  
A Oßwald ◽  
I Renner-Müller ◽  
M Reincke ◽  
...  
Keyword(s):  
In Vivo Model

An in vivo Model for the Assessment of Acute Fibrinolytic Capacity of the Endothelium

1997 ◽  
Vol 78 (04) ◽  
pp. 1242-1248 ◽  
Author(s):  
David E Newby ◽  
Robert A Wright ◽  
Christopher A Ludlam ◽  
Keith A A Fox ◽  
Nicholas A Boon ◽  
...  
Keyword(s):  
Blood Flow ◽  
Substance P ◽  
Sodium Nitroprusside ◽  
Plasma Concentrations ◽  
In Vivo Model ◽  
Forearm Circulation ◽  
Von Willebrand ◽  
Local Release ◽  
Willebrand Factor

SummaryThe effects on blood flow and plasma fibrinolytic and coagulation parameters of intraarterial substance P, an endothelium dependent vasodilator, and sodium nitroprusside, a control endothelium independent vasodilator, were studied in the human forearm circulation. At subsystemic locally active doses, both substance P (2-8 pmol/min) and sodium nitroprusside (2-8 μg/min) caused dose-dependent vasodilatation (p <0.001 for both) without affecting plasma concentrations of PAI-1, von Willebrand factor antigen or factor VIII:C activity. Substance P caused local increases in t-PA antigen and activity (p <0.001) in the infused arm while sodium nitroprusside did not. At higher doses, substance P increased blood flow and t-PA concentrations in the noninfused arm. We conclude that brief, locally active and subsystemic infusions of intraarterial substance P cause a rapid and substantial local release of t-PA which appear to act via a flow and nitric oxide independent mechanism. This model should provide a useful and selective method of assessing the in vivo capacity of the forearm endothelium to release t-PA acutely.

Closure of Fetoscopic Access Sites with Amniotic Extracellular Matrix Scaffolds in an In Vivo Model

2006 ◽  
Vol 66 (S 01) ◽  
Author(s):  
N Ochsenbein-Kölble ◽  
J Jani ◽  
G Verbist ◽  
L Lewi ◽  
K Marquardt ◽  
...  
Keyword(s):  
Extracellular Matrix ◽  
In Vivo Model ◽  
Extracellular Matrix Scaffolds

Reduction of glomerular hyperfiltration in normoalbuminuric IDDM patients by 6 mo of aldose reductase inhibition

Diabetes ◽  
1991 ◽  
Vol 40 (5) ◽  
pp. 527-531 ◽  
Author(s):  
M. M. Pedersen ◽  
J. S. Christiansen ◽  
C. E. Mogensen
Keyword(s):  
Aldose Reductase ◽  
Glomerular Hyperfiltration ◽  
Aldose Reductase Inhibition
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