scholarly journals A Case of Salicylate Toxicity Presenting with Acute Focal Neurologic Deficit in a 61-Year-Old Woman with a History of Stroke

2020 ◽  
Vol 21 ◽  
Author(s):  
Tessa M. Delaney ◽  
Jason T. Helvey ◽  
Jason F. Shiffermiller
Keyword(s):  
Neurologic Deficit ◽  
History Of ◽  
Focal Neurologic Deficit

scholarly journals An acute ischemic stroke after multiple bee stings in an elderly patient

2013 ◽  
Vol 4 (2) ◽  
pp. 55-58
Author(s):  
Niranjan Mambally Rachaiah ◽  
Rajashekar Hirisave Kalegowda ◽  
Rashmi Bhadravathy Krishnaswamy
Keyword(s):  
Computed Tomography ◽  
Elderly Patient ◽  
Ischemic Stroke ◽  
Acute Ischemic Stroke ◽  
Neurological Complications ◽  
Neurologic Deficit ◽  
Allergic Reactions ◽  
Medical Sciences ◽  
Wasp Stings ◽  
Focal Neurologic Deficit

Although bee stings can cause local and systemic allergic reactions, neurological complications are rare. There are few reports of stroke following bee or wasp stings. We report a 70-year-old healthy man developed a focal neurologic deficit 5 hours after multiple bee stings, which was confirmed to be an acute ischemic stroke on computed tomography (CT) scan.DOI: http://dx.doi.org/10.3126/ajms.v4i2.7892Asian Journal of Medical Sciences 4(2013) 55-58

Cerebrovascular Disease 2: Stroke (DRAFT)

2018 ◽  
Author(s):  
Tamara Kaplan ◽  
Tracey Milligan
Keyword(s):  
Cerebrovascular Disease ◽  
Neurologic Deficit ◽  
Definition Of ◽  
Focal Neurologic Deficit

The video in this chapter explores cerebrovascular disease, and focuses on stroke. It discusses the definition of stroke as a sudden focal neurologic deficit that persists for more than 24 hours, as well as ischemic strokes (embolic, thrombotic, or due to hypoperfusion), and intraparenchymal hemorrhagic strokes.

scholarly journals Family history of stroke and severity of neurologic deficit after stroke

Neurology ◽  
2006 ◽  
Vol 67 (8) ◽  
pp. 1396-1402 ◽  
Author(s):  
J. F. Meschia ◽  
L. D. Case ◽  
B. B. Worrall ◽  
R. D. Brown ◽  
T. G. Brott ◽  
...  
Keyword(s):  
Family History ◽  
Neurologic Deficit ◽  
History Of

scholarly journals Pyogenic Lumbar Facet Joint Infection with Foot Drop

2021 ◽  
Vol 2021 ◽  
pp. 1-5
Author(s):  
Haruka Maehara ◽  
Toshihiro Sano ◽  
Yuki Yanagawa ◽  
Kyuichi Hashimoto ◽  
Nobuaki Tadokoro
Keyword(s):  
Low Back Pain ◽  
Back Pain ◽  
Facet Joint ◽  
Joint Infection ◽  
Neurologic Deficit ◽  
Neurological Dysfunction ◽  
Foot Drop ◽  
Low Back ◽  
Paravertebral Muscles ◽  
History Of

Pyogenic facet joint infection (PFJI) is a relatively rare spinal infection. Clinical suspicion of this condition is a key for diagnosis. We report a case of PFJI which required decompression surgery for severe neurological dysfunction. The patient was a 44-year-old woman who had a previous history of orthotic therapy for idiopathic scoliosis. The patient was admitted to our hospital with a history of two days of high fever and severe low back pain. There was no neurologic deficit, and blood tests revealed high levels of inflammatory markers. There was a slight amount of fluid that had collected at L4/5 facet joint in lumbar MRI. She was admitted for examination and treatment of fever of unknown origin and low back pain. Antibiotic treatment started the day after hospitalization since the first report of the blood culture taken upon admission tested positive to gram-positive cocci. As low back pain and fever persisted, an MRI was taken again on the fifth day of hospitalization. Repeated MRI showed fluid extension from the left facet joint to paravertebral muscles and epidural space. She was diagnosed with PFJI, and facet joint puncture was performed. At this time, it became clear that she had foot drop on the right, the contralateral side of the PFJI. She underwent irrigation, debridement, and partial laminectomy. Methicillin-sensitive Staphylococcus aureus (MSSA) was detected in blood cultures at the time of hospitalization, in the puncture fluid and tissue collected during surgery. The patient recovered completely from foot drop after the operation and a three-month course of antibiotics. As the imaging findings may be inadequate in the early stages of onset and PFJI potentially causes neurologic deficit such as foot drop, neurological findings need to be carefully observed even after hospitalization and one should reexamine the MRI if symptoms or clinical findings did not improve or were aggravated.

Republished: Aberrant cervical vasculature anastomosis as cause of neck pain and successful treatment with embolization technique

2016 ◽  
Vol 8 (11) ◽  
pp. e46-e46
Author(s):  
Lucy He ◽  
Travis R Ladner ◽  
Mark Cobb ◽  
J Mocco
Keyword(s):  
Pain Relief ◽  
Neck Pain ◽  
Successful Treatment ◽  
Endovascular Embolization ◽  
Neurologic Deficit ◽  
Occipital Artery ◽  
Complete Pain Relief ◽  
Arterial Trunk ◽  
Focal Neurologic Deficit

We report a patient with non-dermatomal radiating neck pain without focal neurologic deficit. Traditional workup could not identify an anatomic or biomechanical cause. Imaging showed a deep cervical vessel centered in the region of pain. Angiography later identified an aberrant anastomosis of this vessel with the occipital artery. Subsequent endovascular embolization of this arterial trunk resulted in complete pain relief.

Unexpected Focal Neurologic Deficit on Emergence From Anesthesia

1987 ◽  
Vol 67 (5) ◽  
pp. 823-826 ◽  
Author(s):  
SUSAN B. OLIVER ◽  
ROY F. CUCCHIARA ◽  
MARK A. WARNER ◽  
JESSE J. MUIR
Keyword(s):  
Neurologic Deficit ◽  
Focal Neurologic Deficit

Abnormal ECG patterns during the acute phase of subarachnoid hemorrhage in patients without previous heart disease

Open Medicine ◽  
2006 ◽  
Vol 1 (2) ◽  
pp. 148-157 ◽  
Author(s):  
Penka Atanassova ◽  
Maria Tokmakova ◽  
Albena Djurkova ◽  
Vulcho Naydenov ◽  
Nedka Chalakova ◽  
...  
Keyword(s):  
Heart Disease ◽  
Subarachnoid Hemorrhage ◽  
Acute Phase ◽  
Myocardial Dysfunction ◽  
Neurologic Deficit ◽  
Historical Study ◽  
Ecg Changes ◽  
Heart Pathology ◽  
History Of ◽  
Single Cohort

AbstractSubarachnoid hemorrhage (SAH) occurs primarily during early to mid-adulthood; approximately 30% of individuals with SAH die within 2 weeks, and mortality is 30% to 45%. SAH happens suddenly, without patients being aware of previous heart abnormalities. Here, we performed a pilot single cohort (historical) study to examine the hypothesis that early abnormal electrocardiographic (ECG) changes may reveal unknown but “silent” heart pathologies in SAH patients without previous heart disease (PHD). Data were collected retrospectively on 56 consecutive patients during the acute phase of SAH (29 men, 27 women; mean age 49.0 ± 6.2 years) with different degrees of neurologic deficit (Hunt-Hess scale assessment) in a 2-year period single-cohort study. Repolarization abnormalities were most frequent (p<0.05) and were independent of a history of PHD, although it corresponded to a higher risk for such abnormalities (odds ratio OR=3.21; CI95%=1.01–10.22). ECG changes in patients without PHD were similar to those in PHD patients, confirming the hypothesis that SAH is associated with previously “silent” heart pathology. The increased frequency of ECG changes in PHD patients and their high incidence in no-PHD patients suggested a neurogenic form of myocardial dysfunction following SAH. Notably, repolarization changes were more frequent in patients with less severe deficit (p<0.05), whereas rhythm and conductive abnormalities were more frequent in patients with more severe neurologic deficit.

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