Heart Failure due to Tension Hydrothorax after Left Pneumonectomy

2013 ◽  
Vol 16 (6) ◽  
pp. 319 ◽  
Author(s):  
Kim Maguire ◽  
Calvin Leung ◽  
Visali Kodali ◽  
Brice Taylor ◽  
Jacques-Pierre Fontaine ◽  
...  
Keyword(s):  
Heart Failure ◽  
Cardiac Output ◽  
Rare Complication ◽  
Right Heart Failure ◽  
Symptomatic Improvement ◽  
Extrapleural Pneumonectomy ◽  
Pleural Mesothelioma ◽  
Low Cardiac Output ◽  
Mediastinal Shift ◽  
Left Pleural Effusion

Tension hydrothorax is a rare complication of pneumonectomy for pleural mesothelioma and an exceptionally rare cause of heart failure. We describe a patient who had undergone extrapleural pneumonectomy, chemotherapy, and radiation for pleural mesothelioma and who developed heart failure symptoms within months of the completion of treatment. Investigation showed a massive left pleural effusion resulting in tension hydrothorax, mediastinal shift, and evidence of right heart failure with constrictive physiology and low cardiac output. Therapeutic thoracentesis resulted in increase in cardiac output and symptomatic improvement.

scholarly journals Low Cardiac Output Leads Hepatic Fibrosis in Right Heart Failure Model Rats

PLoS ONE ◽  
2016 ◽  
Vol 11 (2) ◽  
pp. e0148666 ◽  
Author(s):  
Yoshitaka Fujimoto ◽  
Takashi Urashima ◽  
Daisuke Shimura ◽  
Reiji Ito ◽  
Sadataka Kawachi ◽  
...  
Keyword(s):  
Heart Failure ◽  
Cardiac Output ◽  
Hepatic Fibrosis ◽  
Right Heart Failure ◽  
Failure Model ◽  
Right Heart ◽  
Low Cardiac Output ◽  
Heart Failure Model

scholarly journals Initiation of Angiotensin Receptor-Neprilysin Inhibitor in Heart Failure With Low Cardiac Output

2019 ◽  
Vol 74 (18) ◽  
pp. 2326-2327 ◽  
Author(s):  
Trejeeve Martyn ◽  
Kathleen D. Faulkenberg ◽  
Dmitry M. Yaranov ◽  
Chonyang L. Albert ◽  
Colleen Hutchinson ◽  
...  
Keyword(s):  
Heart Failure ◽  
Cardiac Output ◽  
Angiotensin Receptor ◽  
Low Cardiac Output ◽  
Neprilysin Inhibitor ◽  
Angiotensin Receptor Neprilysin Inhibitor

Effect of experimental heart failure on peripheral sympathetic vasoconstriction

1988 ◽  
Vol 254 (4) ◽  
pp. H727-H733 ◽  
Author(s):  
J. R. Wilson ◽  
W. Matthai ◽  
V. Lanoce ◽  
M. Frey ◽  
N. Ferraro
Keyword(s):  
Heart Failure ◽  
Cardiac Output ◽  
Chronic Heart Failure ◽  
Neurotransmitter Release ◽  
Ventricular Pacing ◽  
Low Cardiac Output ◽  
Vascular Responses ◽  
Experimental Heart Failure ◽  
Sympathetic Vasoconstriction ◽  
Rapid Ventricular Pacing

To investigate whether heart failure impairs peripheral sympathetic vasoconstriction, hindlimb vascular responses to lumbar chain stimulation (0.5-20 Hz) were studied in normal dogs and in dogs with chronic heart failure produced by rapid ventricular pacing. At lumbar chain stimulation rates of 0.5-3 Hz, hindlimb vascular responses were comparable in both groups. However, at stimulation rates of 5-20 Hz, vascular responses were significantly attenuated in the dogs with heart failure. Vascular responses to norepinephrine (0.1, 1, and 10 micrograms/min) were not altered. These findings suggest that chronic heart failure results in impaired sympathetic vasoconstriction, probably because of reduced neurotransmitter release. This abnormality may interfere with the capacity of the failing circulation to compensate for a low cardiac output and thereby intensify the severity of heart failure.

scholarly journals Low Cardiac Output Stimulates Vasopressin Release in Patients With Stage D Heart Failure

2014 ◽  
Vol 78 (9) ◽  
pp. 2259-2267 ◽  
Author(s):  
Teruhiko Imamura ◽  
Koichiro Kinugawa ◽  
Masaru Hatano ◽  
Takeo Fujino ◽  
Toshiro Inaba ◽  
...  
Keyword(s):  
Heart Failure ◽  
Cardiac Output ◽  
Vasopressin Release ◽  
Low Cardiac Output ◽  
Stage D

Application of levosimendan in acute heart failure patients with symptoms of low cardiac output: case series report

2013 ◽  
pp. 275-278 ◽  
Author(s):  
Jan Biegus ◽  
Robert Zymliński ◽  
Katarzyna Kulej ◽  
Joanna Szachniewicz ◽  
Waldemar Banasiak ◽  
...  
Keyword(s):  
Heart Failure ◽  
Cardiac Output ◽  
Acute Heart Failure ◽  
Case Series ◽  
Low Cardiac Output ◽  
Heart Failure Patients ◽  
Case Series Report ◽  
Series Report

How Prostacyclin Improves Cardiac Output in Right Heart Failure in Conjunction with Pulmonary Hypertension

2007 ◽  
Vol 175 (8) ◽  
pp. 846-850 ◽  
Author(s):  
François Kerbaul ◽  
Serge Brimioulle ◽  
Benoı̂t Rondelet ◽  
Céline Dewachter ◽  
Ives Hubloue ◽  
...  
Keyword(s):  
Heart Failure ◽  
Pulmonary Hypertension ◽  
Cardiac Output ◽  
Right Heart Failure ◽  
Right Heart

scholarly journals LOW CARDIAC OUTPUT STIMULATES VASOPRESSIN RELEASE IN PATIENTS WITH STAGE D HEART FAILURE: ITS RELEVANCE TO POOR PROGNOSIS AND REVERSAL BY SURGICAL TREATMENT

2014 ◽  
Vol 63 (12) ◽  
pp. A909
Author(s):  
Teruhiko Imamura ◽  
Koichiro Kinugawa ◽  
Masaru Hatano ◽  
Hironori Muraoka ◽  
Takeo Fujino ◽  
...  
Keyword(s):  
Heart Failure ◽  
Cardiac Output ◽  
Surgical Treatment ◽  
Poor Prognosis ◽  
Vasopressin Release ◽  
Low Cardiac Output ◽  
Stage D

scholarly journals Constrictive pericarditis after heart transplantation: a case report

2020 ◽  
Vol 4 (4) ◽  
pp. 1-6
Author(s):  
Vincent Tchana-Sato ◽  
Arnaud Ancion ◽  
François Ansart ◽  
Jean Olivier Defraigne
Keyword(s):  
Heart Failure ◽  
Pericardial Effusion ◽  
Heart Transplantation ◽  
Constrictive Pericarditis ◽  
Rare Complication ◽  
Volume Overload ◽  
Restrictive Cardiomyopathy ◽  
Right Heart Failure ◽  
Clinical Problem ◽  
Idiopathic Dilated Cardiomyopathy

Abstract Background Constrictive pericarditis (CP) is a disease characterized by inflammation, progressive fibrosis, and thickening of the pericardium. Constrictive pericarditis after heart transplantation (HT) is a rare phenomenon, with a reported incidence of 1.4–3.9%. It is an important clinical problem which shares similar clinical features with entities such as restrictive cardiomyopathy. Therefore, it poses diagnostic challenges and therapeutic dilemmas even for experienced clinicians. Case summary A 53-year-old patient developed a zoster infection with pericardial effusion 9 months after HT for idiopathic dilated cardiomyopathy. Two months later, he presented with leg oedema and ascites and was treated by diuretics for volume overload. He was readmitted 8 months later with features of right heart failure. Multimodal imaging investigations were suggestive of CP. He successfully recovered after a radical pericardiectomy. Discussion Constrictive pericarditis is a rare complication in HT. Heart transplant recipients (HTR) with a history of post-operative pericardial effusion, or with rejection episodes are at high risk of developing CP. Differentiating CP from other conditions that cause apparent congestive heart failure in HTR is challenging. Management of CP is mainly surgical pericardiectomy.

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