Campylobacter Infection

PEDIATRICS ◽  
1983 ◽  
Vol 72 (3) ◽  
pp. 437-437
Author(s):  
STANFORD T. SHULMAN ◽  
DONALD MOEL
Keyword(s):  
Hemolytic Uremic Syndrome ◽  
Campylobacter Jejuni ◽  
Enteric Pathogen ◽  
Bloody Diarrhea ◽  
Early Symptom ◽  
White Girl ◽  
Mother And Daughter ◽  
Uremic Syndrome ◽  
Campylobacter Infection

To the Editor.— Chamovitz et al1 recently reported the occurrence of hemolytic-uremia syndrome with evidence of Campylobacter jejuni infection in both a mother and daughter. Although they indicated that there were no previous reports of the association of this enteric pathogen with hemolytic-uremic syndrome, we must point out the report of Denneberg et al,2 who noted this association in 1982. In addition, we have recently treated a 22-month-old white girl with classic hemolytic-uremic syndrome who had bloody diarrhea as an early symptom.

Campylobacter jejuni-Associated Hemolytic-Uremic Syndrome in a Mother and Daughter

PEDIATRICS ◽  
1983 ◽  
Vol 71 (2) ◽  
pp. 253-256
Author(s):  
Bruce N. Chamovitz ◽  
Alan I. Hartstein ◽  
Steven R. Alexander ◽  
Annie B. Terry ◽  
Priscilla Short ◽  
...  
Keyword(s):  
Hemolytic Uremic Syndrome ◽  
Campylobacter Jejuni ◽  
Systematic Investigation ◽  
Stool Culture ◽  
Immunofluorescent Antibody ◽  
Mother And Daughter ◽  
Antibody Titers ◽  
Uremic Syndrome ◽  
Indirect Immunofluorescent ◽  
Stool Cultures

A mother and daughter with Campylobacter jejuni-associated hemolytic-uremic syndrome (HUS) are discussed. The mother was hospitalized with bloody diarrhea and HUS; C jejuni was isolated from her stool. The 2-year-old daughter had been admitted five days prior to her mother with HUS following a three-day prodrome of vomiting and diarrhea. Multiple stool cultures were negative for enteric pathogens; however, cultures were not obtained until the eighth hospital day and after antibiotic therapy. Extensive investigation failed to identify another cause for the diarrheal illness or HUS in our patients. Indirect immunofluorescent antibody titers for C jejuni were 1:32 and 1:16 for the mother and daughter, respectively. An asymptomatic 9-month-old son had C jejuni isolated from his stool and had an immunofluorescent antibody titer of 1:64. Three other family members were asymptomatic, stool-culture negative, and had immunofluorescent antibody titers ≤ 1:4. The susceptibility to develop HUS following an enteric antigenic stimulus is illustrated by the patients presented. The need for systematic investigation of all HUS cases for potential susceptibility markers, as well as an exhaustive etiologic search, is emphasized.

Incomplete hemolytic-uremic syndrome in Argentinean children with bloody diarrhea

1995 ◽  
Vol 127 (3) ◽  
pp. 364-367 ◽  
Author(s):  
Eduardo L. López ◽  
María Marta Contrini ◽  
Susana Devoto ◽  
María Fabiana De Rosa ◽  
María Gabriela Graña ◽  
...  
Keyword(s):  
Hemolytic Uremic Syndrome ◽  
Bloody Diarrhea ◽  
Uremic Syndrome

Colicinogeny of O157:H7 enterohemorrhagic Escherichia coli and the shielding of colicin and phage receptors by their O-antigenic side chains

1991 ◽  
Vol 37 (2) ◽  
pp. 97-104 ◽  
Author(s):  
D. E. Bradley ◽  
S. P. Howard ◽  
H. Lior
Keyword(s):  
Escherichia Coli ◽  
Molecular Weight ◽  
Hemolytic Uremic Syndrome ◽  
Key Words ◽  
Bloody Diarrhea ◽  
Enterohemorrhagic Escherichia Coli ◽  
Side Chains ◽  
Uremic Syndrome ◽  
O Antigens ◽  
Clinical Conditions

Twenty O157:H7 enterohemorrhagic Escherichia coli strains from patients with different clinical conditions were tested for colicinogeny and the presence of Verotoxin (VT) genes. From bloody diarrhea cases, 7/8 isolates and from hemolytic uremic syndrome cases 3/5 isolates all synthesized what appeared to be colicin D. The remaining strains, which included 7 from asymptomatic sources, were noncolicinogenic. The plasmid determining the colicin was found to be 1.4 kb larger than the 5.2-kb pColD. The colicin D protein had a molecular weight of about 90 000, whereas the O157 colicin was 87 000. The plasmid was designated pColD157 to reflect these differences. Of O157:H7 isolates 17/20 had genes for both of the Verotoxins VT1 and VT2, and the remaining 3/20 for VT1 only. There was no correlation between the presence of VT determinants and colicinogeny or symptoms. The O157:H7 strains exhibited significant resistance to other colicins and bacteriophages. Key words: O-antigens, enterohemorrhagic, colicin, Verotoxin, bacteriophages.

scholarly journals Investigation of an outbreak of bloody diarrhea complicated with hemolytic uremic syndrome

2014 ◽  
Vol 4 (4) ◽  
pp. 249 ◽  
Author(s):  
Otar Chokoshvili ◽  
Khatuna Lomashvili ◽  
Naile Malakmadze ◽  
Marika Geleishvil ◽  
Jonas Brant ◽  
...  
Keyword(s):  
Hemolytic Uremic Syndrome ◽  
Bloody Diarrhea ◽  
Uremic Syndrome

Campylobacter jejuni and Hemolytic-Uremic Syndrome

Nephron ◽  
1999 ◽  
Vol 82 (4) ◽  
pp. 363-364 ◽  
Author(s):  
Miguel Sillero ◽  
Jaume Almirall
Keyword(s):  
Hemolytic Uremic Syndrome ◽  
Campylobacter Jejuni ◽  
Uremic Syndrome

Tumor necrosis factor concentrations in hemolytic uremic syndrome patients and children with bloody diarrhea in Argentina

1995 ◽  
Vol 14 (7) ◽  
pp. 594-597 ◽  
Author(s):  
EDUARDO L. LÓPEZ ◽  
MARÍA MARTA CONTRINI ◽  
SUSANA DEVOTO ◽  
MARÍA FABIANA DE ROSA ◽  
MARÍA GABRIELA GRAÑA ◽  
...  
Keyword(s):  
Tumor Necrosis Factor ◽  
Hemolytic Uremic Syndrome ◽  
Tumor Necrosis ◽  
Bloody Diarrhea ◽  
Uremic Syndrome ◽  
Necrosis Factor
Sign in / Sign up

Export Citation Format

Share Document